Chronic Lead Intoxication at an Indoor Firing Range in Taiwan

1995 ◽  
Vol 33 (4) ◽  
pp. 371-372 ◽  
Author(s):  
Tang-Tat Chau ◽  
Wang-Yi Chen ◽  
Tien-Mu Hsiao ◽  
Hong-Wen Liu
2013 ◽  
Vol 14 (4) ◽  
pp. 395 ◽  
Author(s):  
Ahmed M. Soliman ◽  
Ehab A. Abu-Basha ◽  
Salah A. H. Youssef ◽  
Aziza M. Amer ◽  
Patricia A. Murphy ◽  
...  

1999 ◽  
Vol 131 (9) ◽  
pp. 716 ◽  
Author(s):  
Ana I. Sanchez-Fructuoso ◽  
Dolores Prats ◽  
Alberto Barrientos

1998 ◽  
Vol 160 (1) ◽  
pp. 47-53 ◽  
Author(s):  
S.Allen Counter ◽  
Leo H Buchanan ◽  
H.Diana Rosas ◽  
Fernando Ortega

Blood ◽  
1965 ◽  
Vol 26 (2) ◽  
pp. 181-189 ◽  
Author(s):  
ROBERT DRUYAN ◽  
BIRGITTA HAEGER-ARONSEN ◽  
WILFRIED VON STUDNITZ ◽  
JAN WALDENSTRÖM

Abstract Serum concentrations of ALA and PBG have been measured in normal subjects, patients with AIP, and lead workers. Both porphyrin precursors are significantly increased in serum from porphyric patients, and serum ALA is elevated in lead workers. Endogenous clearance measurements, when compared with creatinine clearances, are consistent with significant tubular reabsorption of ALA and PBG under physiologic circumstances, and with an "overflow" mechanism for increased excretion in AIP or lead intoxication. Three of 8 porphyric patients showed an aminoaciduria; tyrosine was involved in 2, tryptophan in the other 3. Evidence to suggest a specific renal defect contributing to increased ALA excretion in chronic lead intoxication was not obtained.


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