scholarly journals Can you explain the difference between “acute glomerulonephritis” and “acute nephritic syndrome”?

2018 ◽  
Vol 31 (2) ◽  
pp. 109-113
Author(s):  
Koichi Nakanishi
Keyword(s):  
Acute Glomerulonephritis ◽  
Nephritic Syndrome ◽  
Acute Nephritic Syndrome ◽  
The Difference

MO302ACUTE POST-INFECTIOUS GLOMERULONEPHRITIS IN ADULTS: A TUNISIAN SINGLE CENTER EXPERIENCE

2021 ◽  
Vol 36 (Supplement_1) ◽  
Author(s):  
Imen El Meknassi ◽  
Mrabet Sanda ◽  
Guedri Yosra ◽  
Zellema Dorsaf ◽  
Azzabi Awatef ◽  
...  
Keyword(s):  
Acute Kidney Injury ◽  
Kidney Injury ◽  
Acute Glomerulonephritis ◽  
Industrialized Countries ◽  
Single Center Experience ◽  
Nephritic Syndrome ◽  
Stage Renal Disease ◽  
End Stage ◽  
Post Infectious

Abstract Background and Aims Acute post-infectious glomerulonephritis (APIGN) is a reactive immunological disease. Its prevalence in industrialized countries is declining contrasting with developed ones. It is uncommon in adults but the prognosis may be reserved. The aim of our study was to evaluate the epidemiological, clinical and histological features of APIGN as well as its prognosis. Method A retrospective and descriptive study was conducted in our department. Were included all cases of histologically proven APIGN between December 2006 and December 2017. Results We had collected 38 cases. The mean age was 37.7 ± 17.8 years. The sex ratio was 1.92. Twelve (31.6%) patients were diabetic and four of them had already a chronic kidney disease (CKD). APIGN was preceded by an infection in 27 cases with an average interval of 10 ± 5 days. The most common site of infection was the respiratory tract (15 cases). At presentation, 27 patients had nephritic syndrome and 13 had nephrotic-range proteinuria. Hematuria was observed in 97.4%, peripheral edema in 84.2% and hypertension in 73.7% of cases. Most patients (78.9%) had acute kidney injury and 10 (26.3%) patients required dialysis. Renal biopsy had shown benign acute glomerulonephritis in 31 cases and malignant form in 7 cases. An underlying nephropathy was found in 12 cases with mostly a diabetic nephropathy. Corticosteroids were used in 3 cases of benign APIGN and 5 cases of malignant form. During the follow-up, CKD was noted in 14(36.8%) patients including 7(18.4%) patients who progressed to end-stage renal disease. Poor prognostic factors were diabetes, the presence of an underlying nephropathy in the biopsy, acute kidney injury and the need for dialysis. Conclusion The APIGN is uncommon in adults, yet its prognosis may be reserved with progression to CKD.

Wilson’s Disease Presenting With Haematuria and Swelling

2012 ◽  
Vol 23 (1) ◽  
pp. 51-52
Author(s):  
Pradip Kumar Dutta ◽  
Syed Md Jabed ◽  
Md Abul Kashem ◽  
Saibal Das ◽  
Md Nurul Huda
Keyword(s):  
Fanconi Syndrome ◽  
Wilson’S Disease ◽  
Disease Gene ◽  
Wilson's Disease ◽  
Late Diagnosis ◽  
Nephritic Syndrome ◽  
Rare Manifestation ◽  
Acute Nephritic Syndrome

Wilson’s disease gene located in Chromosime 3 is also expressed in Kidneys. So Wilson’s disease may have renal manifestations either as primary even or secondarily as Hepato renal syndrome. Patients commonly manifest as Fanconi syndrome or Urolithiasis. Haematuria and proteinuria is a rare manifestation. Here we are presenting a case who initially presented with haematuria and proteinuria (Acute nephritic syndrome) which masked features of Wilson’s disease and late diagnosis. JCMCTA 2012; 23(1): 51-52

scholarly journals Glomerular Diseases Associated with Malignancies: Histopathological Pattern and Association with Circulating Autoantibodies

Antibodies ◽  
2020 ◽  
Vol 9 (2) ◽  
pp. 18
Author(s):  
Sophia Lionaki ◽  
Smaragdi Marinaki ◽  
Konstantinos Panagiotellis ◽  
Ioanna Tsoumbou ◽  
George Liapis ◽  
...  
Keyword(s):  
Membranous Nephropathy ◽  
Kidney Biopsy ◽  
Rapidly Progressive Glomerulonephritis ◽  
Glomerular Diseases ◽  
Transmembrane Glycoprotein ◽  
Phospholipase A2 Receptor ◽  
Nephritic Syndrome ◽  
Acute Nephritic Syndrome ◽  
Circulating Autoantibodies ◽  
End Stage

Aim: Glomerular diseases (GD) associated with malignancies (AM, GDAM) have unique features, which are important to recognize, in the light of the progress made in cancer therapy. We aimed to describe the clinical and histopathological characteristics of patients with GDAM in relation to the presence of circulating autoantibodies, pointing to potential immune pathogenic pathways connecting cancer to GD. Materials and Methods: The included patients were studied retrospectively on the basis of a kidney biopsy proving GD and a related biopsy to establish the diagnosis of AM. We recorded patients’ demographics, serological and laboratory parameters, histopathological findings, and the type of malignancy, GD, and therapy. Results: In total, 41 patients with GDAM, with a mean age of 63.1 (±10.7) years, were studied. In 28 (68.3%) cases, GD was associated with a solid tumor, and in 13 (31.7%) patients with a lymphoid malignancy. The most frequent histopathological pattern was membranous nephropathy (43.9%). Overall, at the time of GD diagnosis, 17% of the patients were positive for antinuclear antibodies (ANA), and 12.2% for antineutrophil cytoplasmic autoantibodies (ANCA), all against myeloperoxidase (MPO). In addition, 93.3% of the patients who had membranous nephropathy were negative for transmembrane glycoprotein M-type phospholipase A2 receptor (PLA2R) antibody. Sixteen patients (39.0%) presented with acute nephritic syndrome, of whom five (31.25%) developed rapidly progressive glomerulonephritis. In a mean follow-up time of 36.1 (±28.3) months, nine (21.95%) patients ended up with end-stage kidney disease, and eight (19.5%) died. Conclusion: We found that 3.2% of patients who underwent a native kidney biopsy in our institution during the past decade, for any reason, were identified as having some type of GD associated with a malignancy. Serology indicated a significant presence of ANA or MPO-ANCA antibodies in patients with nephritic syndrome and the absence of PLA2R antibodies in patients with membranous nephropathy.

scholarly journals Studies on the renin-aldosterone system in the acute nephritic syndrome

1981 ◽  
Vol 19 (3) ◽  
pp. 445-453 ◽  
Author(s):  
Bernardo Rodríguez-Iturbe ◽  
Bruno Baggio ◽  
José Colina-Chourio ◽  
Silvana Favaro ◽  
Rafael García ◽  
...  
Keyword(s):  
Nephritic Syndrome ◽  
Acute Nephritic Syndrome

scholarly journals CLINICAL PROFILE AND SHORT-TERM OUTCOME OF ACUTE NEPHRITIC SYNDROME IN CHILDREN

2017 ◽  
Vol 6 (88) ◽  
pp. 6157-6160
Author(s):  
Surya Kandashamparambil Kamalakarababu ◽  
Ansu Sam ◽  
Sajini Varghese
Keyword(s):  
Clinical Profile ◽  
Short Term ◽  
Term Outcome ◽  
Short Term Outcome ◽  
Nephritic Syndrome ◽  
Acute Nephritic Syndrome

GLOMERULONEPHRITIS AND THE ACUTE NEPHRITIC SYNDROME

2010 ◽  
pp. 79-88
Author(s):  
Michael J. Field ◽  
David C. Harris ◽  
Carol A. Pollock
Keyword(s):  
Nephritic Syndrome ◽  
Acute Nephritic Syndrome

Crescentic postinfectious glomerulonephritis in an adult patient with juvenile nasopharyngeal angiofibroma

2021 ◽  
Vol 14 (4) ◽  
pp. e242785
Author(s):  
Sheryll Anne Manalili ◽  
Paolo Nikolai So ◽  
Maria Ana Louise Naidas ◽  
Anthony Russell Villanueva
Keyword(s):  
Crescentic Glomerulonephritis ◽  
Antineutrophil Cytoplasmic Antibody ◽  
Juvenile Nasopharyngeal Angiofibroma ◽  
Nasopharyngeal Swab ◽  
Nasopharyngeal Angiofibroma ◽  
Nephritic Syndrome ◽  
Postinfectious Glomerulonephritis ◽  
Acute Nephritic Syndrome ◽  
Gram Negative Organisms ◽  
Diffuse Proliferative Glomerulonephritis

Crescentic glomerulonephritis is usually associated with an acute nephritic syndrome with rapidly declining renal function. Postinfectious cases usually have a higher possibility of recovery. Juvenile nasopharyngeal angiofibroma (JNA) is a rare, locally aggressive tumour affecting mostly young men. A 28-year-old man presented with recurrent JNA initially excised 2 years prior. The patient was initially managed as a case of airway obstruction and pneumonia. He developed tea-coloured urine, oedema and acute kidney failure requiring dialysis while awaiting surgery. Urine and immunological studies (low C3, negative antineutrophil cytoplasmic antibody and antinucleosomal antibody and high antistreptolysin O) suggested a nephritic aetiology. Nasopharyngeal swab cultures of the mass revealed gram-negative organisms. Kidney biopsy showed diffuse proliferative glomerulonephritis compatible with a postinfectious glomerulonephritis with 77% cellular crescents. The mass was excised with histopathology consistent with JNA. The patient was eventually discharged off dialysis.

Sign in / Sign up

Export Citation Format

Share Document