Faculty Opinions recommendation of Surface traffic of dendritic CaV1.2 calcium channels in hippocampal neurons.

2011 ◽  
Author(s):  
Annette Dolphin
Keyword(s):  
Calcium Channels ◽  
Hippocampal Neurons

scholarly journals N-methyl-D-aspartate receptor-induced proteolytic conversion of postsynaptic class C L-type calcium channels in hippocampal neurons.

1996 ◽  
Vol 93 (8) ◽  
pp. 3362-3367 ◽  
Author(s):  
J. W. Hell ◽  
R. E. Westenbroek ◽  
L. J. Breeze ◽  
K. K. Wang ◽  
C. Chavkin ◽  
...  
Keyword(s):  
Calcium Channels ◽  
Hippocampal Neurons ◽  
Aspartate Receptor ◽  
Class C

scholarly journals Different Localizations and Functions of L-Type and N-Type Calcium Channels during Development of Hippocampal Neurons

2000 ◽  
Vol 227 (2) ◽  
pp. 581-594 ◽  
Author(s):  
Elena Pravettoni ◽  
Alberto Bacci ◽  
Silvia Coco ◽  
Paolo Forbicini ◽  
Michela Matteoli ◽  
...  
Keyword(s):  
Calcium Channels ◽  
Hippocampal Neurons

Fine Tuning of Intracellular Ca2+ Content by Pharmacological Agents – A Strategy to Prevent Synapse Loss in Alzheimer Disease Hippocampal Neurons

2021 ◽  
Vol 17 (12) ◽  
pp. 1065-1071
Author(s):  
Elena Popugaeva
Keyword(s):  
Alzheimer Disease ◽  
Calcium Channels ◽  
Hippocampal Neurons ◽  
Long Term Potentiation ◽  
Fine Tuning ◽  
Dominant Form ◽  
Pharmacological Agents ◽  
Chemical Structures ◽  
Synapse Loss ◽  
Β Amyloid

: Alzheimer disease is the dominant form of elderly dementia. Today all clinical trials that target β-amyloid have failed to indicate that β-amyloid may not be a causative agent in AD pathogenesis. Thus there is a need to search for alternative ways to treat AD patients. : Neuronal store-operated calcium entry is a fine-tuning mechanism that regulates intracellular Ca2+ content. Recent evidence suggests that store-operated calcium channels may be targeted with pharmacological agents in order to prevent synapse loss, recover long-term potentiation and change behavior. : Current mini-review discusses basic chemical structures that modulate intracellular calcium dysbalance via targeting store-operated calcium channels and their applicability as anti-AD pharmacological agents.

scholarly journals Activity-Dependent Facilitation of CaV1.3 Calcium Channels Promotes KCa3.1 Activation in Hippocampal Neurons

2017 ◽  
Vol 37 (46) ◽  
pp. 11255-11270 ◽  
Author(s):  
Giriraj Sahu ◽  
Hadhimulya Asmara ◽  
Fang-Xiong Zhang ◽  
Gerald W. Zamponi ◽  
Ray W. Turner
Keyword(s):  
Calcium Channels ◽  
Hippocampal Neurons ◽  
Activity Dependent
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