Faculty Opinions recommendation of Hepatocyte cytotoxicity is facilitated by asialoglycoprotein receptor.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.13338960.14706061 ◽

2011 ◽

Author(s):

Carol Casey ◽

Terrence Donohue

Keyword(s):

Asialoglycoprotein Receptor

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Autoantibodies to asialoglycoprotein receptor and their relevance to liver function and injury in the woodchuck model of hepatitis B

Journal of Hepatology ◽

10.1016/s0168-8278(01)80762-5 ◽

2001 ◽

Vol 34 (0) ◽

pp. 206-207

Author(s):

T Michalak

Keyword(s):

Hepatitis B ◽

Liver Function ◽

Asialoglycoprotein Receptor ◽

Woodchuck Model

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Autoantibodies to asialoglycoprotein receptor (ASGPR) measured by a novel ELISA–revival of a disease-activity marker in autoimmune hepatitis (AIH)

Zeitschrift für Gastroenterologie ◽

10.1055/s-0029-1246505 ◽

2010 ◽

Vol 48 (01) ◽

Author(s):

K Conrad ◽

D Roggenbuck ◽

E Feist ◽

G Hausdorf ◽

T Buettner ◽

...

Keyword(s):

Disease Activity ◽

Autoimmune Hepatitis ◽

Asialoglycoprotein Receptor ◽

Activity Marker

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Defective asialoglycoprotein receptor endocytosis mediated by tyrosine kinase inhibitors. Requirement for a tyrosine in the receptor internalization signal

Journal of Biological Chemistry ◽

10.1016/s0021-9258(19)78084-3 ◽

1994 ◽

Vol 269 (15) ◽

pp. 11011-11017

Author(s):

R.J. Fallon ◽

M. Danaher ◽

R.L. Saylors ◽

A. Saxena

Keyword(s):

Tyrosine Kinase ◽

Tyrosine Kinase Inhibitors ◽

Kinase Inhibitors ◽

Asialoglycoprotein Receptor ◽

Receptor Internalization ◽

Receptor Endocytosis

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Endocytosis and degradation mediated by the asialoglycoprotein receptor in isolated rat hepatocytes.

Journal of Biological Chemistry ◽

10.1016/s0021-9258(19)68175-5 ◽

1982 ◽

Vol 257 (3) ◽

pp. 1201-1207 ◽

Author(s):

P.H. Weigel ◽

J.A. Oka

Keyword(s):

Rat Hepatocytes ◽

Asialoglycoprotein Receptor ◽

Isolated Rat Hepatocytes ◽

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Transcriptional and post-transcriptional regulation of the asialoglycoprotein receptor in normal and neoplastic rat liver.

Journal of Biological Chemistry ◽

10.1016/s0021-9258(18)67254-0 ◽

1986 ◽

Vol 261 (26) ◽

pp. 12400-12407

Author(s):

B E Huber ◽

I B Glowinski ◽

S S Thorgeirsson

Keyword(s):

Transcriptional Regulation ◽

Rat Liver ◽

Asialoglycoprotein Receptor ◽

Post Transcriptional Regulation

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Characterization of the asialoglycoprotein receptor on isolated rat hepatocytes.

Journal of Biological Chemistry ◽

10.1016/s0021-9258(18)43708-8 ◽

1980 ◽

Vol 255 (13) ◽

pp. 6111-6120 ◽

Author(s):

P.H. Weigel

Keyword(s):

Rat Hepatocytes ◽

Asialoglycoprotein Receptor ◽

Isolated Rat Hepatocytes ◽

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Impairment of the asialoglycoprotein receptor by ethanol oxidation

Biochemical Pharmacology ◽

10.1016/s0006-2952(96)00524-2 ◽

1996 ◽

Vol 52 (10) ◽

pp. 1499-1505 ◽

Author(s):

Dahn L. Clemens ◽

Christine M. Halgard ◽

Jack R. Cole ◽

Rodney M. Miles ◽

Michael F. Sorrell ◽

...

Keyword(s):

Ethanol Oxidation ◽

Asialoglycoprotein Receptor

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Recycling of the asialoglycoprotein receptor in isolated rat hepatocytes. Dissociation of internalized ligand from receptor occurs in two kinetically and thermally distinguishable compartments.

Journal of Biological Chemistry ◽

10.1016/s0021-9258(17)44450-4 ◽

1983 ◽

Vol 258 (17) ◽

pp. 10253-10262 ◽

Author(s):

J A Oka ◽

P H Weigel

Keyword(s):

Rat Hepatocytes ◽

Asialoglycoprotein Receptor ◽

Isolated Rat Hepatocytes ◽

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Endoplasmic reticulum degradation of a subunit of the asialoglycoprotein receptor in vitro. Vesicular transport from endoplasmic reticulum is unnecessary.

Journal of Biological Chemistry ◽

10.1016/s0021-9258(18)48445-1 ◽

1992 ◽

Vol 267 (1) ◽

pp. 5-8 ◽

Author(s):

L WikstrɆm ◽

H F Lodish

Keyword(s):

Endoplasmic Reticulum ◽

Vesicular Transport ◽

Asialoglycoprotein Receptor ◽

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Recycling of the hepatocyte asialoglycoprotein receptor does not require delivery of ligand to lysosomes.

Journal of Biological Chemistry ◽

10.1016/s0021-9258(20)65096-7 ◽

1982 ◽

Vol 257 (11) ◽

pp. 6007-6009

Author(s):

J U Baenziger ◽

D Fiete

Keyword(s):

Asialoglycoprotein Receptor

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