The surgical treatment of portal hypertension, bleeding esophageal varices and ascites. By M. Judson Mackby, M.D., D.A.B.S., F.I.C.S. 10 × 6½ in. Pp. 250 + xviii. Illustrated. 1961. Springfield, Illinois: Charles C. Thomas. (Oxford: Blackwell Scientific Publications Ltd.) 84s

1961 ◽  
Vol 48 (212) ◽  
pp. 691-691
2018 ◽  
Vol 1 (1) ◽  
pp. 14-16
Author(s):  
Soonthorn Chonprasertsuk

The noncirrhotic portal hypertension is an uncommon cause of bleeding esophagealvarices. This condition must be suspected in patients with preserved liver function. We reporta 25-year old man with SLE disease who presented with hematemesis. He had no historyor risk factors for an underlying liver condition. A huge splenomegaly was detectedby physical examination. The EGD found three large varices with red wale sign, whereas liverfunction tests were unremarkable. The noncirrhotic portal hypertension was diagnosedand confirmed by liver histopathology. Figure 1 แสดงผลการส่องกล้องทางเดินอาหารส่วนบนพบ F3 varices with red wale sign


2019 ◽  
Vol 2019 ◽  
pp. 1-3
Author(s):  
Kaushal Majmudar ◽  
Michael Northcutt ◽  
Robert Gordon ◽  
Claus J. Fimmel

We describe a patient with compensated cirrhosis and portal hypertension who underwent continuous flow LVAD implantation. Shortly after LVAD implantation, the patient developed new onset bleeding esophageal varices and ultimately had a fatal outcome. Our experience suggests that even well-compensated cirrhotic patients with significant portal hypertension are at risk of variceal bleeding after LVAD placement.


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