Studies on the Mechanism of Radiological Contrast Media Induced Renal Failure

1989 ◽  
pp. 463-468 ◽  
Author(s):  
C. J. Powell ◽  
M. Dobrota ◽  
E. Holtz
Keyword(s):  
1989 ◽  
Vol 23 (4) ◽  
pp. 315-317 ◽  
Author(s):  
Barbro Spångberg-Viklund ◽  
Tomas Nikonoff ◽  
Marc Lundberg ◽  
Rutger Larsson ◽  
Tommy Skau ◽  
...  

2005 ◽  
Vol 72 (4) ◽  
pp. 446-456
Author(s):  
C. Alberti ◽  
M. Piovano ◽  
A. Tizzani

Contrast media-induced nephropathy (CN) is an important cause of hospital-acquired acute renal failure. Patients with both diabetes mellitus and renal impairment are at high risk. CN pathophysiology involves activation of the tubulo-glomerular feedback and vasoactive mediators such as renin-angiotensin 2, endothelin, adenosine, ADH, etc. The risk of CN can be minimized by the use of non-ionic, low or isoosmolar, contrast material, adequate hydration and prophylactic pharmacological measures. In patients with chronic renal failure who are undergoing arteriography (e.g. coronary angiography and angioplasty), periprocedural hemofiltration appears effective in preventing further renal damage due to contrast agents.


2013 ◽  
Vol 17 (3) ◽  
pp. 106-107
Author(s):  
P.S. Ngoya ◽  
Z Vawda ◽  
J.W Lotz

Nephrogenic systemic fibrosis (NSF), unknown before March 1997 and first described in 2000, is a systemic disorder characterised by widespread tissue fibrosis. The first known case occurred in 1997, after the use of gadolinium-based contrast agents (GBCAs) at high doses in patients with renal failure had become routine. An overwhelming majority occurred within weeks to months after injection of a GBCA. This note comprises guidelines on the prevention of NSF.


1979 ◽  
Vol 52 (618) ◽  
pp. 510-510
Author(s):  
Judith A. W. Webb ◽  
I. Kelsey Fry ◽  
W. R. Cattell
Keyword(s):  

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