Posterior Fossa Meningioma

2020 ◽  
pp. 291-296
Author(s):  
F. Allan Midyett ◽  
Suresh K. Mukherji
Skull Base ◽  
2000 ◽  
Vol Volume 10 (Number 1) ◽  
pp. 0043-0046 ◽  
Author(s):  
Gregory S. Harrison ◽  
Peter Chovan ◽  
Joung H. Lee

2019 ◽  
Vol 12 (3) ◽  
pp. e228454
Author(s):  
Cristiano Antunes ◽  
Rui Ramos ◽  
Maria João Machado ◽  
Miguel Afonso Filipe

Posterior fossa lesions may present with behavioural changes and/or progressive neurological deficit. Patients may have symptoms for long periods which may be attributed to other causes such as psychiatric diseases. We report a case of a 44-year-old woman with behavioural changes lasting for 5 years who lost her job, marriage and the guard of her sons. Latterly, she developed neurological deficit, hydrocephalus and intracranial hypertension. A giant left pontocerebellar angle mass was diagnosed. A retrosigmoid craniotomy was performed with total removal and cranial nerve function’s preservation. Histology revealed a grade I meningioma. The surgical approach for such huge lesions on pontocerebellar angle is controversial concerning patient’s positioning and surgical route. A brief revision is made. Since nowadays medical imaging is more easily accessible, it is mandatory to have a brain image in patients with behavioural changes and/or neurological deficit to exclude potential structural and curable causes such as in this case.


1986 ◽  
Vol 28 (1) ◽  
pp. 47-52 ◽  
Author(s):  
A. Catz ◽  
I. Reider-Groswasser

2001 ◽  
Vol 44 (4) ◽  
pp. 181-184
Author(s):  
Akinobu Kakigi ◽  
Shoichi Sawada ◽  
Taizo Takeda ◽  
Shunji Takeuchi

2001 ◽  
Vol 95 (5) ◽  
pp. 893-896 ◽  
Author(s):  
Abdul Rahman Al-Anazi ◽  
William Holliday ◽  
Bassem Sheikh ◽  
Fred Gentili

✓ Low-grade adenocarcinoma arising from the endolymphatic sac is an uncommon tumor that is characterized clinically by hearing loss and radiologically by temporal bone destruction. The authors report a case of low-grade adenocarcinoma of endolymphatic sac origin that mimicked a posterior fossa meningioma in both clinical and neuroimaging examinations. In this case, the most unusual and interesting feature was the lack of auditory symptoms and temporal bone destruction. The lesion occurred in a 21-year-old man who presented with headaches that had been increasing in frequency for 6 months and was associated with blurring of vision, diplopia, occasional nausea and vomiting, and gait disturbance. On examination, this patient exhibited bilateral papilledema, left sixth cranial nerve palsy, and gait ataxia. Neuroimaging studies revealed a large right posterior fossa tumor. At surgery, a hemorrhagic papillary adenocarcinoma of endolymphatic sac origin was found.


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