Metabolic Power and Oxygen Consumption in Soccer: Facts and Theories

Author(s):  
Cristian Osgnach ◽  
Pietro E. di Prampero
2020 ◽  
Vol 15 (10) ◽  
pp. 1400-1405
Author(s):  
Sebastian Kaufmann ◽  
Olaf Hoos ◽  
Timo Kuehl ◽  
Thomas Tietz ◽  
Dominik Reim ◽  
...  

Purpose: To analyze the energetic profiles of the Yo-Yo Intermittent Recovery Tests 1 and 2 (YYIR1 and YYIR2). Methods: Intermittent running distance (IR1D and IR2D), time to exhaustion (IR1T and IR2T), and total recovery time between shuttles (IR1R and IR2R) were measured in 10 well-trained male athletes (age 24.4 [2.0] y, height 182 [1] cm, weight 75.8 [7.9] kg). Respiratory gases and blood lactate (BLC) were obtained preexercise, during exercise, and until 15 min postexercise. Metabolic energy, average metabolic power , and energy share (percentage of aerobic [WAER], anaerobic lactic [WBLC], and anaerobic alactic energy system [WPCr]) were calculated using the PCr-La-O2 method. Results: Peak oxygen consumption was possibly higher in YYIR2 (60.3 [5.1] mL·kg−1·min−1) than in YYIR1 (P = .116, 57.7 [4.5] mL·kg−1·min−1, d = −0.58). IR1D, IR1T, and IR1R were very likely higher than IR2D, IR2T, and IR2R, respectively (P < .001, 1876 [391] vs 672 [132] m, d = −2.83; P < .001, 916 [175] vs 304 [57] s, d = −3.03; and P < .001, 460 [100] vs 150 [40] s, d = −2.83). Metabolic energy was most likely lower in YYIR2 than in YYIR1 (P < .001, 493.5 [118.1] vs 984.8 [171.7] kJ, d = 3.24). Average metabolic power was most likely higher in YYIR2 than in YYIR1 (P < .001, 21.5 [1.7] vs 14.5 [2.2] W·kg−1, d = 3.54). When considering aerobic phosphocreatine restoration during breaks between shuttles, WAER (P = .693, 49% [10%] vs 48% [5%], d = −0.16) was similar, WPCr (P = .165, 47% [11%] vs 42% [6%], d = −0.54) possibly higher, and WBLC (P < .001, 4% [1%] vs 10% [3%], d = 1.95) almost certainly lower in YYIR1 than in YYIR2. Conclusions: WAER and WPCr are predominant in YYIR1 and YYIR2 with almost identical WAER. Higher IR1D and IR1T in YYIR1 result in higher metabolic energy but lower average metabolic power and slightly lower peak oxygen consumption. Higher IR1R allows for higher reliance on WPCr in YYIR1, while YYIR2 requires a higher fraction of WBLC.


1987 ◽  
Vol 129 (1) ◽  
pp. 251-263 ◽  
Author(s):  
R. V. Baudinette ◽  
B. J. Gannon ◽  
W. B. Runciman ◽  
S. Wells ◽  
J. B. Love

Breathing, heart and gait frequencies, tidal volume, cardiac output, and rates of oxygen consumption were measured in tammar wallabies (Macropus eugenii Desmarest) hopping on a treadmill. At speeds greater than 1.6 ms-1 the rate of metabolic power consumption was independent of hopping speeds. Blood lactate levels within the speed range where VO2 was independent of speed showed a mean increase of 4.8 mmol l-1. During bipedal hopping, the frequencies of breathing and limb movement are phase-locked in the ratio of 1:1. Inspiration begins as the animal leaves the ground and may be a passive process driven by a visceral piston. A relatively large central tendon in the diaphragm may correlate this function. Unlike breathing frequencies, cardiac frequencies show no entrainment with hopping. The site of dissipation of the presumed large arterial pressure excursion is unknown.


2016 ◽  
Vol 37 (01) ◽  
pp. 77-81 ◽  
Author(s):  
C. Osgnach ◽  
E. Paolini ◽  
V. Roberti ◽  
M. Vettor ◽  
P. di Prampero

1978 ◽  
Vol 17 (04) ◽  
pp. 142-148
Author(s):  
U. Büll ◽  
S. Bürger ◽  
B. E. Strauer

Studies were carried out in order to determine the factors influencing myocardial 201T1 uptake. A total of 158 patients was examined with regard to both 201T1 uptake and the assessment of left ventricular and coronary function (e. g. quantitative ventriculography, coronary arteriography, coronary blood flow measurements). Moreover, 42 animal experiments (closed chest cat) were performed. The results demonstrate that:1) 201T1 uptake in the normal and hypertrophied human heart is linearly correlated with the muscle mass of the left ventricle (LVMM);2) 201T1 uptake is enhanced in the inner (subendocardial) layer and is decreased in the outer (subepicardial) layer of the left ventricular wall. The 201T1 uptake of the right ventricle is 40% lower in comparison to the left ventricle;3) the basic correlation between 201T1 uptake and LVMM is influenced by alterations of both myocardial flow and myocardial oxygen consumption; and4) inotropic interventions (isoproterenol, calcium, norepinephrine) as well as coronary dilatation (dipyridamole) may considerably augment 201T1 uptake in accordance with changes in myocardial oxygen consumption and/or myocardial flow.It is concluded that myocardial 201T1 uptake is determined by multiple factors. The major determinants have been shown to include (i) muscle mass, (ii) myocardial flow and (iii) myocardial oxygen consumption. The clinical data obtained from patient groups with normal ventricular function, with coronary artery disease, with left ventricular wall motion abnormalities and with different degree of left ventricular hypertrophy are correlated with quantitated myocardial 201T1 uptake.


1976 ◽  
Vol 35 (03) ◽  
pp. 712-716 ◽  
Author(s):  
D. Del Principe ◽  
G Mancuso ◽  
A Menichelli ◽  
G Maretto ◽  
G Sabetta

SummaryThe authors compared the oxygen consumption in platelets from the umbilical cord blood of 36 healthy newborn infants with that of 27 adult subjects, before and after thrombin addition (1.67 U/ml). Oxygen consumption at rest was 6 mμmol/109/min in adult control platelets and 5.26 in newborn infants. The burst in oxygen consumption after thrombin addition was 26.30 mμmol/109/min in adults and 24.90 in infants. Dinitrophenol did not inhibit the burst of O2 consumption in platelets in 8 out of 10 newborn infants, while the same concentration caused a decrease in 9 out of 10 adult subjects. Deoxyglucose inhibited the burst in O2 consumption in newborn infant and adult platelets by about 50%. KCN at the concentration of 10−4 M completely inhibited basal oxygen consumption but did not completely inhibit the burst after thrombin. At the concentration of 10−3 M, it inhibited both basal O2 consumption and the burst in infants and adult subjects.


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