Organotypic development of neonate rabbit hippocampus in roller tube culture

1984 ◽  
Vol 65 (1) ◽  
pp. 53-61 ◽  
Author(s):  
U. De Boni ◽  
A. A. Chong ◽  
L. A. Hawthorn
Keyword(s):  
Tube Culture ◽  
Rabbit Hippocampus ◽  
Roller Tube ◽  
Roller Tube Culture

Improved development of the chick embryo using roller-tube culture

1995 ◽  
Vol 11 (7) ◽  
pp. 259-260 ◽  
Author(s):  
D CONNOLLY ◽  
L MCNAUGBTON ◽  
R KRUMLAUF ◽  
J COOKE
Keyword(s):  
Chick Embryo ◽  
Tube Culture ◽  
Roller Tube ◽  
Roller Tube Culture

scholarly journals Rapid clonal propagation of ginger (Zingiber officinale Roscoe) by roller tube culture.

1988 ◽  
Vol 38 (4) ◽  
pp. 437-442 ◽  
Author(s):  
Yuji NOGUCHI ◽  
Osamu YAMAKAWA
Keyword(s):  
Clonal Propagation ◽  
Zingiber Officinale ◽  
Tube Culture ◽  
Roller Tube ◽  
Zingiber Officinale Roscoe ◽  
Roller Tube Culture

Immature rabbit hippocampus is damaged by systemic but not intraventricular kainic acid

1984 ◽  
Vol 13 (2) ◽  
pp. 219-227 ◽  
Author(s):  
J.E. Franck ◽  
P.A. Schwartzkroin
Keyword(s):  
Kainic Acid ◽  
Rabbit Hippocampus

An automated recording incubator for tube culture of microorganisms

1980 ◽  
Vol 2 (3) ◽  
pp. 194-200 ◽  
Author(s):  
Tomoaki Morimoto ◽  
Hiroshi Itoh ◽  
Ichiro Chibata
Keyword(s):  
Tube Culture

Conditioning-Specific Biophysical Alterations in Rabbit Hippocampus

1988 ◽  
pp. 89-104 ◽  
Author(s):  
John F. Disterhoft ◽  
Douglas A. Coulter ◽  
Daniel L. Alkon
Keyword(s):  
Rabbit Hippocampus

Aberrant pattern formation in myosin heavy chain mutants of Dictyostelium

Development ◽  
1994 ◽  
Vol 120 (3) ◽  
pp. 591-601 ◽  
Author(s):  
D. Traynor ◽  
M. Tasaka ◽  
T. Takeuchi ◽  
J. Williams
Keyword(s):  
Myosin Heavy Chain ◽  
Heavy Chain ◽  
Cell Types ◽  
Culture Conditions ◽  
Wild Type ◽  
Multicellular Aggregate ◽  
A Cells ◽  
Air Water Interface ◽  
Shape Changes ◽  
Roller Tube Culture

In mutant Dictyostelium strains that fail to accumulate the myosin heavy chain (MHC A), development is relatively normal up to the tight aggregate stage but is arrested prior to formation of the apical tip (DeLozanne and Spudich 1987, Knecht and Loomis, 1987). We show that in aggregates formed by such MHC A deficient (MHC A-) strains the proportions of pstA and pstB cells, the two prestalk cell types, and of prespore cells are similar to those found during normal development but their distribution is radically different. During the initial stages of normal slug formation, pstA cells move to the tip, pstB cells accumulate in the base and prespore cells occupy the remainder of the aggregate. In the aggregates initially formed by MHC A- mutants pstA cells are present in a central core, pstB cells are present in the cortex and prespore cells lie sandwiched between them. Eventually, cells within the cortex differentiate into mature stalk cells but spores are never formed. Mixing experiments, in which MHC A- cells are allowed to co-aggregate with an excess of normal cells, show that MHC A- prestalk cells enter the aggregate relatively normally but are unable to enter the slug tip or to migrate into the stalk at culmination and that MHC A- prespore cells accumulate in the lower part of the spore head during culmination. Thus MHC A- cells appear to be able to move within the multicellular aggregate but are incapable of participating in normal morphogenesis. The structures formed by MHC A- cells are very similar to those of the agglomerates that form when wild-type cells are developed in roller-tube culture, conditions that result in loss of the polarity imparted by the presence of an air-water interface. We propose formation of such a structure by MHC A- cells to be a default response, caused by their inability to undertake the shape changes and intercalatory cell movements that are necessary to form and extend the tip.

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