Maximizing the clinical outcome with mTOR inhibitors in the renal transplant recipient: defining the role of calcineurin inhibitors

2004 ◽  
Vol 17 (6) ◽  
Author(s):  
Bj�rn Nashan
Keyword(s):  
Renal Transplant ◽  
Clinical Outcome ◽  
Transplant Recipient ◽  
Renal Transplant Recipient ◽  
Calcineurin Inhibitors ◽  
Mtor Inhibitors

A Numerical Scale Comparison of Renal Transplant Recipient Experience with and Opinions about Calcineurin Inhibitors

2004 ◽  
Vol 97 (2) ◽  
pp. c35-c40 ◽  
Author(s):  
G.V. Ramesh Prasad ◽  
Michelle M. Nash ◽  
Philip A. McFarlane ◽  
Jeffrey S. Zaltzman
Keyword(s):  
Renal Transplant ◽  
Transplant Recipient ◽  
Renal Transplant Recipient ◽  
Calcineurin Inhibitors ◽  
Numerical Scale

scholarly journals Predicting clinical outcome in the elderly renal transplant recipient

2000 ◽  
Vol 57 (5) ◽  
pp. 2144-2150 ◽  
Author(s):  
Shannon E. Doyle ◽  
Arthur J. Matas ◽  
Kristen Gillingham ◽  
Mark E. Rosenberg
Keyword(s):  
Renal Transplant ◽  
Clinical Outcome ◽  
Transplant Recipient ◽  
Renal Transplant Recipient ◽  
The Elderly

scholarly journals Tacrolimus Induced Early Graft Dysfunction Secondary to Acute Tubular Necrosis in Renal Transplant Recipient - A Case Report

2012 ◽  
Vol 2 (1) ◽  
pp. 63-65 ◽  
Author(s):  
Md Tariful Hoque ◽  
Palash Mitra ◽  
Tasrina Shamnaz Samdani ◽  
Golzar Hossain ◽  
Zainal Abedin ◽  
...  
Keyword(s):  
Renal Transplant ◽  
Transplant Recipient ◽  
Renal Transplant Recipient ◽  
Acute Tubular Necrosis ◽  
Medical Condition ◽  
Immunosuppressive Agents ◽  
Esrd Patient ◽  
Calcineurin Inhibitors ◽  
Ischaemic Injury ◽  
Tubular Necrosis

Acute tubular necrosis (ATN) is a medical condition involving damage of the tubular cells of the nephron following a toxic or ischaemic injury. If the cause of ATN is removed then recovery is likely. Calcineurin inhibitors (ciclosporin & tacrolimus) are used as immunosuppressive agents in renal transplant recipient. But either of these two drugs can cause acute nephrotoxicity. A 62 years old known diabetic, hypertensive & ESRD patient after undergoing living related donor renal transplantation started passing significant amount of urine, but 12 hrs after transplantation the amount of urine output started to decrease and the patient became anuric after 17hrs of transplantation. Immediately the patient was evaluated thoroughly and calcineurin inhibitor (tacrolimus) was witheld. The patient again started passing urine from 4th POD. DOI: http://dx.doi.org/10.3329/birdem.v2i1.12365 Birdem Med J 2012; 2(1) 63-65

scholarly journals Role of Mesna in treating intractable hematuria in a post-renal transplant recipient – A rare case report

2015 ◽  
Vol 9 (2) ◽  
pp. 77-78
Author(s):  
Rohan Jaju ◽  
Mukund Andankar ◽  
Umesh Oza ◽  
R.K. Garg ◽  
Jamal Azmi ◽  
...  
Keyword(s):  
Case Report ◽  
Renal Transplant ◽  
Transplant Recipient ◽  
Renal Transplant Recipient ◽  
Rare Case ◽  
Rare Case Report

Role of Lovastatin in Prevention of Acute Rejection Episodes in Renal Transplant Recipient

1999 ◽  
Vol 67 (7) ◽  
pp. S157
Author(s):  
R K Sharma ◽  
K M Sahu ◽  
A Gupta ◽  
S Gulati ◽  
D K Agarwal ◽  
...  
Keyword(s):  
Renal Transplant ◽  
Transplant Recipient ◽  
Acute Rejection ◽  
Renal Transplant Recipient

scholarly journals Metastatic lung adenocarcinoma- associated thrombotic microangiopathy in a renal transplant recipient

2018 ◽  
Vol 11 (1) ◽  
pp. e226707 ◽  
Author(s):  
Eswari Vilayur ◽  
Jillian de Malmanche ◽  
Paul Trevillian ◽  
David Ferreira
Keyword(s):  
Renal Transplant ◽  
Transplant Recipient ◽  
Renal Transplant Recipient ◽  
Thrombotic Microangiopathy ◽  
Calcineurin Inhibitors ◽  
Diagnostic Challenge ◽  
Complement Regulatory Proteins ◽  
Vein Thrombosis ◽  
Metastatic Lung ◽  
Deep Vein

Thrombotic microangiopathy (TMA) after renal transplantation can be a diagnostic challenge. TMA can occur with calcineurin inhibitors, allograft rejection, infection, mutations in complement regulatory proteins and autoimmunity. A 52-year-old male renal transplant recipient presented with extensive deep vein thrombosis. He developed transfusion-dependent microangiopathic haemolytic anaemia with thrombocytopenia. He did not respond calcineurin inhibitor cessation, eculizumab or plasma exchange. ADAMTS13 and complement levels were normal. Infection and autoimmune screens were negative. A diagnosis of metastatic adenocarcinoma was made on bone marrow biopsy. This represents a rare case of malignancy-associated TMA in a renal transplant recipient. Early diagnosis can facilitate the prompt initiation of chemotherapy which is the only treatment option.

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