scholarly journals PTRA is useful for renal artery angina by atherosclerotic plaque rupture with unilateral functioning kidney

2021 ◽  
Author(s):  
Nobuhisa Morimoto ◽  
Seiichiro Honda ◽  
Ayumi Terai ◽  
Madoka Tanabe ◽  
Megumi Otani ◽  
...  
2021 ◽  
Author(s):  
Zhuowen Yang ◽  
Jianting Yao ◽  
Jianxin Wang ◽  
Cong Zhang ◽  
Yang Cao ◽  
...  

Pathological angiogenesis is a critical contributor to atherosclerotic plaque rupture. However, there are few effective theranostic strategies to stabilize plaques by suppressing neovascularization. A polymeric nanosystem using 3 nm manganese...


2019 ◽  
Vol 5 (1 (P)) ◽  
pp. 43
Author(s):  
Erwinanto Erwinanto

Atherosclerotic plaque rupture is closely related to acute coronary syndromes.Stabilization of atherosclerotic plaque which slashes plaque rupture is as importantas regression ofplaque size for reducing cardiovascular events. Dyslipidemia therapy targeting to decrease LDL cholesterol reduces cardiovascular events such as acute myocard infarct, stroke, and death which are suggested to be the result of plaque stabilization. Dyslipidemia therapy also regress atherosclerotic plaque into a smaller volume. Plaque regression improves coronary flow responsible for the reduction of myocardial infarction incidence in patients with coronary heart disease (CHD).This paper consists of two parts. The first part discusses the evidence of cardiovascular event reduction with statin. The second part describes dyslipidemia management based on the 2017 Indonesian Heart Association (PERKI) Guideline on the Management of Dyslipidemia


2000 ◽  
Vol 55 (6) ◽  
pp. 480-484 ◽  
Author(s):  
A.J. Bank ◽  
A. Versluis ◽  
S.M. Dodge ◽  
W.H. Douglas

Author(s):  
Ramses Galaz ◽  
Rosaire Mongrain ◽  
Valerie Pazos ◽  
Richard Leask ◽  
Jean Claude Tardif

Atherosclerotic plaque rupture has been extensively associated with acute myocardial infarctions, the leading cause of death in developed countries. When a plaque ruptures in the coronary arteries, it often leads to a clot formation that restricts the flow of blood downstream of the coronaries causing damage to the myocardial tissue. Hemodynamic factors combined with certain plaque morphologies and material properties play an important role in the mechanical failure of the plaque [1]. It is essential for clinicians to understand the vulnerability of plaques and the particular physiological conditions that lead to their mechanical failure. Our study focused on understanding the physical interaction between blood flow, plaque morphology and tissue properties to assess the plaque’s degree of vulnerability.


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