Vena cava inferior ligation in congestive heart failure

1955 ◽  
Vol 50 (1) ◽  
pp. 112-128 ◽  
Author(s):  
J. Bernath ◽  
R. Guillemot ◽  
P. Samuel ◽  
R.Heim de Balsac
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Vena Cava ◽  
Vena Cava Inferior

scholarly journals Efficacy of Transhepatic Anterior Approach to the Inferior Vena Cava for Malignant Pheochromocytoma Invading Right Posterior Lobe of the Liver: A Case Report

2019 ◽  
Vol 103 (1-2) ◽  
pp. 80-86
Author(s):  
Gen Tsujio ◽  
Kenjiro Kimura ◽  
Yukie Tauchi ◽  
Go Ohira ◽  
Ryosuke Amano ◽  
...  
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Anterior Approach ◽  
Inferior Vena ◽  
Vena Cava ◽  
Right Hepatectomy ◽  
Liver Hanging Maneuver ◽  
Hanging Maneuver ◽  
Right Lobe ◽  
The Right

Introduction: The anterior approach to the inferior vena cava (IVC) by the liver hanging maneuver is effective in resecting large retrohepatic tumors without mobilizing the right lobe. Case presentation: A 50-year-old man was referred to our hospital with a diagnosis of pheochromocytoma. He had severe congestive heart failure and cardiac ejection fraction was 15%. Abdominal magnetic resonance imaging (MRI) and ultrasonography (US) showed an adrenal mass about 80 mm in diameter. The tumor-infiltrated posterior segment of the right hepatic lobe and tumor were widely attached to the IVC. After treatment of congestive heart failure with conservative therapy, surgery was planned. Right adrenectomy and right hepatectomy were performed, the latter using the liver hanging maneuver to avoid mobilizing the right lobe, and we were able to minimize blood pressure fluctuations and perform the operation safely. The histopathologic diagnosis was malignant pheochromocytoma. Conclusions: We performed right hepatectomy without mobilizing the right lobe by the liver hanging maneuver and minimized stimulation of the tumor. We could perform the operation safely using the liver hanging maneuver, which seems effective in such cases.

Increased Excretion of Aldosterone in Urine From Dogs With Right-Sided Congestive Heart Failure and From Dogs With Thoracic Inferior Vena Cava Constriction

1956 ◽  
Vol 187 (1) ◽  
pp. 45-50 ◽  
Author(s):  
James O. Davis ◽  
M. Jay Goodkind ◽  
Maurice M. Pechet ◽  
Wilmot C. Ball
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Inferior Vena Cava ◽  
Cardiac Failure ◽  
Inferior Vena ◽  
Vena Cava ◽  
Bilateral Adrenalectomy ◽  
Chromatographic Fractionation

Extracts of urine from 11 normal dogs, 9 dogs with cardiac failure and 10 dogs with thoracic inferior vena cava constriction and ascites were assayed for aldosterone-like activity. The activity in urine from dogs with heart failure and from dogs with thoracic caval constriction was equivalent to 52 and 167 µg/day of DCA, respectively, and renal Na excretion was uniformly low. No activity was detected in 4 days' urine from normal dogs but chromatographic fractionation and assay of a 26-day collection of normal dog urine showed Na-retaining and increased K-excreting activity in the aldosterone fraction only. Following bilateral adrenalectomy and discontinuation of DCA therapy in 2 dogs with caval constriction, all urinary aldosterone-like activity disappeared and a natriuresis occurred. Assays of chromatographic fractions of extracts of urine from dogs with heart failure and from dogs with caval constriction revealed activity only in the aldosterone fraction.

Ascites Formation Without Sodium Intake in Dogs With Thoracic Inferior Vena Cava Constriction and in Dogs With Right-Sided Congestive Heart Failure

1957 ◽  
Vol 188 (3) ◽  
pp. 578-582 ◽  
Author(s):  
Wilmot C. Ball ◽  
James O. Davis ◽  
M. Jay Goodkind
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Inferior Vena Cava ◽  
Inferior Vena ◽  
Sodium Intake ◽  
Venous Pressure ◽  
Main Pulmonary Artery ◽  
Vena Cava ◽  
Edema Formation ◽  
Secondary Phenomenon

Dogs were subjected to constriction of the thoracic inferior vena cava or main pulmonary artery and were deprived of food for a period of 4 days thereafter. In association with a high venous pressure, 60% of the dogs formed ascites despite the absence of Na intake. Measurements of T-1824 dye space provided evidence that plasma volume was not increased. An increase in urinary excretion of aldosterone was associated with a markedly reduced urinary Na excretion. The data are interpreted as supporting the concept that elevated venous pressure initiates edema formation in congestive heart failure and that renal Na retention occurs as a secondary phenomenon.

Doppler Velocimetry in Superior Vena Cava Provides Useful Information on the Right Circulatory Function in Patients with Congestive Heart Failure

2001 ◽  
Vol 18 (6) ◽  
pp. 469-477 ◽  
Author(s):  
Stefano Ghio ◽  
Franco Recusani ◽  
Roberta Sebastiani ◽  
Catherine Klersy ◽  
Claudia Raineri ◽  
...  
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Superior Vena Cava ◽  
Superior Vena ◽  
Vena Cava ◽  
Doppler Velocimetry ◽  
The Right ◽  
Circulatory Function

Fulminante intrakardiale Thrombose bei Budd-Chiari-Syndrom und Thrombose der Vena cava inferior

VASA ◽  
2002 ◽  
Vol 31 (1) ◽  
pp. 62-65 ◽  
Author(s):  
F. Eckel ◽  
Huber ◽  
Heidecke ◽  
Moessmer ◽  
Berger ◽  
...  
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Hepatic Failure ◽  
Vena Cava ◽  
Factor V Leiden ◽  
Acute Hepatic Failure ◽  
Hepatic Veins ◽  
Factor V ◽  
Recombinant Hirudin ◽  
The Right

The most common cause of edema of the legs and dyspnea is congestive heart failure. Further differential diagnosis such as renal or hepatic failure have to be considered. We report the case of a previous healthy 65-year-old woman who developed dyspnea and massive edema of the legs followed by acute hepatic and renal failure. Imaging studies showed a thrombosis of the inferior vena cava (IVC) caused by a tumor between the right kidney and the IVC. Histological examination revealed a leiomyosarcoma of the IVC. Hepatic failure due to venous outflow obstruction (Budd-Chiari syndrome, BCS) was diagnosed. Coagulation profile showed a complex disorder due to acute hepatic failure. Factor V Leiden and prothrombin gene mutation G20210A could be excluded. The thrombosis extended from the femoral veins up to the right atrium. After 11 days of anticoagulation with heparin platelet counts decreased by more than 50%. Suspecting a heparin-induced thrombocytopenia the patient was placed on recombinant hirudin (lepirudin) for anticoagulation. Hepatic venogram showed a thrombosis of the hepatic vein orifices but not of the hepatic veins. The tumor and the thrombi were removed surgically. When the cardiopulmonary bypass was terminated new intracardiac thrombi occurred. Despite immediate surgical intervention the patient finally died due to right ventricular failure cuased by the fulminate intracardiac thrombosis. In conclusion, thrombosis of the IVC may mimic congestive heart failure and may cause BCS. Neoplasms and coagulation disorders may cause thrombosis of the IVC.

Sign in / Sign up

Export Citation Format

Share Document