Neurophysiological evaluation of the central nervous impulse propagation in patients with sensorimotor disturbances

1988 ◽  
Vol 70 (1) ◽  
pp. 16-25 ◽  
Author(s):  
Maria D. Caramia ◽  
Giorgio Bernardi ◽  
Flora Zarola ◽  
Paolo M. Rossini
JAMA ◽  
1966 ◽  
Vol 197 (1) ◽  
pp. 51b-51
Keyword(s):  

Nature ◽  
1964 ◽  
Vol 204 (4965) ◽  
pp. 1238-1238
Author(s):  
B. FRANKENHAEUSER
Keyword(s):  

1998 ◽  
Vol 62 (10) ◽  
pp. 760-764 ◽  
Author(s):  
Yoshinori Kobayashi ◽  
Yasushi Miyauchi ◽  
Naomi Kawaguchi ◽  
Kazuko Ohmura ◽  
Hirokazu Saitoh ◽  
...  

Glia ◽  
2010 ◽  
Vol 58 (13) ◽  
pp. 1544-1552 ◽  
Author(s):  
Yuan-Ting Sun ◽  
Thy-Sheng Lin ◽  
Shun-Fen Tzeng ◽  
Eric Delpire ◽  
Meng-Ru Shen

2001 ◽  
Vol 82 (6) ◽  
pp. 466-467
Author(s):  
R. G. Mustafina ◽  
F. G. Sitdikov ◽  
G. Kh. Samigullin

Intellectual efficiency parameters of pupils of the seventh eighth forms going in for sports and mathematics as well as pupils learning by standard program, are studied. The studies were carried out by measuring out the work performance, using the V.Y. Anfilov letter tables. The great lability of nervous processes in girls going in for sports and the favourable effect of motor loads on the organism of girls in improving the nervous impulse course process were shown. The data obtained confirms the necessity of differentiating loads not only by age and by education regime, but by sex as well, because the rate of changes in nervous processes in growing organism in children of both sexes is different.


2021 ◽  
Author(s):  
Tobias J Buscham ◽  
Maria A. Eichel-Vogel ◽  
Anna M Steyer ◽  
Olaf Jahn ◽  
Nicola Strenzke ◽  
...  

Oligodendrocytes facilitate rapid impulse propagation along the axons they myelinate and support their long-term integrity. However, the functional relevance of many myelin proteins has remained unknown. Here we find that expression of the tetraspan-transmembrane protein CMTM5 (Chemokine-like factor-like MARVEL-transmembrane domain containing protein 5) is highly enriched in oligodendrocytes and CNS myelin. Genetic disruption of the Cmtm5-gene in oligodendrocytes of mice does not impair the development or ultrastructure of CNS myelin. However, oligodendroglial Cmtm5-deficiency causes an early-onset progressive axonopathy, which we also observe in global and in tamoxifen-induced oligodendroglial Cmtm5-mutants. Presence of the Wlds mutation ameliorates the axonopathy, implying a Wallerian degeneration-like pathomechanism. These results indicate that CMTM5 is involved in the function of oligodendrocytes to maintain axonal integrity rather than myelin biogenesis.


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