Reaction of different parts of the cerebral vascular system in asphyxia

1967 ◽  
Vol 18 (2) ◽  
pp. 239-252 ◽  
Author(s):  
G.I. Mchedlishvili ◽  
L.G. Ormotsadze ◽  
L.S. Nikolaishvili ◽  
D.G. Baramidze
Keyword(s):  
Vascular System ◽  
Different Parts ◽  
Cerebral Vascular

Vascular Networks of the Periphery of the Fingernail

1995 ◽  
Vol 20 (4) ◽  
pp. 488-492 ◽  
Author(s):  
R. WOLFRAM-GABEL ◽  
H. SICK
Keyword(s):  
Vascular System ◽  
Vascular Network ◽  
Vascular Networks ◽  
Nail Bed ◽  
Sterile Part ◽  
Different Parts

The vascular networks of the periphery of the fingernail have been studied on fingers of adults, foetuses and neonates, by injecting the vascular system with gelatinous Indian ink. The nail is an avascular structure, partially covered by the nail wall. It is lying on the nail bed which is prolonged forwards with the hyponychium. Each part of the nail apparatus presents a characteristic vascular network derived from dorsal collaterals arising from the palmar digital vessels and from their arcades. These networks are papillary, pseudopapillary, reticular and subdermal. Their morphology and density vary according to their localization and correspond with histological variations of the different parts of the nail apparatus. Thus, the germinal part of the nail bed shows poor vascularization. It is responsible for the colour of the lunula. The sterile part and the hyponychium have dense vascular networks with glomus bodies.

Postmortem Angiography of Cerebral Vascular System

1962 ◽  
Vol 7 (6) ◽  
pp. 545-559 ◽  
Author(s):  
B. M. STEIN ◽  
W. F. McCORMICK ◽  
J. N. RODRIGUEZ ◽  
J. M. TAVERAS
Keyword(s):  
Vascular System ◽  
Postmortem Angiography ◽  
Cerebral Vascular

About cerebral vascular weakness

1934 ◽  
Vol 30 (2) ◽  
pp. 207-207
Author(s):  
Е. Kretschmer
Keyword(s):  
Vascular System ◽  
Cerebral Vascular

Kretschmer introduces a new concept of "cerebral vascular weakness" by analogy, or, as a. himself says, in the same sense in which they speak in relation to another part of the vascular system, "cardiac weakness".

scholarly journals Apaf-1, Bcl-xL, Cytochrome c, and Caspase-9 Form the Critical Elements for Cerebral Vascular Protection by Erythropoietin

2003 ◽  
Vol 23 (3) ◽  
pp. 320-330 ◽  
Author(s):  
Zhao Zhong Chong ◽  
Jing-Qiong Kang ◽  
Kenneth Maiese
Keyword(s):  
Membrane Potential ◽  
Cytochrome C ◽  
Mitochondrial Membrane Potential ◽  
Mitochondrial Membrane ◽  
Cell Injury ◽  
Vascular System ◽  
Dna Integrity ◽  
Vascular Protection ◽  
Caspase 9 ◽  
Cerebral Vascular

Erythropoietin (EPO) plays a prominent role in the regulation of the hematopoietic system, but the potential function of this trophic factor as a cytoprotectant in the cerebral vascular system is not known. The authors examined the ability of EPO to modulate a series of death-related cellular pathways during free radical–induced injury in cerebral microvascular endothelial cells (ECs). Endothelial cell injury was evaluated by trypan blue, DNA fragmentation, membrane phosphatidylserine exposure, apoptotic protease–activating factor-1 (Apaf-1), and Bcl-xL expression, mitochondrial membrane potential, cytochrome c release, and cysteine protease activity. They show that constitutive EPO is present in ECs but is insufficient to prevent cellular injury. Signaling through the EPO receptor, however, remains biologically responsive to exogenous EPO administration to offer significant protection against nitric oxide–induced injury. Exogenous EPO maintains both genomic DNA integrity and cellular membrane asymmetry through parallel pathways that prevent the induction of Apaf-1 and preserve mitochondrial membrane potential in conjunction with enhanced Bcl-xL expression. Consistent with the modulation of Apaf-1 and the release of cytochrome c, EPO also inhibits the activation of caspase-9 and caspase-3–like activities. Identification of novel cytoprotective pathways used by EPO may serve as therapeutic targets for cerebral vascular disease.

scholarly journals Left atrial myxoma and transient ischemic attacks: case report

2021 ◽  
Author(s):  
Leonardo Daniel Reis Santos ◽  
Omar Pereira de Almeida Neto ◽  
Michelle Franco Macedo de Lima ◽  
Nathália Varano
Keyword(s):  
Case Report ◽  
Left Atrial ◽  
Vascular System ◽  
Atrial Myxoma ◽  
Cardiac Tumors ◽  
Transient Ischemic Attacks ◽  
Adequate Treatment ◽  
Left Atrial Myxoma ◽  
History Of ◽  
Cerebral Vascular

Context: Cardiac tumors are rare and myxomas are the most prevalent between them. Although histologically benign, they may cause severe effects given their intracardiac location. Unspecific symptoms compromise the diagnosis, leading to complications such as changes in the cerebral vascular dynamics. Case report following the CARE guidelines. Case report: A 62-year-old woman was admitted to a high complexity hospital in Minas Gerais, with history of unstable angina, aphasia, right hemiparesis, dysarthria, claiming precordialgia with strong intensity. Complained hyporexia and weight loss during the last month. Medical history of 7 transient ischemic attacks (TIA) in the last two years. Physical examination with no abnormalities. Chest x-ray and transesophageal echocardiogram showed bilateral neovascularization and 4.9 x 2.9 cm dimension mass in the left atrium. Coronary angiography revealed proximal calcification and atherosclerotic plaque occluding 40% of the flux in the middle third of the anterior descending artery, pointing to the coexistence of coronary disease and left atrial myxoma. The occurrence of a TIA was determined and the prescription of an anticoagulant to avoid future embolic events. A surgical approach was necessary. Biopsy concluded myxoid and hyaline-rich stroma tumor, evident vascular system, star-shaped cells isolated or forming small groups, confirming myxoma diagnosis. After a ten-day hospitalization, the patient was clinically stable, and was discharged after health education. Conclusion: Cardiac tumors such as myxomas lead to important cerebral vascular consequences, so that the clinical investigation is essential to the differential diagnosis between a stroke and the TIA, to provide adequate treatment and disease prevention.

scholarly journals Presence of Pseudomonas syringae pv actinidiae the causal agent of bacterial canker of kiwifruit on symptomatic and asymptomatic tissues of kiwifruit

2011 ◽  
Vol 64 ◽  
pp. 241-245 ◽  
Author(s):  
J.L. Vanneste ◽  
J. Yu ◽  
D.A. Cornish ◽  
S. Max ◽  
G. Clark
Keyword(s):  
Pseudomonas Syringae ◽  
Vascular System ◽  
Causal Agent ◽  
Bacterial Canker ◽  
Effective Strategy ◽  
Bacterial Isolation ◽  
Chain Reaction ◽  
Leaf Spots ◽  
Polymerase Chain ◽  
Different Parts

Pseudomonas syringae pv actinidiae (Psa) the causal agent of bacterial canker of kiwifruit has recently been found in New Zealand This pathogen has been the object of few studies and little is known about its epidemiology Yet the development of an effective strategy of control requires an understanding of the life cycle of the pathogen in particular determining the location of the bacteria in tissues In this study the presence of Psa on flowers symptomatic and asymptomatic leaves and different parts of canes showing symptoms was determined by polymerase chain reaction (PCR) or by direct bacterial isolation Psa was found associated with angular necrotic leaf spots with white exudate and in canes In canes Psa seemed to be either in the parenchyma leading to production of exudate or in the vascular system leading to wilting Psa was also found on asymptomatic tissues where it was probably surviving as an epiphyte

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