Altered fatty acid metabolism in patients with angiographically documented coronary artery disease

Metabolism ◽  
1994 ◽  
Vol 43 (8) ◽  
pp. 982-993 ◽  
Author(s):  
Edward N. Siguel ◽  
Robert H. Lerman
Keyword(s):  
Coronary Artery Disease ◽  
Fatty Acid ◽  
Coronary Artery ◽  
Fatty Acid Metabolism ◽  
Acid Metabolism ◽  
Artery Disease ◽  
Documented Coronary Artery Disease

W12-P-006 The impact of abnormalities of fatty acid metabolism in nonagenarians with coronary artery disease

2005 ◽  
Vol 6 (1) ◽  
pp. 62-63
Author(s):  
V. Bláha ◽  
D. Solichová ◽  
D. Cenohorský ◽  
R. Hyšpler ◽  
P. Vyroubal ◽  
...  
Keyword(s):  
Coronary Artery Disease ◽  
Fatty Acid ◽  
Coronary Artery ◽  
Fatty Acid Metabolism ◽  
Acid Metabolism ◽  
Artery Disease ◽  
The Impact

Perturbations in fatty acid metabolism and apoptosis are manifested in calcific coronary artery disease: An exploratory lipidomic study

2015 ◽  
Vol 197 ◽  
pp. 192-199 ◽  
Author(s):  
Panagiotis A. Vorkas ◽  
Giorgis Isaac ◽  
Anders Holmgren ◽  
Elizabeth J. Want ◽  
John P. Shockcor ◽  
...  
Keyword(s):  
Coronary Artery Disease ◽  
Fatty Acid ◽  
Coronary Artery ◽  
Fatty Acid Metabolism ◽  
Acid Metabolism ◽  
Artery Disease

Fatty Acid Metabolism in Symptomatic Patients with Mitral Valve Prolapse but without Coronary Artery Disease - Comparison with 201 Tl Myocardial Perfusion Scintigraphy

1987 ◽  
Vol 26 (04) ◽  
pp. 172-176 ◽  
Author(s):  
H. Schicha ◽  
U. Tebbei ◽  
P. Neumann ◽  
D. Emrich ◽  
E. Voth
Keyword(s):  
Coronary Artery Disease ◽  
Fatty Acid ◽  
Coronary Artery ◽  
Mitral Valve ◽  
Acid Metabolism ◽  
Myocardial Perfusion Scintigraphy ◽  
Control Group ◽  
Perfusion Scintigraphy ◽  
Elimination Half ◽  
Artery Disease

Using 123l-ω-heptadecanoic acid (HDA) and 201TI, respectively, myocardial fatty acid metabolism and perfusion were studied in 51 symptomatic patients with mitral valve prolapse (MVP) as diagnosed by ventriculography, and no evidence of coronary artery disease. Twelve subjects with normal coronary arteries and normal ventriculogram served as a control group for the evaluation of elimination kinetics of HDA. In the control group, the mean elimination halflife was 26.1 ± 3.6 min, whereas the patients with MVP had a mean value of ± 6.4 min. In patients with MVP, a high incidence concerning abnormalities of accumulation and/or elimination of HDA occurred, namely accumulation defects in 31 % and both prolonged and shortened elimination half-lives in 16% and 29%, respectively. Myocardial perfusion scintigraphy using 201TI showed abnormalities in 76%. Correlations were found between decreased uptake of HDA and prolonged elimination half-life as well as defects by 201TI, presumably due to ischemia based on small-vessel disease or abnormalities of cellular metabolism.

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