Mechanisms of CNP following SCI: Chemokines in neuronal-glial cell interaction

2022 ◽  
pp. 315-338
Author(s):  
Friederike Knerlich-Lukoschus
Keyword(s):  
2016 ◽  
Vol 74 (10) ◽  
pp. 849-854
Author(s):  
Paulo Victor Sgobbi de Souza ◽  
Wladimir Bocca Vieira de Rezende Pinto ◽  
Flávio Moura Rezende Filho ◽  
Acary Souza Bulle Oliveira

ABSTRACT Motor neuron disease is one of the major groups of neurodegenerative diseases, mainly represented by amyotrophic lateral sclerosis. Despite wide genetic and biochemical data regarding its pathophysiological mechanisms, motor neuron disease develops under a complex network of mechanisms not restricted to the unique functions of the alpha motor neurons but which actually involve diverse functions of glial cell interaction. This review aims to expose some of the leading roles of glial cells in the physiological mechanisms of neuron-glial cell interactions and the mechanisms related to motor neuron survival linked to glial cell functions.


Pathogens ◽  
2016 ◽  
Vol 5 (2) ◽  
pp. 43 ◽  
Author(s):  
Jana Seele ◽  
Roland Nau ◽  
Chittappen Prajeeth ◽  
Martin Stangel ◽  
Peter Valentin-Weigand ◽  
...  

Intervirology ◽  
1983 ◽  
Vol 19 (2) ◽  
pp. 95-104 ◽  
Author(s):  
George J. Dawson ◽  
Teresita S. Elizan ◽  
Solomon L. Mowshowitz ◽  
Rochelle Cohen

2017 ◽  
Vol 74 (18) ◽  
pp. 3275-3291 ◽  
Author(s):  
Zhi-Jun Zhang ◽  
Bao-Chun Jiang ◽  
Yong-Jing Gao

Author(s):  
A. E. Ritchie

The cause of bluecomb disease in turkeys is unknown. Filtration of infective intestinal contents suggests a viral origin. To date, it has not been possible to isolate the etiologic agent in various cell cultures. The purpose of this work was to characterize as many virus-like entities as were recognizable in intestines of both healthy and bluecomb-infected turkeys. By a comparison of the viral populations it was hoped that some insight might be gained into the cause of this disease. Studies of turkey hemorraghic enteritis by Gross and Moore (Avian Dis. 11: 296-307, 1967) have suggested that a bacteriophage-host cell interaction may bear some causal relationship to that disease.


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