Symptomatic Viral Infection is Associated with Impaired Response to Treatment in Children with Acute Asthma

2012 ◽  
Vol 160 (1) ◽  
pp. 82-87 ◽  
Author(s):  
Kristina Rueter ◽  
Joelene Bizzintino ◽  
Andrew C. Martin ◽  
Guicheng Zhang ◽  
Catherine M. Hayden ◽  
...  
Keyword(s):  
Viral Infection ◽  
Acute Asthma ◽  
Response To Treatment

scholarly journals The Effect of Viral Infection on Exhaled Nitric Oxide in Children with Acute Asthma Exacerbations

2015 ◽  
Vol 3 (6) ◽  
pp. 913-919 ◽  
Author(s):  
Jonathan Malka ◽  
Ronina Covar ◽  
Anna Faino ◽  
Jennifer Fish ◽  
Paige Pickering ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Viral Infection ◽  
Acute Asthma ◽  
Exhaled Nitric Oxide ◽  
Asthma Exacerbations ◽  
Acute Asthma Exacerbations

scholarly journals Hopkins’ syndrome

2011 ◽  
Vol 5 (2) ◽  
pp. 69-71
Author(s):  
Maria Roberta Longo ◽  
Raffaele Falsaperla ◽  
Catia Romano ◽  
Eleonora Passaniti ◽  
Piero Pavone
Keyword(s):  
Spinal Cord ◽  
Viral Infection ◽  
Rare Disease ◽  
Corticosteroid Therapy ◽  
Muscle Atrophy ◽  
Acute Asthma ◽  
Atopic Disease ◽  
Asthma Attack ◽  
Immunological Suppression ◽  
Asthma In Children

Hopkins’ syndrome is a rare disease that affects the anterior horn of the spinal cord after an acute episode of asthma in children with atopic disease. A viral infection or immunological suppression in atopic subjects might be the cause of occurrence of this syndrome, although the mechanism due to the etiopathogenesis of the disease still remains unknown.In general, this disease is manifested by a few days to a few weeks after an acute asthma attack, with flaccid paralysis of one or more limbs and in some cases residual muscle atrophy. The response to corticosteroid therapy is good and rare the possibility of recurrence.

Virus infection reduces theophylline metabolism in acute asthma: Increased lymphoid MxA protein as a marker for viral infection

2003 ◽  
Vol 111 (2) ◽  
pp. S145
Author(s):  
T. Itazawa ◽  
Y. Adachi ◽  
Y.S. Adachi ◽  
J. Yamamoto ◽  
M. Hamamichi ◽  
...  
Keyword(s):  
Virus Infection ◽  
Viral Infection ◽  
Acute Asthma ◽  
Theophylline Metabolism

Theophylline metabolism in acute asthma with MxA-indicated viral infection

2006 ◽  
Vol 48 (1) ◽  
pp. 54-57 ◽  
Author(s):  
TOSHIKO ITAZAWA ◽  
YUICHI ADACHI ◽  
MOTOKAZU NAKABAYASHI ◽  
TATSUYA FUCHIZAWA ◽  
GYOKEI MURAKAMI ◽  
...  
Keyword(s):  
Viral Infection ◽  
Acute Asthma ◽  
Theophylline Metabolism

Epstein-Barr viral infection in extranodal lymphoma of the head and neck: correlation with prognosis and response to treatment

2006 ◽  
Vol 48 (5) ◽  
pp. 516-528 ◽  
Author(s):  
A A Bahnassy ◽  
A-R N Zekri ◽  
N Asaad ◽  
S El-Houssini ◽  
H M Khalid ◽  
...  
Keyword(s):  
Viral Infection ◽  
Head And Neck ◽  
Extranodal Lymphoma ◽  
Response To Treatment ◽  
Epstein Barr

Effects of the pseudomonad phytotoxin coronatine on tomato leaf structure and ultrastructure

1995 ◽  
Vol 53 ◽  
pp. 862-863
Author(s):  
D.A. Palmer ◽  
C.L. Bender
Keyword(s):  
Pseudomonas Syringae ◽  
Membrane Integrity ◽  
Leaf Tissue ◽  
Cell Number ◽  
Cell Ultrastructure ◽  
Response To Treatment ◽  
Prominent Symptom ◽  
Chlorophylls A And B ◽  
Cell Dimensions ◽  
Structure And Ultrastructure

Coronatine is a non-host-specific phytotoxin produced by several members of the Pseudomonas syringae group of pathovars. The toxin acts as a virulence factor in P. syringae pv. tomato, allowing the organism to multiply to a higher population density and develop larger lesions than mutant strains unable to produce the toxin. The most prominent symptom observed in leaf tissue treated with coronatine is an intense spreading chlorosis; this has been attributed to a loss of chlorophylls a and b in tobacco. Coronatine's effects on membrane integrity and cell ultrastructure have not been previously investigated. The present study describes changes in tomato leaves in response to treatment with purified coronatine, infection by a coronatine-producing strain of P. syringae pv. tomato, and infection by a cor" mutant.In contrast to H2O-treated tissue, coronatine-treated tissue showed a diffuse chlorosis extending approximately 5 mm from the inoculation site. Leaf thickness, cell number, and cell dimensions were similar for both healthy and coronatine-treated, chlorotic tissue; however, the epidermal cell walls were consistently thicker in coronatine-treated leaves (Figs, la and lb).

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