Domoic acid induced spinal cord lesions in adult mice: Evidence for the possible molecular pathways of excitatory amino acids in spinal cord lesions

2008 ◽  
Vol 29 (4) ◽  
pp. 700-707 ◽  
Author(s):  
Renshi Xu ◽  
Yuhui Tao ◽  
Chengsi Wu ◽  
Juan Yi ◽  
Yunzhu Yang ◽  
...  
Keyword(s):  
Amino Acids ◽  
Spinal Cord ◽  
Excitatory Amino Acids ◽  
Domoic Acid ◽  
Molecular Pathways ◽  
Spinal Cord Lesions ◽  
Adult Mice

The Role of Excitatory Amino Acids in Hypothermic Injury to Mammalian Spinal Cord Neurons

1996 ◽  
Vol 13 (12) ◽  
pp. 809-818 ◽  
Author(s):  
GEERT CRAENEN ◽  
SRDIJA JEFTINIJA ◽  
IVETA GRANTS ◽  
JEN HILL LUCAS
Keyword(s):  
Amino Acids ◽  
Spinal Cord ◽  
Excitatory Amino Acids ◽  
Spinal Cord Neurons ◽  
Hypothermic Injury

Excitatory amino acids rise to toxic levels upon impact injury to the rat spinal cord

1991 ◽  
Vol 547 (2) ◽  
pp. 344-348 ◽  
Author(s):  
Danxia Liu ◽  
Wipawan Thangnipon ◽  
David J. McAdoo
Keyword(s):  
Amino Acids ◽  
Spinal Cord ◽  
Excitatory Amino Acids ◽  
Rat Spinal Cord ◽  
Impact Injury

Systemic Administration of Domoic Acid-Induced Spinal Cord Lesions in Neonatal Rats

2000 ◽  
Vol 23 (1) ◽  
pp. 31-39 ◽  
Author(s):  
Guang H. Jian Wang ◽  
Larry C. Schmued ◽  
Annette M. Andrews ◽  
Andrew C. Scallet ◽  
William H. Slikker ◽  
...  
Keyword(s):  
Spinal Cord ◽  
Domoic Acid ◽  
Systemic Administration ◽  
Neonatal Rats ◽  
Spinal Cord Lesions

The postexcitatory effects of acidic amino acids on spinal neurones

1981 ◽  
Vol 59 (3) ◽  
pp. 239-243 ◽  
Author(s):  
H. McLennan ◽  
Jin-Rong Liu
Keyword(s):  
Amino Acids ◽  
Spinal Cord ◽  
Excitatory Amino Acids ◽  
Aminobutyric Acid ◽  
Renshaw Cells ◽  
Acidic Amino Acids ◽  
Inhibitory Compounds ◽  
Spinal Neurones

The excitation of some neurones in the spinal cord of rats by L-glutamate and L-aspartate is followed by a period of reduced excitability. This effect is not observed after excitation by D-glutamate nor, in the case of Renshaw cells, by acetylcholine. The depressions following L-glutamate were reduced by bicuculline and those after aspartate by strychnine, suggesting that they may have been caused through decarboxylation of the excitatory amino acids to yield the inhibitory compounds γ-aminobutyric acid and β-alanine, respectively.

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