A Simple Model for Carcinogenesis of Colorectal Cancers with Microsatellite Instability

1999 ◽  
pp. 189-221 ◽  
Author(s):  
Nicolas Janin
2001 ◽  
Vol 120 (5) ◽  
pp. A295-A295
Author(s):  
D CHANG ◽  
A GOEL ◽  
L RICCIARDIELLO ◽  
C ARNOLD ◽  
C BOLAND

2014 ◽  
Vol 146 (5) ◽  
pp. S-628
Author(s):  
Young Baek Kim ◽  
Sun-Young Lee ◽  
Jeong Hwan Kim ◽  
In Kyung Sung ◽  
Hyung Seok Park ◽  
...  

2011 ◽  
Vol 459 (1) ◽  
pp. 55-63 ◽  
Author(s):  
Jeong Mo Bae ◽  
Mi Jung Kim ◽  
Jung Ho Kim ◽  
Jae Moon Koh ◽  
Nam-Yun Cho ◽  
...  

2013 ◽  
Vol 145 (3) ◽  
pp. 540-543.e22 ◽  
Author(s):  
Alexandra E. Gylfe ◽  
Johanna Kondelin ◽  
Mikko Turunen ◽  
Heikki Ristolainen ◽  
Riku Katainen ◽  
...  

2016 ◽  
Vol 29 (04) ◽  
pp. 336-344 ◽  
Author(s):  
Jean Ashburn ◽  
Thomas Plesec ◽  
Matthew Kalady

AbstractColorectal serrated polyps are intermediate lesions in the serrated neoplastic pathway, which account for up to 30% of colorectal cancers. This pathway is biologically distinct from the adenoma-to-carcinoma sequence, with associated cancers exhibiting mutations in the BRAF oncogene, DNA promoter hypermethylation, and microsatellite instability. An evolving understanding of these unique lesions has led to the development of a more accurate classification, improved endoscopic identification, and tailored clinical management guidelines. This article reviews serrated polyps and serrated polyposis syndrome.


Apmis ◽  
2014 ◽  
Vol 123 (4) ◽  
pp. 359-360 ◽  
Author(s):  
Eun Mi Je ◽  
Youn Jin Choi ◽  
Yeun Jun Chung ◽  
Nam Jin Yoo ◽  
Sug Hyung Lee

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