Staged management of Budd-Chiari syndrome caused by co-obstruction of the inferior vena cava and main hepatic veins

2013 ◽  
Vol 12 (3) ◽  
pp. 278-285 ◽  
Author(s):  
Yu-Ling Sun ◽  
Yang Fu ◽  
Lin Zhou ◽  
Xiu-Xian Ma ◽  
Zhi-Wei Wang ◽  
...  
Keyword(s):  
Inferior Vena Cava ◽  
Inferior Vena ◽  
Vena Cava ◽  
Hepatic Veins ◽  
Budd Chiari Syndrome ◽  
Chiari Syndrome

Recanalization and Reconstruction of a Chronically Occluded Inferior Vena Cava Through an Existing Transjugular Intrahepatic Portosystemic Shunt in the Setting of Budd-Chiari Syndrome

2021 ◽  
pp. 153857442110020
Author(s):  
Reza Talaie ◽  
Hamed Jalaeian ◽  
Nassir Rostambeigi ◽  
Anthony Spano ◽  
Jafar Golzarian
Keyword(s):  
Inferior Vena Cava ◽  
Transjugular Intrahepatic Portosystemic Shunt ◽  
Inferior Vena ◽  
Vena Cava ◽  
Portosystemic Shunt ◽  
Hepatic Veins ◽  
Budd Chiari Syndrome ◽  
Chiari Syndrome ◽  
Intrahepatic Portosystemic Shunt ◽  
The Right

Budd-Chiari syndrome (BCS) results from the occlusion or flow reduction in the hepatic veins or inferior vena cava and can be treated with transjugular intrahepatic portosystemic shunt when hepatic vein recanalization fails.1-3 Hypercoagulable patients with primary BCS are predisposed to development of new areas of thrombosis within the TIPS shunt or IVC. This case details a patient with BCS, pre-existing TIPS extending to the right atrium, and chronic retrohepatic IVC thrombosis who underwent sharp recanalization of the IVC with stenting into the TIPS stent bridging the patient until his subsequent hepatic transplantation.

Numerical Study of Blood Flow in the Hepatic Portion of the Inferior Vena Cava

2007 ◽  
Author(s):  
M. Matsubara ◽  
M. Watanabe ◽  
S. Watanabe ◽  
K. Konishi ◽  
S. Yamaguchi ◽  
...  
Keyword(s):  
Blood Flow ◽  
Inferior Vena Cava ◽  
Vessel Wall ◽  
Inferior Vena ◽  
Numerical Study ◽  
Vena Cava ◽  
Hepatic Veins ◽  
Partial Occlusion ◽  
Budd Chiari Syndrome ◽  
Chiari Syndrome

Budd-Chiari syndrome (BCS) is complete or partial occlusion in the hepatic veins and the hepatic portion of the inferior vena cava (IVC). The cause of BCS is not well known yet, however: abnormal vessel wall shear stress caused by blood flow is thought to increase the likelihood of developing BCS. In order to reveal the formation mechanism of BCS, we construct several vessel models of the IVC and hepatic veins from medical images and study the characteristics of the blood flow in the vicinity of the junctions of the hepatic veins with the IVC numerically.

scholarly journals Budd Chiari syndrome due to inferior vena cava web complicating pregnancy in patient with bad obstetric history: an interesting case

2019 ◽  
Vol 8 (11) ◽  
pp. 4617
Author(s):  
Richa Subhash Udhwani ◽  
Michelle Nalini Fonseca ◽  
Deepali Swapnil Kapote
Keyword(s):  
Inferior Vena Cava ◽  
Inferior Vena ◽  
Vena Cava ◽  
Rare Condition ◽  
Interesting Case ◽  
Radiology Department ◽  
Hepatic Veins ◽  
Endoscopic Variceal Ligation ◽  
Budd Chiari Syndrome ◽  
Chiari Syndrome

Presenting an unusual case of 27 years old female who presented at 18 weeks of pregnancy with second trimester bleeding per vaginum. Patient had history of recurrent abortions on examination was found to have hypertension and thrombocytopenia. Usg done revealed severe oligohydramnios. Patient was managed conservatively but aborted spontaneously at 22 weeks of gestation. Post-abortionl on day 2 patient developed abdominal distension and liver function tests were found to be deranged. USG and CT abdomen and pelvis was done, which revealed Budd chiari syndrome due to inferior vena cava (IVC) web. This extremely rare condition is characterized by obstruction of inferior vena cava by membrane or fibrous band. This condition is diagnosed by radiological techniques which in our patient revealed classical findings of caudate lobe hypertrophy, non-visualization of hepatic veins, moderate hepatomegaly and spleenomegaly and multiple collaterals. Esophagogastroduodenoscopy done which revealed large varieces for which endoscopic variceal ligation was done. IVCgram and IVC plasty was done by interventional radiology department 6 weeks after abortion. The aim of this case report is to highlight an extremely rare cause of Budd Chiari syndrome and IVC web in patient with recurrent abortion with spleenomegly leading to thrombocytopenia. It is important to rule out other differential diagnosis in these patients like APLA, ITP.

scholarly journals Hydatid Cyst of Liver: A Rare Cause of Secondary Budd-Chiari Syndrome

2013 ◽  
Vol 47 (3) ◽  
pp. 159-161 ◽  
Author(s):  
Mandeep Kang ◽  
Rajesh Gupta ◽  
Divyesh Mahajan
Keyword(s):  
Early Diagnosis ◽  
Inferior Vena Cava ◽  
Hydatid Cyst ◽  
Hydatid Disease ◽  
Inferior Vena ◽  
Vena Cava ◽  
Hepatic Veins ◽  
Budd Chiari Syndrome ◽  
Chiari Syndrome

ABSTRACT Hydatid cyst of the liver, causing compression of the inferior vena cava (IVC) and hepatic veins is a rare cause of secondary Budd-Chiari syndrome (BCS). As the hydatid disease is endemic in India, it is a rare but treatable cause of BCS. The early diagnosis and timely intervention can prevent hepatic complications leading to BCS in the affected patients. How to cite this article Mahajan D, Kang M, Gupta R, Khandelwal N. Hydatid Cyst of Liver: A Rare Cause of Secondary Budd-Chiari Syndrome. J Postgrad Med Edu Res 2013;47(3): 159-161.

scholarly journals Budd-Chiari Syndrome

2017 ◽  
Vol 118 (2-3) ◽  
pp. 69-80 ◽  
Author(s):  
Tomáš Grus ◽  
Lukáš Lambert ◽  
Gabriela Grusová ◽  
Rohan Banerjee ◽  
Andrea Burgetová
Keyword(s):  
Inferior Vena Cava ◽  
Structural Changes ◽  
Inferior Vena ◽  
Survival Rates ◽  
Vena Cava ◽  
Hepatic Veins ◽  
Budd Chiari Syndrome ◽  
Venous Outflow ◽  
Secondary Pathology ◽  
Chiari Syndrome

Budd-Chiari syndrome (BCS) is a rare disease with an incidence of 0.1 to 10 per million inhabitants a year caused by impaired venous outflow from the liver mostly at the level of hepatic veins and inferior vena cava. Etiological factors include hypercoagulable conditions, myeloprolipherative diseases, anatomical variability of the inferior vena cava, and environmental conditions. Survival rates in treated patients range from 42 to 100% depending on the etiology and the presence of risk factors including parameters of Child-Pugh score, sodium and creatinine plasma levels, and the choice of treatment. Without treatment, 90% of patients die within 3 years, mostly due to complications of liver cirrhosis. BCS can be classified according to etiology (primary, secondary), clinical course (acute, chronic, acute or chronic lesion), and morphology (truncal, radicular, and venooclusive type). The diagnosis is established by demonstrating obstruction of the venous outflow and structural changes of the liver, portal venous system, or a secondary pathology by ultrasound, computed tomography, or magnetic resonance. Laboratory and hematological tests are an integral part of the comprehensive workup and are invaluable in recognizing hematological and coagulation disorders that may be identified in up to 75% of patients with BCS. The recommended therapeutic approach to BCS is based on a stepwise algorithm beginning with medical treatment (a consensus of expert opinion recommends anticoagulation in all patients), endovascular treatment to restore vessel patency (angioplasty, stenting, and local thrombolysis), placement of transjugular portosystemic shunt (TIPS), and orthotopic liver transplantation as a last resort rescue treatment.

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