scholarly journals Erratum: Nitrate flux in the Mississippi River

Nature ◽  
2001 ◽  
Vol 414 (6865) ◽  
pp. 710-710
Nature ◽  
2001 ◽  
Vol 414 (6860) ◽  
pp. 166-167 ◽  
Author(s):  
Gregory F. McIsaac ◽  
Mark B. David ◽  
George Z. Gertner ◽  
Donald A. Goolsby

2002 ◽  
Vol 31 (5) ◽  
pp. 1610-1622 ◽  
Author(s):  
Gregory F. McIsaac ◽  
Mark B. David ◽  
George Z. Gertner ◽  
Donald A. Goolsby

2004 ◽  
Vol 61 (7) ◽  
pp. 1102-1112 ◽  
Author(s):  
William B Richardson ◽  
Eric A Strauss ◽  
Lynn A Bartsch ◽  
Emy M Monroe ◽  
Jennifer C Cavanaugh ◽  
...  

We evaluated patterns of denitrification and factors effecting denitrification in the upper Mississippi River. Measurements were taken over 2 years, during which river discharge ranged from record flooding to base flow conditions. Over the period of study, average denitrification enzyme activity was highest in backwater lakes and lowest in the main channel. Throughout the study reach, highest denitrification enzyme activity occurred during fall and lowest occurred in winter. Rates during spring floods (2001) were only slightly higher than during the preceding winter. Mean unamended denitrification rates ranged from 0.02 (fall 2001 in backwaters) to 0.40 µg N·cm–2·h–1 (spring 2001 in backwaters). Laboratory experiments showed that denitrification rates increased significantly with addition of NO3– regardless of sediment C content, while rates increased little with addition of labile C (glucose). Denitrification in this reach of the upper Mississippi River appears to be NO3– limited throughout the growing season and the delivery of NO3– is strongly controlled by river discharge and hydrologic connectivity across the floodplain. We estimate that denitrification removes 6939 t N·year–1 or 6.9% of the total annual NO3– input to the reach. Hydrologic connectivity and resultant NO3– delivery to high-C sediments is a critical determinant of reach-scale processing of N in this floodplain system.


Author(s):  
Anthony Paparo ◽  
Judy A. Murphy ◽  
Robert Dean

In the mid-1950's, fingernail clams virtually disappeared from a 100-mile section of the IL River, a tributary of the Mississippi River, due to unknown causes. A survey of the bottom fauna of the IL River in 1979, revealed that the clams were still absent from the middle reach of the River, where they had been abundant prior to the die-off in the 1950's. Some factor(s) in the River currently prevent the clams from recolonizing areas where they were formerly abundant. Recently, clams exposed to fluoride developed abnormal grooves in the shell matrix. Fluorides are known to be protoplasmic poisons removing essential body calcium by precipitation. Since the shell consists primarily of Ca carbonate, this investigation examines the possible role of fluoride on shell formation and the poisoning of the Ca pump which can directly inhibit lateral ciliary activity on the gill.


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