scholarly journals Ephemeral collision complexes mediate chemically termolecular transformations that affect system chemistry

2017 ◽  
Vol 9 (11) ◽  
pp. 1078-1082 ◽  
Author(s):  
Michael P. Burke ◽  
Stephen J. Klippenstein
2004 ◽  
Vol 120 (16) ◽  
pp. 7245-7248 ◽  
Author(s):  
Sarah M. Harper ◽  
Sunny W.-P. Hu ◽  
Stephen D. Price

2019 ◽  
Vol 11 (25) ◽  
pp. 96-102
Author(s):  
Sugini
Keyword(s):  
Total P ◽  

Latar Belakang : Asupan tinggi energi dari lemak jenuh meningkatkan risiko CHD khususnya pada wanita. Kelainan profil lipid ini erat hubungannya dengan proses perkembangan atherosklerosis. Penelitian epidemiologi, laboratorium dan klinik menunjukkan hubungan peningkatan kejadian penyakit kardiovaskular dan penyakit degeneratif lainnya dengan kadar kolesterol yang tinggi. Metode : Rancangan Penelitian adalah crossecional. Subyek penelitian adalah 30 karyawati RSUD Kendal umur 35 – 50 tahun.Pemeriksaan kadar kolesterol total, kadar kolesterol LDL diukur dengan vitros system chemistry analyser 300 di laboratorium patologi klinik RSUD Kendal. Asupan gizi diukur dengan metode food recall 24 dihitung dengan program Nutrisurvey. Analisis data menggunakan uji Shapiro Wilk dan uji korelasi Spearman. Hasil dan Pembahasan : Hasil analisa data adalah ada hubungan asupan energi dengan kadar kolesterol total (p=0,016, r=0,434), ada hubungan kuat asupan lemak dengan kadar kolesterol total (p=0,01, r=0,593), ada hubungan asupan lemak dengan kadar LDL (p=0,011, r=0,456) dan tidak ada hubungan asupan energi dengan kadar LDL (p=0,1119, r=0,291), Asupan tinggi energi mengakibatkan penimbunan lemak terutama trigliserida. Hal ini akan meningkatkan VLDL dan IDL darah yang akan berujung dengan peningkatan kolesterol total. Semakin tinggi seseorang mengkonsumsi makanan berlemak, maka timbunan kadar lemak dan trigliserida di dalam tubuh akan meningkat. Hal ini akan meningkatkan VLDL dan IDL darah. Simpulan : Ada hubungan asupan energi dengan kadar kolesterol total (p=0,016, r=0,434), ada hubungan kuat asupan lemak dengan kadar kolesterol total (p=0,01, r=0,593) dan kadar LDL (p=0,011, r=0,456).


1969 ◽  
Vol 3 (5) ◽  
pp. 323-326 ◽  
Author(s):  
J.V. Dugan ◽  
J.H. Rice ◽  
J.L. Magee

1978 ◽  
Vol 2 (3) ◽  
pp. 153-181 ◽  
Author(s):  
G.C.W. Comley
Keyword(s):  

1979 ◽  
Vol 83 (13) ◽  
pp. 1766-1770 ◽  
Author(s):  
Salvatore. Andini ◽  
Luciano. Ferrara ◽  
Piero A. Temussi ◽  
Francesco. Lelj ◽  
Teodorico. Tancredi

2005 ◽  
Vol 16 (11) ◽  
pp. 5400-5409 ◽  
Author(s):  
Wei Cui ◽  
Lisa R. Sproul ◽  
Susan M. Gustafson ◽  
Heinrich J.G. Matthies ◽  
Susan P. Gilbert ◽  
...  

Nod, a nonmotile kinesinlike protein, plays a critical role in segregating achiasmate chromosomes during female meiosis. In addition to localizing to oocyte chromosomes, we show that functional full-length Nod-GFP (NodFL-GFP) localizes to the posterior pole of the oocyte at stages 9–10A, as does kinesin heavy chain (KHC), a plus end-directed motor. This posterior localization is abolished in grk mutants that no longer maintain the microtubule (MT) gradient in the oocyte. To test the hypothesis that Nod binds to the plus ends of MTs, we expressed and purified both full-length Nod (NodFL) and a truncated form of Nod containing only the motorlike domain (Nod318) from Escherichia coli and assessed their interactions with MTs in vitro. Both NodFL and Nod318 demonstrate preferential binding to the ends of the MTs, displaying a strong preference for binding to the plus ends. When Nod318-GFP:MT collision complexes were trapped by glutaraldehyde fixation, the preference for binding to plus ends versus minus ends was 17:1. NodFL and Nod318 also promote MT polymerization in vitro in a time-dependent manner. The observation that Nod is preferentially localized to the plus ends of MTs and stimulates MT polymerization suggests a mechanism for its function.


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