Role of the littoral vegetation in the phosphorus and nitrogen balance of the Lake Drontermeer

1978 ◽  
Vol 20 (2) ◽  
pp. 935-938
Author(s):  
Maarten Loenen ◽  
Alie H. Koridon
1977 ◽  
Vol 53 (3) ◽  
pp. 215-220 ◽  
Author(s):  
D. G. Sapir ◽  
T. Pozefsky ◽  
J. P. Knochel ◽  
M. Walser

1. Administration of dexamethasone, 8 mg/day (0·02 mmol/day), for 5 days to normal subjects produced negative nitrogen balance, due to early and sustained increases in urinary urea nitrogen excretion. 2. In eight subjects ingesting 0·9–1·6 g of protein day−1 kg−1 body weight the cumulative increment in urea nitrogen excretion averaged + 12·5 g (sem 2·8, P < 0·01) over the 5 days of glucocorticoid administration. 3. Increases in urinary urea nitrogen excretion could be related to both plasma alanine and blood glutamine changes by using a multiple regression equation. 4. These results suggest that corticosteroids induce increased release of alanine and glutamine by peripheral tissues, which may augment urea formation and negative nitrogen balance. 5. The correlation between increments in urea nitrogen excretion and increases in plasma arginine remains unexplained.


2007 ◽  
Vol 17 (5) ◽  
pp. 275-279
Author(s):  
Alceu Afonso Jordao ◽  
Fátima Liz dos Santos ◽  
Júlio Sérgio Marchini ◽  
Helio Vannucchi

1982 ◽  
Vol 60 (3) ◽  
pp. 331-334 ◽  
Author(s):  
Maureen C. Hannaford ◽  
Marc B. Goldstein ◽  
Robert G. Josse ◽  
Mitchell L. Halperin

The purpose of these experiments was to determine if augmented renal ammoniagenesis in chronic metabolic acidosis could increase the negative nitrogen balance during prolonged fasting. To explore this question, rats and rabbits were fasted for up to 10 days because acidosis would markedly augment ammonium excretion in the rat but not in the rabbit. Since the ketoacidosis of fasting was mild in both species (< 2 mM) and ketonuria virtually absent, a hydrochloric acid load was given to stimulate renal ammoniagenesis. Under these conditions, nitrogen balance was significantly more negative during acidosis in the rat but not in the rabbit. This increment in nitrogen excretion appeared as ammonium with no detectable difference in urea nitrogen excretion. Therefore, it appears that if more nitrogen is excreted as ammonium, net protein breakdown increases to furnish the substrate for ammoniagenesis rather than reducing the excretion of the other nitrogenous waste component urea. The implications of these findings will be discussed.


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