Characteristics of Adults with Attention Deficit Hyperactivity Disorder Plus Substance Use Disorder: The Role of Psychiatric Comorbidity

2005 ◽  
Vol 14 (4) ◽  
pp. 319-327 ◽  
Author(s):  
Timothy E. Wilens ◽  
Anne Kwon ◽  
Sarah Tanguay ◽  
Rhea Chase ◽  
Hadley Moore ◽  
...  
Addiction ◽  
2013 ◽  
Vol 109 (2) ◽  
pp. 262-272 ◽  
Author(s):  
Katelijne van Emmerik-van Oortmerssen ◽  
Geurt van de Glind ◽  
Maarten W. J. Koeter ◽  
Steve Allsop ◽  
Marc Auriacombe ◽  
...  

2020 ◽  
Vol 26 (Suppl. 4-5) ◽  
pp. 179-190
Author(s):  
María C. Vélez-Pastrana ◽  
Rafael A. González ◽  
Alexandra Ramos-Fernández ◽  
Rafael R. Ramírez Padilla ◽  
Frances R. Levin ◽  
...  

CNS Spectrums ◽  
2009 ◽  
Vol 14 (S6) ◽  
pp. 10-12 ◽  
Author(s):  
Oscar Bukstein

In examining the challenges in treating comorbid attention-deficit/hyperactivity disorder (ADHD) and substance use disorder (SUD), there are a number of issues regarding misinformation and misconception that exist for clinicians. Like other ADHD comorbidities, there is a lack of screening, particularly among adult clinical populations who have the psychiatric comorbidity or, for patients with SUDs, there is the issue of prioritization, which condition to treat first, and determining the place of medication management—particularly stimulant medication—paramount in treatment planning.


CNS Spectrums ◽  
1999 ◽  
Vol 4 (5) ◽  
pp. 55-58
Author(s):  
Amy Bauer ◽  
Ranga Ram ◽  
Kim M. Schindler ◽  
Michele T. Pato ◽  
Fabio Macciardi ◽  
...  

AbstractSubstance use disorder (SUD) pedigrees identified through an attention deficit/hyperactivity disorder (ADHD) proband may be helpful in teasing apart the genetic risks for both ADHD and SUD (ie, alcohol or drug use). Pedigrees segregating for both SUD and ADHD may represent a subset of both of these common disorders that share a related genetic basis. We determined the number of SUD and ADHD pedigrees in a sample of 175 ADHD probands. We found 52 ADHD pedigrees, indicating that at least 29.7% were familial cases. We also found 50 SUD pedigrees; 13 families contained both an alcohol and a drug pedigree, 35 families were alcohol-only pedigrees, and two families were drug-only pedigrees. The incidence of drug-only pedigrees is significantly higher (P<0.01) in families with familial ADHD. This was also true for families with both drug and alcohol pedigrees (P<0.01). The total number of SUD pedigrees and the families with alcohol-alone pedigrees were not significantly different in ADHD pedigrees compared with nonfamilial ADHD families.


CNS Spectrums ◽  
2008 ◽  
Vol 13 (S12) ◽  
pp. 12-15 ◽  
Author(s):  
Jeffrey H. Newcorn

Attention-deficit/hyperactivity disorder (ADHD) is highly co-morbid across the life span. However, co-morbidity is not uniform across time; individual co-morbid conditions tend to occur at different times developmentally, with rates often reflecting lifetime occurrence (Slide 1). In addition to changes in the rates of co-morbidity, the nature of co-morbidity may also differ in late adolescence/adulthood, when co-morbid conditions can be especially impairing (eg, antisocial disorder, substance use disorder [SUD], more severe mood disorders).


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