scholarly journals Persistence of attacks of cluster headache after trigeminal nerve root section

Brain ◽  
2002 ◽  
Vol 125 (5) ◽  
pp. 976-984 ◽  
Author(s):  
Manjit S. Matharu ◽  
Peter J. Goadsby
Keyword(s):  
Cluster Headache ◽  
Trigeminal Nerve ◽  
Nerve Root ◽  
Root Section ◽  
Trigeminal Nerve Root

scholarly journals Microvascular decompression for trigeminal neuralgia caused by trigeminocerebellar artery

2020 ◽  
Vol 3 (2) ◽  
pp. V4
Author(s):  
Norio Ichimasu ◽  
Nobuyuki Nakajima ◽  
Ken Matsushima ◽  
Michihiro Kohno ◽  
Yutaka Takusagawa
Keyword(s):  
Trigeminal Neuralgia ◽  
Trigeminal Nerve ◽  
Basilar Artery ◽  
Microvascular Decompression ◽  
Nerve Root ◽  
Cerebellar Hemisphere ◽  
Link Type ◽  
Trigeminal Nerve Root ◽  
Close Relationship

In this operative video, the authors demonstrate the case of a 53-year-old woman who presented with typical right trigeminal neuralgia by a trigeminocerebellar artery (TCA). The TCA was first defined by Marinković as a unique branch of the basilar artery supplying both the trigeminal nerve root and the cerebellar hemisphere. As a result of the close relationship between this vessel and the nerve root, the TCA might compress the nerve root, thereby causing trigeminal neuralgia. However, few cases of trigeminal neuralgia caused by TCA have been reported. This video shows the microvascular decompression for trigeminal neuralgia by the TCA.The video can be found here: https://youtu.be/UnGsCQRK6aY

Vasculature and neurovascular relationships of the trigeminal nerve root

2010 ◽  
Vol 153 (5) ◽  
pp. 1051-1057 ◽  
Author(s):  
Mila Ćetković ◽  
Vaso Antunović ◽  
Slobodan Marinković ◽  
Vera Todorović ◽  
Zdravko Vitošević ◽  
...  
Keyword(s):  
Trigeminal Nerve ◽  
Nerve Root ◽  
Trigeminal Nerve Root

scholarly journals Isolated Trigeminal Neuropathy due to Trigeminal Nerve Root Hemorrhage

1999 ◽  
Vol 26 (3) ◽  
pp. 204-206 ◽  
Author(s):  
S. Almeida ◽  
C. Chalk ◽  
J. Minuk ◽  
R. Del Carpio ◽  
M. Guerin ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Head Trauma ◽  
Trigeminal Nerve ◽  
Nerve Root ◽  
Sensory Neuropathy ◽  
Trigeminal Neuropathy ◽  
Trigeminal Nerve Root ◽  
Structural Lesion ◽  
Symptoms And Signs

Background:Isolated trigeminal neuropathy is uncommon; causes include trauma, inflammation, or neoplasm.Methods:We report a patient who fell and struck his head during a myocardial infarction, was treated with streptokinase, and developed symptoms and signs of an isolated trigeminal sensory neuropathy.Results:Imaging showed hemorrhage in the trigeminal nerve root; follow-up imaging showed resolution of the hemorrhage, but no underlying structural lesion.Conclusion:A combination of head trauma plus thrombolysis resulted in an isolated trigeminal neuropathy.

scholarly journals MICROVASCULAR DECOMPRESSION IN THE TREATMENT OF PATIENTS WITH TRIGEMINAL NEURALGIA

2019 ◽  
Vol 19 (1-2) ◽  
pp. 101-106
Author(s):  
D. M Lazarchuk ◽  
G. N Alekseev ◽  
O. O Kamadey ◽  
S. N Chemidronov
Keyword(s):  
Trigeminal Neuralgia ◽  
Trigeminal Nerve ◽  
Microvascular Decompression ◽  
Nerve Root ◽  
Treatment Method ◽  
Neurovascular Conflict ◽  
Neurosurgical Department ◽  
Trigeminal Nerve Root ◽  
Trigeminal Root ◽  
Cranial Fossa

This work highlights the main variant treatment of patients with trigeminal neuralgia, with a proven neurovascular conflict, microvascular decompression of the trigeminal root. Microvascular decompression is the main radical treatment method which allows to relieve hyperfunctional syndrome manifested by prosopalgia. In the course of this study, the variant anatomy of the neurovascular conflict in patients with trigeminal neuralgia was described in detail. The group of patients whose clinical diagnosis at the stage of selection was based on a neurological examination and taking into account the progression of symptoms as well as the performed instrumental examination (CT angiography). The results are described in the article. Atrophic changes of the root of the trigeminal nerve are visualized and described. The nature of its blood supply is classified according to the type of the origin of the artery or arterial branches of the trigeminal nerve root. The main types of neurovascular conflict classified according to the type of blood vessel are presented. Variant neuroanatomy of the trigeminal nerve root as well as the interaction with the arteries of the vertebrobasilar basin and the veins of the posterior cranial fossa are described. The course of microvascular decompression of the trigeminal nerve root, used in the neurosurgical department of Samara Regional Clinical Hospital n.a. V.D. Seredavin is described

scholarly journals An Animal Model for Trigeminal Neuralgia by Compression of the Trigeminal Nerve Root

2012 ◽  
Vol 2;15 (2;3) ◽  
pp. 187-196
Author(s):  
Yun-Qing Li
Keyword(s):  
Animal Model ◽  
Trigeminal Neuralgia ◽  
Trigeminal Nerve ◽  
Nerve Root ◽  
Sham Operation ◽  
Calcitonin Gene ◽  
Trigeminal Nerve Root ◽  
Trigeminal Root ◽  
Isolectin B4 ◽  
Heat Hyperalgesia

Background: Microvascular compression of the trigeminal nerve root is a major cause of most trigeminal neuralgia (TN) in patients; however, no reliable animal model to further study the pathogenesis of TN currently exists. Objective: Our objective was to establish a novel and practical animal model for TN by chronic compression of the trigeminal (CCT) nerve root in rats, which would provide a better animal model to mimic the clinical feature of TN on the research of the pathogenesis of TN. Study Design: A randomized, double blind, controlled animal trial. Methods: Sixteen adult male Sprague-Dawley rats (200-220 g) were randomly divided into 2 groups: one group that received chronic compression of the trigeminal nerve root (the CCT group, n=8) and another group that received sham operation without compression (the sham operation group, n=8). A small plastic filament was retrogressively inserted into the intracalvarium from the inferior orbital fissure until it reached the trigeminal nerve root for compression in CCT group. Animal behaviors were observed for 4 weeks after operation. Immunohistochemistry of glial fibrillary acidic protein (GFAP), isolectin B4 (IB4), substance P (SP) and calcitonin gene-related peptide (CGRP) were performed in the trigeminal root entry zone (TREZ) and medullary dorsal horn (MDH). Results: The orofacial mechanical allodynia and heat hyperalgesia in the CCT rats were obviously increased after the operation and lasted for 28 days. Increased facegrooming behavior was also observed in the CCT rats and continued for over 21 days, returning to baseline by day 28. Immunohistochemistry for GFAP in the TREZ revealed a progressive extension of astrocytic processes in the ipsilateral TREZ of rats in the CCT group. Furthermore, the IB4 positive immunoreactive nonpeptidergic C-fiber terminals in the MDH were reduced for 4 weeks after the operation. Both SP and CGRP, expressed in the peptidergic C-fiber terminals, were found to be decreased in the ipsilateral MDH of CCT animals after the trigeminal nerve root injury. Limitations: CCT animal model with a plastic filament only imitated the mechanical compression of the trigeminal root but not to display the complex vascular physiological feature as the microvascular in the TN patient. Conclusions: The chronic compression of the trigeminal nerve root in rats effectively induced persistent orofacial neuropathic pain behaviors, and it would provide a novel and practical animal model for future research on the pathogenesis of TN. Key words: trigeminal neuralgia, nerve root compression, animal model, mechanical allodynia, heat hyperalgesia, substance P, calcitonin gene-related peptide, isolectin B4

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