Alcohol: Preventing Harmful Drinking

2011 ◽  
Vol 4 (11) ◽  
pp. 634-639 ◽  
Author(s):  
Matthew David Gee

Alcohol is more harmful than any other drug overall, when physical, psychological and social harm are taken into account. There were 8664 alcohol-related deaths in the UK in 2009. Every GP is estimated to see around 364 heavy drinkers every year and will see more who are drinking less heavily but whose alcohol consumption is still putting their health at risk. GPs are therefore in an ideal position to prevent the significant mortality and morbidity associated with high alcohol consumption. This can be done by early identification of those who are drinking more than is healthy and by providing brief interventions before drinking becomes harmful.

2021 ◽  
Author(s):  
Fernando Ezquer ◽  
Maria Elena Quintanilla ◽  
Francisco Moya‐Flores ◽  
Paola Morales ◽  
José Manuel Munita ◽  
...  

2021 ◽  
Vol 21 (1) ◽  
Author(s):  
Bo-Yeon Kim ◽  
Hyewon Nam ◽  
Jeong-Ju Yoo ◽  
Yoon-Young Cho ◽  
Dug-Hyun Choi ◽  
...  

Abstract Background This study was performed to investigate the association between the amount of alcohol consumption or binge drinking and obesity-related comorbidities in Korean men. Methods A total of 103,048 men aged 19 years or older were investigated in the 2016 Korean Community Health Survey. The participants were divided into five groups according to the standard number of alcoholic drinks consumed per week. Results Of the total participants, 20.7% were in the high alcohol consumption group, consuming more than 28 drinks per week. After adjustment for clinical factors, high alcohol consumption was significantly associated with higher odds ratios (ORs) of obesity (OR, 1.449; 95% confidence interval [CI], 1.412 to 1.591; P < 0.0001), hypertension (OR, 1.76; 95% CI, 1.636 to 1.894; P < 0.0001), and dyslipidemia (OR, 1.356; 95% CI, 1.247 to 1.474; P < 0.0001). In contrast, mild to moderate alcohol consumption was associated with a lower risk of diabetes (OR, 0.799; 95% CI, 0.726 to 0.88; P = 0.0015) and high alcohol consumption was not associated with a higher risk of diabetes (OR, 0.945; 95% CI, 0.86 to 1.039; P = 0.0662). Among drinkers, except for social drinkers, binge drinking was significantly associated with higher risks of obesity, hypertension, diabetes, and dyslipidemia. Conclusions High alcohol consumption was associated with higher risks of obesity, hypertension, and dyslipidemia in Korean men. In contrast, high consumption was not associated with a higher risk of diabetes. In particular, binge drinkers were associated with higher risks of obesity, hypertension, diabetes, and dyslipidemia compared to non-binge drinkers.


Author(s):  
Ute Mons ◽  
Thomas Gredner ◽  
Gundula Behrens ◽  
Christian Stock ◽  
Hermann Brenner

2019 ◽  
Vol 50 (3) ◽  
pp. 484-498 ◽  
Author(s):  
Andries T. Marees ◽  
Dirk J. A. Smit ◽  
Jue-Sheng Ong ◽  
Stuart MacGregor ◽  
Jiyuan An ◽  
...  

AbstractBackground. Frequency and quantity of alcohol consumption are metrics commonly used to measure alcohol consumption behaviors. Epidemiological studies indicate that these alcohol consumption measures are differentially associated with (mental) health outcomes and socioeconomic status (SES). The current study aims to elucidate to what extent genetic risk factors are shared between frequency and quantity of alcohol consumption, and how these alcohol consumption measures are genetically associated with four broad phenotypic categories: (i) SES; (ii) substance use disorders; (iii) other psychiatric disorders; and (iv) psychological/personality traits.Methods. Genome-Wide Association analyses were conducted to test genetic associations with alcohol consumption frequency (N = 438 308) and alcohol consumption quantity (N = 307 098 regular alcohol drinkers) within UK Biobank. For the other phenotypes, we used genome-wide association studies summary statistics. Genetic correlations (rg) between the alcohol measures and other phenotypes were estimated using LD score regression.Results. We found a substantial genetic correlation between the frequency and quantity of alcohol consumption (rg = 0.52). Nevertheless, both measures consistently showed opposite genetic correlations with SES traits, and many substance use, psychiatric, and psychological/personality traits. High alcohol consumption frequency was genetically associated with high SES and low risk of substance use disorders and other psychiatric disorders, whereas the opposite applies for high alcohol consumption quantity.Conclusions. Although the frequency and quantity of alcohol consumption show substantial genetic overlap, they consistently show opposite patterns of genetic associations with SES-related phenotypes. Future studies should carefully consider the potential influence of SES on the shared genetic etiology between alcohol and adverse (mental) health outcomes.


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