Neurological emergencies

Author(s):  
Punit S. Ramrakha ◽  
Kevin P. Moore ◽  
Amir Sam

Coma: assessment 332 Coma: immediate management 334 Coma: clues from examination 336 Coma: management 338 Limb weakness: assessment 340 Limb weakness: localizing the lesion 342 Acute dizziness: assessment 344 Acute dizziness: management 346 Acute loss of vision 348 Approach to acute/sub-acute visual loss 350 Painful red eye: assessment ...

2012 ◽  
Vol 2012 ◽  
pp. 1-3
Author(s):  
Mohammed M. Ziaei ◽  
Hadi Ziaei

Purpose. To present a unique case of Non-Hodgkin’s-Lymphoma- (NHL) associated compressive optic neuropathy.Method. An 89-year-old male presenting with acute unilateral visual loss and headache.Results. Patient was initially diagnosed with occult giant cell arteritis; however after visual acuity deteriorated despite normal inflammatory markers, an urgent MRI scan revealed an extensive paranasal sinus mass compressing the optic nerve.Conclusion. Paranasal sinus malignancies occasionally present to the ophthalmologist with signs of optic nerve compression and must be included in the differential diagnosis of acute visual loss.


2000 ◽  
Vol 23 (2) ◽  
pp. 89-94 ◽  
Author(s):  
Holger Allroggen ◽  
Richard J. Abbott ◽  
Kim Bibby

1970 ◽  
Vol 12 (3) ◽  
pp. 172-174
Author(s):  
Aggeliki Kolea ◽  
Aggelos Baltatzidis ◽  
Vasileios Margaritis ◽  
Belal Almoghrabi ◽  
Irini Kaldi ◽  
...  

A 38-year-old man presented with an acute right homonymous visual field defect due to occipital lobe infarct caused by hyperhomocysteinaemia. Visual field testing, magnetic resonance imaging, laboratory studies, and genetic analysis were carried out. On magnetic resonance imaging, a left occipital lesion with bright signal on the diffusion-weighted and fluid-attenuated inversion recovery images suggested a diagnosis of an acute infarct. Blood tests revealed raised homocysteine of 52.08 μmol/L (reference range, <15 μmol/L) and genetic analysis showed the patient to be homozygote to 5, 10-methylenetetrahydrofolate reductase deficiency. Hyperhomocysteinaemia is a rare causes of acute visual loss due to cerebral ischaemia and should always be suspected and investigated with the appropriate tests to diagnose the condition and limit further vision deterioration.


2018 ◽  
Vol 10 (5) ◽  
pp. e8-e8 ◽  
Author(s):  
Justin Bauer ◽  
Kartik Kansagra ◽  
Kuo H Chao ◽  
Lei Feng

Cavernous sinus thrombosis (CST) is a rare condition that can cause death, neurologic disability, and visual loss. A pre-teen with septic CST leading to ocular hypertension and acute visual loss was treated at our institution with thrombectomy and thrombolysis of the cavernous sinuses and superior ophthalmic veins. Successful recanalization of the bilateral cavernous sinuses and superior ophthalmic veins was achieved in two separate procedures without complication. The patient showed immediate symptomatic relief. He was neurologically intact without visual deficits at the 2 month follow-up. This is the first report in the literature showing the feasibility of cavernous sinus thrombectomy using current devices and techniques. Early endovascular therapy may help preserve vision in patients with acute CST.


2017 ◽  
pp. 98-100
Author(s):  
Sherif Gonem

Author(s):  
Punit S. Ramrakha ◽  
Kevin P. Moore ◽  
Amir H. Sam

This chapter discusses neurological emergencies, including coma, limb weakness, acute dizziness, acute loss of vision, painful red eye, acute bacterial meningitis, acute viral encephalitis, head injury, raised intracranial pressure (ICP), intracranial space-occupying lesion, haemorrhage (intracerebral, extradural, subdural, subarachnoid), status epilepticus (tonic–clonic), stroke, cerebral infarction syndromes, brainstem stroke, cerebellar stroke, transient ischaemic attacks (TIAs), confusional states and delirium, acute alcohol withdrawal, neuromuscular respiratory failure, myasthenic crises, spinal cord compression, Guillain–Barré syndrome (GBS), botulism, tetanus, the Glasgow Coma Scale (GCS), examination of brainstem function, and brain death.


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