Acute Visual Loss

Purpose. To present a unique case of Non-Hodgkin’s-Lymphoma- (NHL) associated compressive optic neuropathy.Method. An 89-year-old male presenting with acute unilateral visual loss and headache.Results. Patient was initially diagnosed with occult giant cell arteritis; however after visual acuity deteriorated despite normal inflammatory markers, an urgent MRI scan revealed an extensive paranasal sinus mass compressing the optic nerve.Conclusion. Paranasal sinus malignancies occasionally present to the ophthalmologist with signs of optic nerve compression and must be included in the differential diagnosis of acute visual loss.

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Acute visual loss following administration of metronidazole: A case report

Neuro-Ophthalmology ◽

10.1076/0165-8107(200004)2321-6ft089 ◽

2000 ◽

Vol 23 (2) ◽

pp. 89-94 ◽

Cited By ~ 3

Author(s):

Holger Allroggen ◽

Richard J. Abbott ◽

Kim Bibby

Keyword(s):

Case Report ◽

Visual Loss ◽

Acute Visual Loss

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Acute Visual Loss as a First Sign of Hyperhomocysteinaemia

Asian Journal of Ophthalmology ◽

10.35119/asjoo.v12i3.308 ◽

1970 ◽

Vol 12 (3) ◽

pp. 172-174

Author(s):

Aggeliki Kolea ◽

Aggelos Baltatzidis ◽

Vasileios Margaritis ◽

Belal Almoghrabi ◽

Irini Kaldi ◽

...

Keyword(s):

Magnetic Resonance Imaging ◽

Magnetic Resonance ◽

Visual Field ◽

Genetic Analysis ◽

Visual Loss ◽

Methylenetetrahydrofolate Reductase ◽

Occipital Lobe ◽

Field Testing ◽

Resonance Imaging ◽

Acute Visual Loss

A 38-year-old man presented with an acute right homonymous visual field defect due to occipital lobe infarct caused by hyperhomocysteinaemia. Visual field testing, magnetic resonance imaging, laboratory studies, and genetic analysis were carried out. On magnetic resonance imaging, a left occipital lesion with bright signal on the diffusion-weighted and fluid-attenuated inversion recovery images suggested a diagnosis of an acute infarct. Blood tests revealed raised homocysteine of 52.08 μmol/L (reference range, <15 μmol/L) and genetic analysis showed the patient to be homozygote to 5, 10-methylenetetrahydrofolate reductase deficiency. Hyperhomocysteinaemia is a rare causes of acute visual loss due to cerebral ischaemia and should always be suspected and investigated with the appropriate tests to diagnose the condition and limit further vision deterioration.

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Acute visual loss in a patient with optic disc drusen

Clinical Ophthalmology ◽

10.2147/opth.s42233 ◽

2013 ◽

pp. 795 ◽

Cited By ~ 2

Author(s):

Deborah Tan ◽

Sharon Tow

Keyword(s):

Optic Disc ◽

Visual Loss ◽

Acute Visual Loss ◽

Optic Disc Drusen

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Republished: Transfemoral thrombectomy in the cavernous sinus and superior ophthalmic vein

Journal of NeuroInterventional Surgery ◽

10.1136/neurintsurg-2017-013571.rep ◽

2018 ◽

Vol 10 (5) ◽

pp. e8-e8 ◽

Cited By ~ 2

Author(s):

Justin Bauer ◽

Kartik Kansagra ◽

Kuo H Chao ◽

Lei Feng

Keyword(s):

Cavernous Sinus ◽

Visual Loss ◽

Sinus Thrombosis ◽

Symptomatic Relief ◽

Rare Condition ◽

Visual Deficits ◽

Acute Visual Loss ◽

Neurologic Disability ◽

Neurologically Intact

Cavernous sinus thrombosis (CST) is a rare condition that can cause death, neurologic disability, and visual loss. A pre-teen with septic CST leading to ocular hypertension and acute visual loss was treated at our institution with thrombectomy and thrombolysis of the cavernous sinuses and superior ophthalmic veins. Successful recanalization of the bilateral cavernous sinuses and superior ophthalmic veins was achieved in two separate procedures without complication. The patient showed immediate symptomatic relief. He was neurologically intact without visual deficits at the 2 month follow-up. This is the first report in the literature showing the feasibility of cavernous sinus thrombectomy using current devices and techniques. Early endovascular therapy may help preserve vision in patients with acute CST.

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Diagnosis in Acute Medicine ◽

10.4324/9781315383798-24 ◽

2017 ◽

pp. 98-100

Author(s):

Sherif Gonem

Keyword(s):

Visual Loss ◽

Acute Visual Loss

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A 16-Year Study on Incidence and Progression of Diseased Sphenoethmoidal (Onodi) Cell

BioMed Research International ◽

10.1155/2020/2410415 ◽

2020 ◽

Vol 2020 ◽

pp. 1-5

Author(s):

Ethan I. Huang ◽

Chia-Ling Kuo ◽

Li-Wen Lee

Keyword(s):

Computed Tomography ◽

Decision Making ◽

Optic Nerve ◽

Endoscopic Sinus Surgery ◽

Visual Loss ◽

Sinus Surgery ◽

Acute Visual Loss ◽

Computed Tomography Images ◽

Onodi Cell

Traumatic operative injury of the optic nerve in an endoscopic sinus surgery may cause immediate or delayed blindness. It should be cautioned when operating in a sphenoethmoidal cell, or known as Onodi cell, with contact or bulge of the optic canal. It remains unclear how frequent progression to visual loss occurs and how long it progresses to visual loss because of a diseased sphenoethmoidal cell. Research to discuss these questions is expected to help decision making to treat diseased sphenoethmoidal cells. From July 2001 to June 2017, 216 patients received conservative endoscopic sinus surgery without opening a diseased sphenoethmoidal cell. We used their computed tomography images of paranasal sinuses to identify diseased sphenoethmoidal cells that could be associated with progression to visual loss. Among the 216 patients, 52.3% had at least one sphenoethmoidal cell, and 14.8% developed at least one diseased sphenoethmoidal cell. One patient developed acute visual loss 4412 days after the first computed tomography. Our results show that over half of the patients have a sphenoethmoidal cell but suggest a rare incidence of a diseased sphenoethmoidal cell progressing to visual loss during the follow-up period.

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