Cloning and Characterization of cDNA of Avirulence Geneavr9of the Fungal PathogenCladosporium fulvum, Causal Agent of Tomato Leaf Mold

1991 ◽  
Vol 4 (1) ◽  
pp. 52 ◽  
Author(s):  
Jan A. L. van Kan
2008 ◽  
Vol 75 (2) ◽  
pp. 542-546 ◽  
Author(s):  
Hélène Lacroix ◽  
Pietro D. Spanu

ABSTRACT In this study, we have constructed and expressed inverted repeat chimeras from the first exons of the six known hydrophobins of the fungus Cladosporium fulvum, the causal agent of tomato leaf mold. We used quantitative PCR to measure specifically the expression levels of the hydrophobins. The targeted genes are silenced to different degrees, but we also detected clear changes in the expression levels of nontargeted genes. This work highlights the difficulties that are likely to be encountered when attempting to silence more than one gene in a multigene family.


2014 ◽  
Vol 56 (0) ◽  
pp. 91-93
Author(s):  
Hideki Watanabe ◽  
Yasunori Muramoto ◽  
Masatoshi Adachi ◽  
Motoyuki Yajima

1964 ◽  
Vol 42 (11) ◽  
pp. 1555-1558 ◽  
Author(s):  
D. L. Bailey ◽  
E. A. Kerr

A new race of Cladosporium fulvum Cke., the tomato leaf mold pathogen, appeared on the tomato lines Purdue 38 and Purdue 135 in commercial greenhouses in the vicinity of Cincinnati, Ohio, in the fall of 1962. These lines incorporate from L. hirsutum the gene Cf4, which confers immunity to races 1 to 9. Greenhouse inoculations indicate that race 10 is similar to race 1 in pathogenicity against Cf1, Cf2, and Cf3. Varieties such as Vetomold, Tucker's Forcing, and Manalucie, which contain Cf2, are accordingly immune from race 10.


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