Impact of Pre- and Postnatal Exposure to Environmental Tobacco Smoke (Ets) on Sudden Infant Death Syndrome (Sids)

Epidemiology ◽  
2009 ◽  
Vol 20 ◽  
pp. S51-S52
Author(s):  
Dimitri Hauri ◽  
Anke Huss ◽  
Evelyn Mohler ◽  
Patrizia Frei ◽  
Martin Röösli
Keyword(s):  
Sudden Infant Death Syndrome ◽  
Environmental Tobacco Smoke ◽  
Tobacco Smoke ◽  
Infant Death ◽  
Sudden Infant Death ◽  
Sudden Infant ◽  
Postnatal Exposure

Impact of Environmental Tobacco Smoke (ETS) on Sudden Infant Death Syndrome (SIDS) in European Children

Epidemiology ◽  
2007 ◽  
Vol 18 (Suppl) ◽  
pp. S58
Author(s):  
E Boldo ◽  
M Öberg ◽  
S Medina ◽  
N Valero ◽  
K Patja ◽  
...  
Keyword(s):  
Sudden Infant Death Syndrome ◽  
Environmental Tobacco Smoke ◽  
Tobacco Smoke ◽  
Infant Death ◽  
Sudden Infant Death ◽  
Sudden Infant

scholarly journals Health Impact Assessment of Environmental Tobacco Smoke in European Children: Sudden Infant Death Syndrome and Asthma Episodes

2010 ◽  
Vol 125 (3) ◽  
pp. 478-487 ◽  
Author(s):  
Elena Boldo ◽  
Sylvia Medina ◽  
Mattias Öberg ◽  
Vladimíra Puklová ◽  
Odile Mekel ◽  
...  
Keyword(s):  
Impact Assessment ◽  
Sudden Infant Death Syndrome ◽  
Environmental Tobacco Smoke ◽  
Tobacco Smoke ◽  
Infant Death ◽  
Health Impact Assessment ◽  
Sudden Infant Death ◽  
Health Impact ◽  
Sudden Infant

Antimony leaching from cot mattresses and sudden infant death syndrome (SIDS)

1998 ◽  
Vol 17 (3) ◽  
pp. 138-139 ◽  
Author(s):  
R O Jenkins ◽  
P J Craig ◽  
W Goessler ◽  
K J Irgolic
Keyword(s):  
Elevated Temperature ◽  
Sudden Infant Death Syndrome ◽  
Infant Death ◽  
Sudden Infant Death ◽  
High Variability ◽  
Sudden Infant ◽  
Postnatal Exposure ◽  
Antimony Content ◽  
Healthy Infants ◽  
Potential Sources

1 Polyvinyl chloride (PVC) cot mattress covers from SIDS cases were investigated as potential sources of soluble (potentially ingestable) antimony in the cot environment. 2 Body fluids (urine, saliva) and proprietary domestic detergents/sterilizing fluids markedly enhanced leaching of antimony from PVC. Release of antimony was also enhanced at both low and high pH and by elevated temperature. The extent of antimony leaching did not correlate well with PVC content of this element. 3 These data do not support the assumption that postmortem analysis of antimony content proves exposure to gaseous antimony trihydride from mattress PVC. 4 Ingestion of antimony released from PVC could account for the high variability associated with reported detectable levels of antimony in liver from both SIDS and other infants. It could also explain suspected additional postnatal exposure to this element, which gives rise to elevated levels of Sb in the hair of some healthy infants.

Prenatal and Postnatal Environmental Tobacco Smoke Exposure and Children’s Health

PEDIATRICS ◽  
2004 ◽  
Vol 113 (Supplement_3) ◽  
pp. 1007-1015 ◽  
Author(s):  
Joseph R. DiFranza ◽  
C. Andrew Aligne ◽  
Michael Weitzman
Keyword(s):  
Environmental Tobacco Smoke ◽  
Tobacco Smoke ◽  
Behavioral Problems ◽  
Fetal Development ◽  
Sudden Infant Death ◽  
Environmental Tobacco Smoke Exposure ◽  
Tobacco Smoke Exposure ◽  
Sudden Infant ◽  
Postnatal Exposure ◽  
Tract Infections

Children’s exposure to tobacco constituents during fetal development and via environmental tobacco smoke (ETS) exposure is perhaps the most ubiquitous and hazardous of children’s environmental exposures. A large literature links both prenatal maternal smoking and children’s ETS exposure to decreased lung growth and increased rates of respiratory tract infections, otitis media, and childhood asthma, with the severity of these problems increasing with increased exposure. Sudden infant death syndrome, behavioral problems, neurocognitive decrements, and increased rates of adolescent smoking also are associated with such exposures. Studies of each of these problems suggest independent effects of both pre- and postnatal exposure for each, with the respiratory risk associated with parental smoking seeming to be greatest during fetal development and the first several years of life.

scholarly journals Sudden infant death syndrome: deletions of glutathione-S-transferase genes M1 and T1 and tobacco smoke exposure

2021 ◽  
Author(s):  
Anthea Mawick ◽  
Heidi Pfeiffer ◽  
Marielle Vennemann
Keyword(s):  
Sudden Infant Death Syndrome ◽  
Tobacco Smoke ◽  
Infant Death ◽  
Sudden Infant Death ◽  
Smoke Exposure ◽  
Tobacco Smoke Exposure ◽  
Control Group ◽  
Sudden Infant ◽  
Gene Deletions ◽  
Group A

AbstractIn developed countries, sudden infant death syndrome (SIDS) is the leading cause of death in infants in their first year of life. The risk of SIDS is increased if parents smoked during pregnancy and in presence of the child. Glutathione S-transferases (GSTs) catalyse the conjugation of glutathione with electrophilic compounds and toxins, making them less reactive and easier to excrete. As a gene dose effect was observed for GSTM1 and GSTT1, the aim of this study was to investigate whether there is a connection between homozygous or heterozygous gene deletions of GSTM1 or GSTT1 and the occurrence of SIDS. We found that heterozygous deletion of GSTM1 occurred significantly more frequently in the SIDS case group compared to the control group. A homozygous deletion of GSMT1 was slightly more frequently in the control group. A homozygous gene deletion of GSTT1 showed no significant difference between the SIDS group and the control group. We also found that in the SIDS group, the number of victims that were exposed to cigarette smoke was significantly higher than the number of victims without cigarette smoke exposure and that the mean lifetime of children whose mothers smoked was shorter in comparison with non-smoking mothers. In SIDS cases with homozygous gene deletions of GSTM1, the median life span of children with tobacco smoke exposure was 60 days shorter than without smoke exposure. In conclusion, the absence of these two genes is not the only trigger for SIDS but could be a critical aspect of SIDS aetiology, particularly in SIDS cases with smoking parents.

Sudden Infant Death Syndrome: Police Can Make a Difference

1998 ◽  
Author(s):  
Linda Esposito ◽  
Larry Minda ◽  
Claire Forman
Keyword(s):  
Sudden Infant Death Syndrome ◽  
Infant Death ◽  
Sudden Infant Death ◽  
Sudden Infant
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