scholarly journals Exact variational dynamics of the multimode Bose-Hubbard model based on SU(M) coherent states

2021 ◽  
Vol 103 (4) ◽  
Author(s):  
Yulong Qiao ◽  
Frank Grossmann
Author(s):  
E. Picari ◽  
A. Ponno ◽  
L. Zanelli

AbstractWe prove that the flow of the discrete nonlinear Schrödinger equation (DNLS) is the mean field limit of the quantum dynamics of the Bose–Hubbard model for N interacting particles. In particular, we show that the Wick symbol of the annihilation operators evolved in the Heisenberg picture converges, as N becomes large, to the solution of the DNLS. A quantitative $$L^p$$ L p -estimate, for any $$p \ge 1$$ p ≥ 1 , is obtained with a linear dependence on time due to a Gaussian measure on initial data coherent states.


2003 ◽  
Vol 17 (27) ◽  
pp. 4859-4866
Author(s):  
F. MADOURI ◽  
Y. HASSOUNI ◽  
M. EL BAZ

An extended Hubbard model with phonons is considered. q-coherent states relative to the superconducting quantum symmetry of the model are constructed and their properties studied. It is shown that they can have energy expectation lower than eigenstates constructed via conventional processes and that they exhibit ODLRO.


2020 ◽  
Vol 43 ◽  
Author(s):  
Peter Dayan

Abstract Bayesian decision theory provides a simple formal elucidation of some of the ways that representation and representational abstraction are involved with, and exploit, both prediction and its rather distant cousin, predictive coding. Both model-free and model-based methods are involved.


2001 ◽  
Vol 7 (S2) ◽  
pp. 578-579
Author(s):  
David W. Knowles ◽  
Sophie A. Lelièvre ◽  
Carlos Ortiz de Solόrzano ◽  
Stephen J. Lockett ◽  
Mina J. Bissell ◽  
...  

The extracellular matrix (ECM) plays a critical role in directing cell behaviour and morphogenesis by regulating gene expression and nuclear organization. Using non-malignant (S1) human mammary epithelial cells (HMECs), it was previously shown that ECM-induced morphogenesis is accompanied by the redistribution of nuclear mitotic apparatus (NuMA) protein from a diffuse pattern in proliferating cells, to a multi-focal pattern as HMECs growth arrested and completed morphogenesis . A process taking 10 to 14 days.To further investigate the link between NuMA distribution and the growth stage of HMECs, we have investigated the distribution of NuMA in non-malignant S1 cells and their malignant, T4, counter-part using a novel model-based image analysis technique. This technique, based on a multi-scale Gaussian blur analysis (Figure 1), quantifies the size of punctate features in an image. Cells were cultured in the presence and absence of a reconstituted basement membrane (rBM) and imaged in 3D using confocal microscopy, for fluorescently labeled monoclonal antibodies to NuMA (fαNuMA) and fluorescently labeled total DNA.


Author(s):  
Charles Bouveyron ◽  
Gilles Celeux ◽  
T. Brendan Murphy ◽  
Adrian E. Raftery

Author(s):  
Jonathan Jacky ◽  
Margus Veanes ◽  
Colin Campbell ◽  
Wolfram Schulte
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