Essay on Contemporary Issues in Ethology: Behavioral Ecology and the Study of Partner Choice

Ethology ◽  
2010 ◽  
Vol 99 (4) ◽  
pp. 265-277 ◽  
Author(s):  
Lee Alan Dugatkin ◽  
Andrew Sih
2012 ◽  
Author(s):  
Kristin L. Laidre ◽  
Harry Stern ◽  
Mads P. Heide-Jorgensen ◽  
Marianne Rasmussen ◽  
Mikkel V. Jensen
Keyword(s):  

2013 ◽  
Author(s):  
Kristin L. Laidre ◽  
Harry Stern ◽  
Mads P. Heide-Joergensen ◽  
Marianne Rasmussen ◽  
Mikkel V. Jensen
Keyword(s):  

2019 ◽  
Author(s):  
MARC MANGEL ◽  
COLIN W. CLARK

2021 ◽  
Vol 10 (6) ◽  
pp. 212
Author(s):  
Jeffrey Winking ◽  
Jeremy Koster

There exists a paucity of evolution-oriented research focusing on why relationships end, particularly in comparison to the substantial literature centered around individual preferences that define the beginning of relationships. In contrast, there is a long tradition in the fields of sociology and family studies of exploring divorce; however, this body of research is largely limited to studies of Western populations. We address these gaps in the literature with an examination of patterns of divorce among a small-scale horticultural population in Nicaragua. We test a number of hypotheses derived from behavioral ecology perspective regarding the timing and causes of divorce. Results lend support to all but one of the hypotheses. Overall divorce rates are comparable to U.S. rates; however, they tend to occur earlier in marriages. Children appear to provide a slight buffering effect against divorce, although age in marriage does not. Gender differences in the reported causes of divorce fall along the lines that would be expected due to differences in partner preferences reported in previous research. Finally, this population also exhibits a similar peculiar pattern exhibited by Western populations, in which divorce is more costly for women, and yet women are slightly more likely to initiate divorces than husbands.


Genetics ◽  
1999 ◽  
Vol 153 (2) ◽  
pp. 607-620 ◽  
Author(s):  
Jeremy M Grushcow ◽  
Teresa M Holzen ◽  
Ken J Park ◽  
Ted Weinert ◽  
Michael Lichten ◽  
...  

Abstract Checkpoint gene function prevents meiotic progression when recombination is blocked by mutations in the recA homologue DMC1. Bypass of dmc1 arrest by mutation of the DNA damage checkpoint genes MEC1, RAD17, or RAD24 results in a dramatic loss of spore viability, suggesting that these genes play an important role in monitoring the progression of recombination. We show here that the role of mitotic checkpoint genes in meiosis is not limited to maintaining arrest in abnormal meioses; mec1-1, rad24, and rad17 single mutants have additional meiotic defects. All three mutants display Zip1 polycomplexes in two- to threefold more nuclei than observed in wild-type controls, suggesting that synapsis may be aberrant. Additionally, all three mutants exhibit elevated levels of ectopic recombination in a novel physical assay. rad17 mutants also alter the fraction of recombination events that are accompanied by an exchange of flanking markers. Crossovers are associated with up to 90% of recombination events for one pair of alleles in rad17, as compared with 65% in wild type. Meiotic progression is not required to allow ectopic recombination in rad17 mutants, as it still occurs at elevated levels in ndt80 mutants that arrest in prophase regardless of checkpoint signaling. These observations support the suggestion that MEC1, RAD17, and RAD24, in addition to their proposed monitoring function, act to promote normal meiotic recombination.


2021 ◽  
Vol 173 ◽  
pp. 115-136
Author(s):  
J.S. Martin ◽  
S.E. Koski ◽  
T. Bugnyar ◽  
A.V. Jaeggi ◽  
J.J.M. Massen

Sign in / Sign up

Export Citation Format

Share Document