A Possible Role of the Specialized Conduction System in the Conversion of Ventricular Tachycardia to Ventricular Fibrillation*

1982 ◽  
Vol 5 (5) ◽  
pp. 683-687 ◽  
Author(s):  
PAUL SCHROEDER ◽  
CHALMERS LYONS
Keyword(s):  
Ventricular Tachycardia ◽  
Ventricular Fibrillation ◽  
Conduction System

Prognosis of patients with ventricular tachycardia and ventricular fibrillation: Role of the underlying etiology

2000 ◽  
pp. 411-422
Author(s):  
Hans-Joachim Trappe ◽  
Pedro Brugada ◽  
Mario Talajic ◽  
Paolo Della Bella ◽  
Roman Lezaun ◽  
...  
Keyword(s):  
Ventricular Tachycardia ◽  
Ventricular Fibrillation

scholarly journals Proarrhythmic Effect of RF ablation on the Right Ventricular Moderator Band

2021 ◽  
Author(s):  
Jonathan Willner ◽  
Parth Makker ◽  
Roy John
Keyword(s):  
Ventricular Tachycardia ◽  
Ventricular Fibrillation ◽  
Right Ventricular ◽  
Ventricular Arrhythmias ◽  
Conduction System ◽  
Rf Ablation ◽  
Moderator Band ◽  
Coupling Interval ◽  
The Right ◽  
Durable Suppression

The right ventricular moderator band (MB) is increasingly being recognized as a source for PVCs and PVC-mediated ventricular fibrillation. Monomorphic PVCs, non-sustained monomorphic VT and ventricular fibrillation are all documented arrhythmias originating from the MB. The benign PVCs usually have a coupling interval in excess of 400 msec. When PVCs trigger VF, coupling intervals are typically short, less than 300 msec. We report here a case of long-standing frequent monomorphic PVCs with a coupling interval of > 400 msec from the right ventricular distal conduction system embedded in the moderator band that progressed to non-sustained ventricular tachycardia. Following suppression of the arrhythmia with RF ablation, the arrhythmia recurred with PVCs at a shorter coupling interval (<300 msec), with frequent repetitive non-sustained polymorphic VT and triggering of sustained ventricular fibrillation. The use of a cryoballoon to ablate over the course of the moderator band resulted in complete and durable suppression of ventricular arrhythmias.

Purkinje involvement in arrhythmias after coronary artery reperfusion

2002 ◽  
Vol 282 (4) ◽  
pp. H1189-H1196 ◽  
Author(s):  
David O. Arnar ◽  
James B. Martins
Keyword(s):  
Ventricular Tachycardia ◽  
Coronary Artery ◽  
Ventricular Fibrillation ◽  
Cycle Length ◽  
Ischemic Myocardium ◽  
Risk Zone ◽  
Purkinje System ◽  
Mean Cycle

Previous studies have indicated that the endocardium may be responsible for a large portion of ventricular tachycardia (VT) seen with reperfusion of ischemic myocardium. To evaluate the role of the Purkinje system in nonreentrant VT arising from the endocardium after reperfusion, the anterior descending coronary artery was occluded for 20 min and then reperfused in 23 dogs after instrumentation of the risk zone with 21 multipolar plunge needles. VT with focal Purkinje origin was defined as a focal endocardial VT with Purkinje potentials recorded before the earliest endocardial myopotential. A total of 19 VTs (mean cycle length 214 ± 2 ms) were observed with 11 (58%) having focal Purkinje origin. Fifty-eight percent of the VTs degenerated to ventricular fibrillation, with occurrences of two or more independent foci per complex (seen in 7 of 11 compared with 1 of 8 nonsustained VTs). In conclusion, these data show that Purkinje tissue may be important in the genesis of reperfusion VT.

scholarly journals Prognosis of patients with ventricular tachycardia and ventricular fibrillation: Role of the underlying etiology

1988 ◽  
Vol 12 (1) ◽  
pp. 166-174 ◽  
Author(s):  
Hans-Joachim Trappe ◽  
Pedro Brugada ◽  
Mario Talajic ◽  
Paolo Della Bella ◽  
Roman Lezaun ◽  
...  
Keyword(s):  
Ventricular Tachycardia ◽  
Ventricular Fibrillation

Ventricular tachyarrhythmias: Ventricular tachycardia and ventricular fibrillation

2018 ◽  
Author(s):  
Michael Jones ◽  
Norman Qureshi ◽  
Kim Rajappan
Keyword(s):  
Ventricular Tachycardia ◽  
Ventricular Fibrillation ◽  
Heart Rhythm ◽  
Ventricular Myocardium ◽  
Conduction System ◽  
Ventricular Tachyarrhythmias ◽  
Normal Heart Rate ◽  
Ventricular Tissue ◽  
Life Threatening ◽  
Broad Complex

Ventricular tachyarrhythmias are abnormal patterns of electrical activity arising from the ventricular tissue (myocardium and conduction tissue). Ventricular tachycardia (VT) is an abnormal rapid heart rhythm originating from the ventricles. The rhythm may arise from the ventricular myocardium and/or from the distal conduction system. The normal heart rate is usually regular, between 60 and 100 bpm, and there is synchronized atrial and ventricular contraction. In VT, the ventricles contract at a rate greater than 120 bpm and typically from 150 to 300 bpm, and are no longer coordinated with the atria. There is still organized contraction of the ventricles in VT, with discrete QRS complexes. It is a potentially life-threatening arrhythmia, with the risk of degenerating into ventricular fibrillation and resulting in sudden cardiac death. It is characterized by a broad-complex tachycardia on ECG.

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