Progress of cassava mosaic virus disease and whitefly vector populations in single and mixed stands of four cassava varieties grown under epidemic conditions in Uganda

2001 ◽  
Vol 138 (2) ◽  
pp. 161-170 ◽  
Author(s):  
W S SSERUBOMBWE ◽  
J M THRESH ◽  
G W OTIM-NAPE ◽  
D O S OSIRU
2021 ◽  
Author(s):  
Olufemi Joseph Alabi ◽  
Rabson Mulenga

Abstract Like other CMGs, cassava is the primary host of East African cassava mosaic virus (EACMV) and related viruses, although the virus has been detected in other plant species (Ogbe et al., 2006; Alabi et al. 2015). Analysis of the genomes of different isolates of EACMV-type viruses show considerable genetic variability and genome plasticity relative to ACMV isolates. The primary means of virus spread is via movement of contaminated vegetative cassava cuttings and secondary spread occurs via the whitefly vector, Bemisia tabaci. Perhaps the most notable documentation of invasiveness of EACMV-type viruses is the regional pandemic of a severe CMD in East Africa caused by EACMV-UG which began in Uganda in the early to mid-1990s (Gibson et al., 1996; Otim-Nape et al., 1997) on popular and widely cultivated cassava varieties and soon spread to other countries in East Africa, including Kenya and Tanzania (Otim-Nape et al., 1997; Legg, 1999). The pandemic resulted in famine-related deaths (Otim-Nape et al., 1998) due to complete devastation of affected cassava farms in the region. EACMV is not on the IUCN or ISSG alert list.


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