Association of Myroides odoratimimus in immunocompromized piglets with post weaning multisystemic wasting syndrome

2019 ◽  
Vol 127 (6) ◽  
pp. 1635-1645
Author(s):  
M. Choudhary ◽  
B.K. Choudhary ◽  
B.C. Bera ◽  
S.P. Chaudhari ◽  
D.K. Giri ◽  
...  
Keyword(s):  
Wasting Syndrome ◽  
Myroides Odoratimimus

Peroxisome proliferation: Organelles or activity as an endpoint?

1993 ◽  
Vol 51 ◽  
pp. 396-397
Author(s):  
Catherine A. Taylor ◽  
Bruce M. Jarnot
Keyword(s):  
Acid Oxidation ◽  
Peroxisome Proliferation ◽  
Post Dose ◽  
Oxidation Activity ◽  
Wasting Syndrome ◽  
Perfluorodecanoic Acid ◽  
Hypolipidemic Agent ◽  
Commercially Important ◽  
Acyl Coa Oxidase ◽  
Mitochondrial Disruption

Peroxisome induction can be expressed as an increase in peroxisome area (proliferation) or as an increase in peroxisomal fatty acid oxidation (activity). This study compares proliferation and activity as endpoints for hepatic peroxisome induction by perfluorodecanoic acid (PFDA). Fluorocarboxylic acids such as PFDA represent a class of compounds possessing commercially important surfactant properties. A single 50 mg/Kg ip. dose of PFDA produces a characteristic “wasting syndrome” in male F-344 rats. Symptoms include hypophagia, weight loss, hepatomegaly, and delayed lethality. Hepatic studies reveal changes similar to those seen with the hypolipidemic agent clofibrate. These include mitochondrial disruption, endoplasmic reticulum and peroxisome proliferation, and increased peroxisomal acyl-CoA oxidase activity.Male Fisher-344 rats received a single ip. dose of 2, 20, or 50mg/Kg PFDA dissolved in 1:1 propylene glycol/water and were sacrificed 8 days post-dose. All control rats received an equal volume of vehicle ip. Animals were provided food and water ad libitum, except pair-fed controls which received the same restrictive food intake consumed by their weight-paired dosed partners (50mg/Kg PFDA group) to simulate the hypophagia associated with PFDA.

Testing ecological release as a compensating mechanism for mass mortality in a keystone predator

2020 ◽  
Vol 637 ◽  
pp. 59-69 ◽  
Author(s):  
J Sullivan-Stack ◽  
BA Menge
Keyword(s):  
Community Dynamics ◽  
Limiting Factor ◽  
Sea Star ◽  
Top Predator ◽  
Oregon Coast ◽  
Ecological Release ◽  
Keystone Predator ◽  
Pisaster Ochraceus ◽  
Wasting Syndrome

Top predator decline has been ubiquitous across systems over the past decades and centuries, and predicting changes in resultant community dynamics is a major challenge for ecologists and managers. Ecological release predicts that loss of a limiting factor, such as a dominant competitor or predator, can release a species from control, thus allowing increases in its size, density, and/or distribution. The 2014 sea star wasting syndrome (SSWS) outbreak decimated populations of the keystone predator Pisaster ochraceus along the Oregon coast, USA. This event provided an opportunity to test the predictions of ecological release across a broad spatial scale and determine the role of competitive dynamics in top predator recovery. We hypothesized that after P. ochraceus loss, populations of the subordinate sea star Leptasterias sp. would grow larger, more abundant, and move downshore. We based these predictions on prior research in Washington State showing that Leptasterias sp. competed with P. ochraceus for food. Further, we predicted that ecological release of Leptasterias sp. could provide a bottleneck to P. ochraceus recovery. Using field surveys, we found no clear change in density or distribution in Leptasterias sp. populations post-SSWS, and decreases in body size. In a field experiment, we found no evidence of competition between similar-sized Leptasterias sp. and P. ochraceus. Thus, the mechanisms underlying our predictions were not in effect along the Oregon coast, which we attribute to differences in habitat overlap and food availability between the 2 regions. Our results suggest that response to the loss of a dominant competitor can be unpredictable even when based in theory and previous research.

Central diabetes insipidus and cerebral salt wasting syndrome: a challenging coexistence

2017 ◽  
Author(s):  
Maria Manuel Costa ◽  
Cesar Esteves ◽  
Jose Luis Castedo ◽  
Josue Pereira ◽  
Davide Carvalho
Keyword(s):  
Diabetes Insipidus ◽  
Central Diabetes Insipidus ◽  
Cerebral Salt Wasting Syndrome ◽  
Cerebral Salt Wasting ◽  
Salt Wasting ◽  
Wasting Syndrome ◽  
Salt Wasting Syndrome

Severe salt wasting syndrome due to spontaneous epidural haematoma

2019 ◽  
Author(s):  
Aoife Garrrahy ◽  
Osamah Hakami ◽  
Iona Galloway ◽  
Stephen McNally ◽  
Rory Dwyer ◽  
...  
Keyword(s):  
Epidural Haematoma ◽  
Salt Wasting ◽  
Wasting Syndrome ◽  
Salt Wasting Syndrome

Nomenclature Abstract for Myroides odoratimimus odoratimimus (Vancanneyt et al. 1996) García-López et al. 2020.

2003 ◽  
Author(s):  
Charles Thomas Parker ◽  
George M Garrity
Keyword(s):  
Myroides Odoratimimus

V. The epithelial sodium channel and its implication in human diseases

1999 ◽  
Vol 276 (3) ◽  
pp. G567-G571 ◽  
Author(s):  
Edith Hummler ◽  
Jean-Daniel Horisberger
Keyword(s):  
Distal Colon ◽  
Loss Of Function ◽  
Newborn Mice ◽  
Α Subunit ◽  
Salt Wasting ◽  
Lung Fluid ◽  
Wasting Syndrome ◽  
Rate Limiting Step ◽  
Γ Subunit ◽  
Rate Limiting

The epithelial Na+ channel (ENaC) controls the rate-limiting step in the process of transepithelial Na+ reabsorption in the distal nephron, the distal colon, and the airways. Hereditary salt-losing syndromes have been ascribed to loss of function mutations in the α-, β-, or γ-ENaC subunit genes, whereas gain of function mutations (located in the COOH terminus of the β- or γ-subunit) result in hypertension due to Na+ retention (Liddle’s syndrome). In mice, gene-targeting experiments have shown that, in addition to the kidney salt-wasting phenotype, ENaC was essential for lung fluid clearance in newborn mice. Disruption of the α-subunit resulted in a complete abolition of ENaC-mediated Na+ transport, whereas knockout of the β- or γ-subunit had only minor effects on fluid clearance in lung. Disruption of each of the three subunits resulted in a salt-wasting syndrome similar to that observed in humans.

PRES followed by cerebral salt‐wasting syndrome in a child with IgA nephropathy

2021 ◽  
Author(s):  
Kohei Haraguchi ◽  
Takahisa Kimata ◽  
Shohei Akagawa ◽  
Sohsaku Yamanouchi ◽  
Kazunari Kaneko
Keyword(s):  
Iga Nephropathy ◽  
Cerebral Salt Wasting Syndrome ◽  
Cerebral Salt Wasting ◽  
Salt Wasting ◽  
Wasting Syndrome ◽  
Salt Wasting Syndrome

Torque teno sus virus 1 and 2 viral loads in postweaning multisystemic wasting syndrome (PMWS) and porcine dermatitis and nephropathy syndrome (PDNS) affected pigs

2011 ◽  
Vol 153 (3-4) ◽  
pp. 377-381 ◽  
Author(s):  
M. Aramouni ◽  
J. Segalés ◽  
M. Sibila ◽  
G.E. Martin-Valls ◽  
D. Nieto ◽  
...  
Keyword(s):  
Postweaning Multisystemic Wasting Syndrome ◽  
Viral Loads ◽  
Wasting Syndrome ◽  
Torque Teno Sus Virus
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