Spontaneous variations in venous tone in man

1960 ◽  
Vol 15 (2) ◽  
pp. 255-257 ◽  
Author(s):  
Antonio C. Quiroz ◽  
George E. Burch ◽  
German E. Malaret
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Strain Gauge ◽  
Human Subjects ◽  
Venous Pressure ◽  
Venous Tone ◽  
Pressure Transducers ◽  
Gauge Pressure ◽  
Venous Segment

Using the intact isolated venous segment technique, Statham strain-gauge pressure transducers and a multichannel direct recorder, spontaneous changes in venous tone were recorded in human subjects with and without congestive heart failure. Evidence was presented to show the existence of spontaneous variations in venous tone. Specific venous pressure deflections were described similar to, if not identical with, the alpha and beta deflections described in digital plethysmography. Venous pressure deflections associated with respiration and with transmitted arterial pulsations were also described. The effect of Cheyne-Stokes respiration on systemic venous pressure was observed fortuitously in one subject. It was noted that during sleep venous pressure usually decreased but occasionally increased. This increase is probably associated with dreams or other disturbances. Spontaneous venomotor changes were found equally in subjects with and those without congestive heart failure. Submitted on September 14, 1959

scholarly journals Protein-Losing Enteropathy and Plastic Bronchitis After the Fontan Operation

2018 ◽  
Vol 38 (6) ◽  
pp. e5-e12 ◽  
Author(s):  
Christine Peyton
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Complex Disease ◽  
Fontan Procedure ◽  
Fontan Operation ◽  
Venous Pressure ◽  
Venous Hypertension ◽  
Plastic Bronchitis ◽  
Protein Losing Enteropathy ◽  
Fontan Physiology

Protein-losing enteropathy and plastic bronchitis remain challenging to treat despite recent treatment advances. Protein-losing enteropathy and plastic bronchitis have been diagnosed in patients with cardiomyopathy, constrictive pericarditis, and congestive heart failure. This article focuses on patients with protein-losing enteropathy or plastic bronchitis following the Fontan procedure. Patients with single-ventricle physiology who have undergone the Fontan procedure are at risk for these conditions. Fontan physiology predisposes patients to chronically low cardiac output, increased central venous pressure, and congestive heart failure. These altered hemodynamics lead to increased mesenteric vascular resistance, resulting in venous hypertension and congestion in protein-losing enteropathy. Plastic bronchitis is a complex disease in which chronic high lymphatic pressures from Fontan physiology cause acellular bronchial casts to develop. These entities may also occur in patients with normal Fontan hemodynamics. This article also covers medical and surgical interventions for protein-losing enteropathy and plastic bronchitis. (Critical Care Nurse. 2018;38[6]:e5–e12)

Comparison of Iodixanol and Iopromide in Patients With Renal Insufficiency and Congestive Heart Failure Undergoing Coronary Angiography by Hemodynamic Monitoring

Angiology ◽  
2017 ◽  
Vol 68 (10) ◽  
pp. 907-913 ◽  
Author(s):  
Geng Qian ◽  
Yong-qiang Yang ◽  
Wei Dong ◽  
Feng Cao ◽  
Yun-dai Chen
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Contrast Media ◽  
Hemodynamic Monitoring ◽  
Venous Pressure ◽  
Cardiac Preload ◽  
Cardiac Events ◽  
Lung Water ◽  
Water Index ◽  
The Impact

We investigated the impact of contrast media (CM) with different osmolality on cardiac preload in patients with chronic kidney disease (CKD) and congestive heart failure (CHF). Patients with CKD and CHF were equally randomized to receive either iso-osmolar contrast media (IOCM) iodixanol or low-osmolar contrast media iopromide. We measured cardiac preload indexes by invasive hemodynamic monitoring before and after CM injection. Major adverse cardiac events postprocedures were recorded. Increase in extravascular lung water index was only seen in the iopromide group ( P < .001), while global end diastolic index and central venous pressure were all significantly increased from baseline in the both groups ( P < .001, respectively), and the increase in cardiac preload indexes was significantly greater in the iopromide group than in the iodixanol group ( P < 0.001). The overall incidence of acute heart failure was more frequently observed in the iopromide group ( P = 0.027). Low-osmolar contrast media iopromide significantly increased cardiac preload in patients with CKD and CHF undergoing cardiac catheterization procedures compared with IOCM iodixanol.

Ascites Formation Without Sodium Intake in Dogs With Thoracic Inferior Vena Cava Constriction and in Dogs With Right-Sided Congestive Heart Failure

1957 ◽  
Vol 188 (3) ◽  
pp. 578-582 ◽  
Author(s):  
Wilmot C. Ball ◽  
James O. Davis ◽  
M. Jay Goodkind
Keyword(s):  
Heart Failure ◽  
Congestive Heart Failure ◽  
Inferior Vena Cava ◽  
Inferior Vena ◽  
Sodium Intake ◽  
Venous Pressure ◽  
Main Pulmonary Artery ◽  
Vena Cava ◽  
Edema Formation ◽  
Secondary Phenomenon

Dogs were subjected to constriction of the thoracic inferior vena cava or main pulmonary artery and were deprived of food for a period of 4 days thereafter. In association with a high venous pressure, 60% of the dogs formed ascites despite the absence of Na intake. Measurements of T-1824 dye space provided evidence that plasma volume was not increased. An increase in urinary excretion of aldosterone was associated with a markedly reduced urinary Na excretion. The data are interpreted as supporting the concept that elevated venous pressure initiates edema formation in congestive heart failure and that renal Na retention occurs as a secondary phenomenon.

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