scholarly journals Reduced sodium-proton exchange activity in lymphocytes from transgenic rats.

Hypertension ◽  
1994 ◽  
Vol 24 (3) ◽  
pp. 357-361 ◽  
Author(s):  
M Tepel ◽  
T Klaus ◽  
S Laukemper ◽  
W Zidek
Keyword(s):  
Proton Exchange ◽  
Transgenic Rats ◽  
Sodium Proton Exchange ◽  
Exchange Activity

scholarly journals Glultamate 85 Is Involved in the Sodium/Proton Exchange Activity of theEscherichia coliChaA

2010 ◽  
Vol 74 (5) ◽  
pp. 1116-1119 ◽  
Author(s):  
Fuminori FUKAYA ◽  
Kimihiro TANAKA ◽  
Rungaroon WADITEE ◽  
Yoshito TANAKA ◽  
Tatsunosuke NAKAMURA ◽  
...  
Keyword(s):  
Proton Exchange ◽  
Sodium Proton Exchange ◽  
Exchange Activity

scholarly journals Lymphocytes Intracellular PH and Sodium-proton Exchange Activity in Stroke-prone Spontaneously Hypertensive Rats

1991 ◽  
Vol 32 (4) ◽  
pp. 595-595
Author(s):  
Li Di-Yuan ◽  
Ayame Kobayashi ◽  
Yasuo Nara ◽  
Katsumi Ikeda ◽  
Chuzo Mori ◽  
...  
Keyword(s):  
Intracellular Ph ◽  
Spontaneously Hypertensive Rats ◽  
Proton Exchange ◽  
Hypertensive Rats ◽  
Spontaneously Hypertensive ◽  
Sodium Proton Exchange ◽  
Exchange Activity

Sodium/Proton Exchange in the Maintenance of Intracellular pH in FRTL-5 Thyroid Cells*

Endocrinology ◽  
1988 ◽  
Vol 123 (4) ◽  
pp. 1705-1711 ◽  
Author(s):  
CLAUDIO MARCOCCI ◽  
EVELYN F. GROLLMAN
Keyword(s):  
Intracellular Ph ◽  
Proton Exchange ◽  
Thyroid Cells ◽  
Sodium Proton Exchange

Pancreatic beta-cell electrical activity: the role of anions and the control of pH

1983 ◽  
Vol 244 (3) ◽  
pp. C188-C197 ◽  
Author(s):  
G. T. Eddlestone ◽  
P. M. Beigelman
Keyword(s):  
Membrane Potential ◽  
Cell Membrane ◽  
Beta Cell ◽  
Pancreatic Beta Cell ◽  
Proton Exchange ◽  
Disulfonic Acid ◽  
Control Mechanisms ◽  
Exchange System ◽  
Sodium Proton Exchange

The influence of chloride on the mouse pancreatic beta-cell membrane potential and the cell membrane mechanisms controlling intracellular pH (pHi) have been investigated using glass microelectrodes to monitor the membrane potential. It has been shown that chloride is distributed passively across the beta-cell membrane such that chloride potential is equal to the membrane potential. Withdrawal of perifusate chloride or bicarbonate and the application of the drugs 4-acetamido-4'-isethiocyanostilbene-2,2'-disulfonic acid (SITS) and probenecid, both blockers of transmembrane anion movement, have been used to establish that a chloride-bicarbonate exchange system is operative in the cell membrane and that it is one of the control mechanisms of pHi. Amiloride, a specific blocker of the transmembrane sodium proton exchange, has been used to demonstrate that this mechanism is also operative in the beta-cell membrane in the control of pHi. The hypothesis that the calcium-activated potassium permeability is proton sensitive at an intracellular site, a fall in pHi causing a fall in permeability and an increase in pHi causing an increase in permeability, has been used to explain many of the effects observed in this study.

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