Renal oncocytoma: Personal experience

1992 ◽  
Vol 59 (1_suppl) ◽  
pp. 156-159
Author(s):  
S. Invernizzi ◽  
D. Pozza ◽  
G. Longo ◽  
S. Cappoli ◽  
G. Locatelli ◽  
...  

The Authors report their experience on kidney oncocytomas. They found 6 cases of kidney oncocytoma in 140 cases of kidney tumors. In all cases histological slides of neoplastic masses were made with traditional stains, PAS and PAS diastase and with immunohistochemical reaction for keratins (BDK) and vimentin (DAKO clone V9). They describe one of the six cases in which the diagnosis of oncocitary G3 type carcinoma was made, and another of oncocytomatosis with borderline aspects versus oncocytomal renal cell carcinoma.

2004 ◽  
Vol 28 (8) ◽  
pp. 1045-1050 ◽  
Author(s):  
Kurt T Patton ◽  
Maria S Tretiakova ◽  
Jorge L Yao ◽  
Veronica Papavero ◽  
Lei Huo ◽  
...  

2006 ◽  
Vol 130 (12) ◽  
pp. 1865-1870 ◽  
Author(s):  
Brian P. Adley ◽  
Norm D. Smith ◽  
Ritu Nayar ◽  
Ximing J. Yang

Abstract Context.—Birt-Hogg-Dubé (BHD) syndrome is a rare clinicopathologic condition transmitted in an autosomal dominant fashion. This complex entity is characterized by cutaneous fibrofolliculomas, kidney tumors, pulmonary cysts, and spontaneous pneumothorax. Recently, the gene possibly responsible for the clinical manifestations of BHD syndrome has been cloned and characterized. The few reviews of BHD syndrome found in the English literature mostly focus on the skin lesions or genetics, with limited information on other pathologic changes, particularly the kidney lesions. Objective.—To review the literature on this subject with a special emphasis on BHD syndrome-associated renal pathology as well as recent advances in molecular genetic discovery of the BHD syndrome. Data Sources.—We used all data available after performing a literature search using MEDLINE and searching under the headings “Birt-Hogg-Dubé,” “hybrid oncocytic tumors,” and “folliculin.” Conclusions.—The presence of BHD syndrome should be investigated in any patient with multiple bilateral kidney tumors, especially if the predominant histologic type is chromophobe renal cell carcinoma or the so-called hybrid oncocytic tumor. The genetic alteration for BHD syndrome has been mapped to chromosome 17p12q11, and the gene in this region has been cloned and believed to be responsible for the BHD syndrome. The function of the BHD product, called folliculin, is still unknown, although it is speculated to be a tumor suppressor gene. Numerous mutations have been described in the BHD gene. Studies are ongoing to determine the relationship between the BHD gene and development of sporadic renal cell carcinoma and other lesions.


Pathology ◽  
2014 ◽  
Vol 46 ◽  
pp. S135-S136
Author(s):  
Faruk Skenderi ◽  
Monika Ulamec ◽  
Semir Vranić ◽  
Nurija Bilalović ◽  
Kvetoslava Peckova ◽  
...  

2014 ◽  
Vol 8 (11-12) ◽  
pp. 928 ◽  
Author(s):  
Cevahir Özer ◽  
Mehmet Resit Gören ◽  
Tulga Egilmez ◽  
Nebil Bal

Renal oncocytomas accounts for 3% to 9% of primary renal neoplasms. The coexistence of renal cell carcinoma (RCC) within the oncocytoma is extremely rare. We report the case of an asymptomatic 74-year-old man with papillary RCC within oncocytoma managed with left radical nephrectomy.


2019 ◽  
Vol 8 (S2) ◽  
pp. S123-S137
Author(s):  
Keng Lim Ng ◽  
Robert J. Ellis ◽  
Hemamali Samaratunga ◽  
Christudas Morais ◽  
Glenda C. Gobe ◽  
...  

2009 ◽  
Vol 456 (1) ◽  
pp. 85-89 ◽  
Author(s):  
Alma Demirović ◽  
Sanja Cesarec ◽  
Borislav Spajić ◽  
Davor Tomas ◽  
Stela Bulimbašić ◽  
...  

2016 ◽  
Vol 69 (8) ◽  
pp. 661-671 ◽  
Author(s):  
Keng Lim Ng ◽  
Christudas Morais ◽  
Anne Bernard ◽  
Nicholas Saunders ◽  
Hemamali Samaratunga ◽  
...  

2008 ◽  
Vol 52 (3) ◽  
pp. 277-282 ◽  
Author(s):  
K T Mai ◽  
I Teo ◽  
E C Belanger ◽  
S J Robertson ◽  
E C Marginean ◽  
...  

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