scholarly journals The Role Of The Lactate Level In Determining The Risk Rates Of Small Bowel Resection In Incarcerated Hernias

2020 ◽  
Author(s):  
Mutlu Sahin
Keyword(s):  
Small Bowel ◽  
Lactate Level ◽  
Bowel Resection ◽  
Small Bowel Resection

The role of apoptosis during intestinal adaptation after small bowel resection

2000 ◽  
Vol 35 (1) ◽  
pp. 20-24 ◽  
Author(s):  
Carlo F.M. Welters ◽  
Femke E. Piersma ◽  
David M. Hockenbery ◽  
Erik Heineman Maastricht
Keyword(s):  
Small Bowel ◽  
Bowel Resection ◽  
Intestinal Adaptation ◽  
Small Bowel Resection

Role of luminal nutrients and endogenous GLP-2 in intestinal adaptation to mid-small bowel resection

2003 ◽  
Vol 284 (4) ◽  
pp. G670-G682 ◽  
Author(s):  
Elizabeth M. Dahly ◽  
Melanie B. Gillingham ◽  
Ziwen Guo ◽  
Sangita G. Murali ◽  
David W. Nelson ◽  
...  
Keyword(s):  
Small Bowel ◽  
Adaptive Response ◽  
Bowel Resection ◽  
Intestinal Adaptation ◽  
Small Bowel Resection ◽  
Oral Feeding ◽  
Enterocyte Proliferation ◽  
Glucagon Like Peptide ◽  
Mucosal Growth

To elucidate the role of luminal nutrients and glucagon-like peptide-2 (GLP-2) in intestinal adaptation, rats were subjected to 70% midjejunoileal resection or ileal transection and were maintained with total parenteral nutrition (TPN) or oral feeding. TPN rats showed small bowel mucosal hyperplasia at 8 h through 7 days after resection, demonstrating that exogenous luminal nutrients are not essential for resection-induced adaptation when residual ileum and colon are present. Increased enterocyte proliferation was a stronger determinant of resection-induced mucosal growth in orally fed animals, whereas decreased apoptosis showed a greater effect in TPN animals. Resection induced significant transient increases in plasma bioactive GLP-2 during TPN, whereas resection induced sustained increases in plasma GLP-2 during oral feeding. Resection-induced adaptive growth in TPN and orally fed rats was associated with a significant positive correlation between increases in plasma bioactive GLP-2 and proglucagon mRNA expression in the colon of TPN rats and ileum of orally fed rats. These data support a significant role for endogenous GLP-2 in the adaptive response to mid-small bowel resection in both TPN and orally fed rats.

scholarly journals The role of enteral fat as a modulator of body composition after small bowel resection

Surgery ◽  
2014 ◽  
Vol 156 (2) ◽  
pp. 412-418 ◽  
Author(s):  
Pamela M. Choi ◽  
Raphael C. Sun ◽  
Joshua Sommovilla ◽  
Jose Diaz-Miron ◽  
Jaclyn Khil ◽  
...  
Keyword(s):  
Body Composition ◽  
Small Bowel ◽  
Bowel Resection ◽  
Small Bowel Resection

Su1303 - The Role of Aryl Hydrocarbon Receptor Pathway after Small Bowel Resection

2018 ◽  
Vol 154 (6) ◽  
pp. S-1306
Author(s):  
Emily J. Onufer ◽  
Kristen Seiler ◽  
David M. Alvarado ◽  
Misty L. Good ◽  
Matthew A. Ciorba ◽  
...  
Keyword(s):  
Small Bowel ◽  
Aryl Hydrocarbon Receptor ◽  
Bowel Resection ◽  
Small Bowel Resection ◽  
Aryl Hydrocarbon

scholarly journals The role of humoral factors and the ileocecal valve in pathological changes occurring after distal small bowel resection.

1987 ◽  
Vol 151 (2) ◽  
pp. 155-168 ◽  
Author(s):  
MIKIO IMAMURA ◽  
MORIHIKO TODA ◽  
YOSHIROO SUZUKI ◽  
IWAO SASAKI ◽  
TOSHIO SATO ◽  
...  
Keyword(s):  
Small Bowel ◽  
Bowel Resection ◽  
Ileocecal Valve ◽  
Small Bowel Resection ◽  
Pathological Changes ◽  
Humoral Factors ◽  
Distal Small Bowel

scholarly journals Osteoporosis of Crohn’s Disease: A Critical Review

1996 ◽  
Vol 10 (5) ◽  
pp. 317-321
Author(s):  
Neena S Abraham ◽  
Richard N Fedorak
Keyword(s):  
Vitamin D ◽  
Crohn’S Disease ◽  
Bone Density ◽  
Small Bowel ◽  
Crohn's Disease ◽  
Bowel Resection ◽  
Small Bowel Resection ◽  
Treatment Of Osteoporosis ◽  
Present Understanding

Osteoporosis has long been a recognized complication of Crohn’s disease (CD), with a documented incidence ranging from 31% to 65%. The cause of osteoporosis in Crohn’s patients is likely multifactorial; corticosteroids, inflammatory cytokines, small bowel resection and the resultant calcium and vitamin D deficiencies, hypogonadism, malnutrition and the cachexia of inflammation all play a role. However, the mechanism responsible for osteoporosis associated with CD remains unclear. Treatment of decreased bone density in CD patients has been limited to calcium and vitamin D replacement. The present understanding of the pathophysiology, mechanism and treatment of osteoporosis in CD is reviewed, with the focus on the role of steroid-induced osteoporosis and the use of bisphosphonates.

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