scholarly journals Clinical Significance of Adrenal Computed Tomography in Addison's Disease.

1992 ◽  
Vol 39 (6) ◽  
pp. 563-569 ◽  
Author(s):  
ZHONG HUA SUN ◽  
KAORU NOMURA ◽  
SHOHZOH TORAYA ◽  
MAKOTO UJIHARA ◽  
NOBUO HORIBA ◽  
...  
Keyword(s):  
Computed Tomography ◽  
Clinical Significance ◽  
Addison’S Disease ◽  
Addison's Disease

scholarly journals Addison's Disease Revisited in Poland: Year 2008 versus Year 1990

2010 ◽  
Vol 2010 ◽  
pp. 1-5 ◽  
Author(s):  
Anna A. Kasperlik-Zaluska ◽  
Barbara Czarnocka ◽  
Wojciech Jeske ◽  
Lucyna Papierska
Keyword(s):  
Computed Tomography ◽  
Addison’S Disease ◽  
Addison's Disease ◽  
Immunofluorescence Technique ◽  
Imaging Studies ◽  
Immunofluorescence Method ◽  
Modern Technique ◽  
Indirect Immunofluorescence Technique ◽  
Group A ◽  
Group B

This study aimed at comparing two groups of patients with Addison's disease: A, including 180 patients described in 1991 and B, consisting of 138 patients registered since 1991. The incidence of coexisting autoimmune disorders was evaluated and etiological factors were analyzed. Immunological and imaging studies (computed tomography in group B) were performed. Adrenal autoantibodies were examined by an indirect immunofluorescence technique in group A, and by the assay measuring autoantibodies against steroid 21-hydroxylase in group B. Adrenal autoantibodies were revealed in 37% of patients examined by the immunofluorescence method and in 63% investigated by the modern technique. Tuberculosis was found in 52 patients in the group A and in two patients in the group B; metastatic infiltrations of the adrenals in CT were detected in 16 patients. Probable autoimmune Addison's disease was diagnosed in 125/180 patients (69%) in the group A and in 116/138 patients (84%) in the group B.

Computed tomography morphology of the adrenal glands of patients with Addison's disease

1996 ◽  
Vol 40 (1) ◽  
pp. 38-42 ◽  
Author(s):  
AC Ammini ◽  
R Gupta ◽  
C Mukopadhyay ◽  
P Shah ◽  
MS Sandhu ◽  
...  
Keyword(s):  
Computed Tomography ◽  
Adrenal Glands ◽  
Addison’S Disease ◽  
Addison's Disease

scholarly journals Addison’s disease due to bilateral adrenal tuberculosis on 18F-fluorodeoxyglucose positron emission tomography computed tomography

2018 ◽  
Vol 10 (2) ◽  
Author(s):  
Sung Ae Koh
Keyword(s):  
Computed Tomography ◽  
Positron Emission Tomography ◽  
Adrenal Glands ◽  
Addison’S Disease ◽  
Emission Tomography ◽  
Addison's Disease ◽  
Fluorodeoxyglucose Positron Emission Tomography ◽  
Caseous Necrosis ◽  
Positron Emission ◽  
Fluorodeoxyglucose Positron Emission

We present a case of a 60-year-old woman diagnosed with disseminated tuberculosis with bilateral adrenalitis resulting in Addison’s disease. The 18-fluorodeoxyglucose (18-FDG) positron emission tomography computed tomography (CT) was performed, which revealed increased FDG uptake in the neck, mediastinal, and abdominal lymph nodes, and both adrenal glands, similar to the lesions noted on CT. We suspected the patient to have a lymphoma; therefore, axillary biopsy was performed, which revealed chronic granulomatous lesion with focal caseous necrosis.

scholarly journals Residual adrenal function in Addison’s disease

2020 ◽  
Author(s):  
Simon Hs Pearce ◽  
Earn H Gan ◽  
Catherine Napier
Keyword(s):  
Stem Cell ◽  
Clinical Significance ◽  
Disease Duration ◽  
Addison’S Disease ◽  
Adrenal Function ◽  
Stem Cell Population ◽  
Addison's Disease ◽  
Adrenal Failure ◽  
Adrenal Steroidogenesis ◽  
The Moment

Over the last ten years, evidence has accumulated that autoimmune Addison’s disease (AAD) is a heterogeneous disease. Residual adrenal function, characterised by persistent secretion of cortisol, other glucocorticoids and mineralocorticoids is present in around 30% of patients with established AAD, and appears commoner in men. This persistent steroidogenesis is present in some patients with AAD for more than 20 years, but it is commoner in people with shorter disease duration. The clinical significance of residual adrenal function is not fully clear at the moment, but as it signifies an intact adrenocortical stem cell population, it opens up the possibility of regeneration of adrenal steroidogenesis and improvement in adrenal failure for some patients.

Addison's disease as a rare cause of chronically elevated transaminases

2004 ◽  
Vol 42 (05) ◽  
Author(s):  
GC Gurakuqi ◽  
V Stadlbauer ◽  
H Warnkross ◽  
M Trauner
Keyword(s):  
Addison’S Disease ◽  
Addison's Disease ◽  
Elevated Transaminases
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