Dicyclomine in the Sudden Infant Death Syndrome (SIDS)—A Cause of Death or an Incidental Finding?

1986 ◽  
Vol 31 (4) ◽  
pp. 11927J ◽  
Author(s):  
Brad Randall ◽  
Gordon Gerry ◽  
Frank Rance
Keyword(s):  
Cause Of Death ◽  
Sudden Infant Death Syndrome ◽  
Infant Death ◽  
Incidental Finding ◽  
Sudden Infant Death ◽  
Sudden Infant

Sudden infant death syndrome as a socially determined cause of death

1989 ◽  
Vol 36 (1-2) ◽  
pp. 1-8 ◽  
Author(s):  
Charles B. Nam ◽  
Isaac W. Eberstein ◽  
Larry C. Deeb
Keyword(s):  
Cause Of Death ◽  
Sudden Infant Death Syndrome ◽  
Infant Death ◽  
Sudden Infant Death ◽  
Sudden Infant

Renal Developmental Delay Expressed by Reduced Glomerular Number and Its Association with Growth Retardation in Victims of Sudden Infant Death Syndrome and in “Normal” Infants

2000 ◽  
Vol 3 (5) ◽  
pp. 450-454 ◽  
Author(s):  
Darren J. Beech ◽  
Paul D. Sibbons ◽  
C. Vyvyan Howard ◽  
Dick van Velzen
Keyword(s):  
Birth Weight ◽  
Cause Of Death ◽  
Sudden Infant Death Syndrome ◽  
Growth Retardation ◽  
Infant Death ◽  
Sudden Infant Death ◽  
Kidney Cortex ◽  
Sudden Infant ◽  
Cortical Volume ◽  
Glomerular Number

In victims of sudden infant death syndrome (SIDS), renal development has been reported to be significantly impaired. In the present study, we used stereological techniques to estimate volume of kidney cortex and total number of glomeruli in a group of human infants. Infants were classified according to cause of death—SIDS or non-SIDS. Cases were further subdivided according to birth weight—normal birth weight (NBW) or low birth weight (LBW) (we were unable to identify any non-SIDS LBW infants for our study). No significant differences were found between NBW and LBW infants (irrespective of cause of death) for cortical volume, glomerular density, or total glomerular number ( p > 0.140). Kidney cortical volume, glomerular density, and total glomerular number were not significantly different between SIDS and non-SIDS infants (p > 0.510). Glomerular number was only significantly less in SIDS infants of LBW (p = 0.032) than in controls according to the Wilcoxon rank sum test; using the Kruskal-Wallis for one-way analysis, no significant difference was found (p > 0.010). These results contrast with those from previous studies, as a reduction in glomerular number was not noted in SIDS NBW infants, and the mean value for the control (non-SIDS NBW) group was significantly reduced ( p < 0.01) from those of previous studies. This indicates that glomerular number reduction is seen in SIDS NBW and non-SIDS NBW cases and is therefore directly associated with growth retardation rather than with SIDS.

THE SUDDEN INFANT DEATH SYNDROME

PEDIATRICS ◽  
1972 ◽  
Vol 50 (6) ◽  
pp. 964-965
Author(s):  
William B. Forsyth ◽  
John E. Allen ◽  
Joseph W. Brinkley ◽  
Alice D. Chenoweth ◽  
Gertrude Hunter ◽  
...  
Keyword(s):  
Cause Of Death ◽  
Sudden Infant Death Syndrome ◽  
Infant Death ◽  
Sudden Infant Death ◽  
The United States ◽  
Sudden Infant ◽  
Infant Deaths ◽  
Lower Socioeconomic ◽  
Cot Death ◽  
The Family

The finding of an apparently healthy infant dead in his crib is as big a shock to physicians as to the parents. Sudden infant death syndrome (SIDS) is a definite entity that cannot be predicted and, therefore, cannot be prevented. Commonly known as "crib death" or "cot death," SIDS causes approximately 10,000 infant deaths each year in the United States. It is responsible for approximately three deaths of every 1,000 births. After the first week of life, SIDS is the most important single cause of death of infants under 1 year of age; it ranks second only to accidents as the greatest cause of death in children less than 15 years of age. It is more frequent in winter months, has a higher incidence in males and among lower socioeconomic groups, and has a peak incidence at 3 months of age. SIDS can he confirmed, but not explained, at autopsy by changes in the tissues of the respiratory system and the absence of other lethal lesions. This statement was prepared to provide guidelines for the pediatrician for his contact with the family and the agencies in the community when SIDS occurs. Because guilt feelings are usually associated with SIDS, the parents will need counsel and support to help alleviate these feelings, especially when they begin to wonder what they did wrong. The family should be encouraged to talk freely about their feelings, and they can be told about the usual reactions to losing a child. The counseling given initially may not be effective because of the emotional shock immediately following the infant's death; therefore, the pediatrician should again meet with the family a day or two after the death.

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