scholarly journals Pregnancy following Brown-Séquard syndrome: A rare case report

2021 ◽  
Vol 8 (4) ◽  
pp. 572-576
Author(s):  
Shree Bharathi ◽  
Niveditha Jha ◽  
Sasirekha Rengaraj ◽  
Veena Ranjan
Keyword(s):  
Spinal Cord ◽  
Past History ◽  
Breech Presentation ◽  
Multidisciplinary Care ◽  
Spinal Cord Lesion ◽  
Lower Limbs ◽  
Sensory Recovery ◽  
Brown Sequard Syndrome ◽  
Cord Lesion

Brown-Séquard syndrome is an incomplete spinal cord lesion characterized by hemisection injury of the cord. We present a case of pregnancy, delivery and postpartum course following this rare neurological condition. A 42-year-old woman presented with past history of idiopathic hemicord myelitis leading to right sided hemiplegia with decreased contralateral sensation of pain and temperature, consistent with Brown-Séquard syndrome, which was treated with steroids and Therapeutic Plasma Exchange. Thereafter, she had near-complete motor recovery and complete sensory recovery over the next 3months. Three years later, she presented to us at 37+2 weeks of gestation with residual hemiparesis with motor power grade of 4/5 in right upper and lower limbs. She underwent Caesarean section for breech presentation, which was done under general anaesthesia in view of prior spinal cord lesion. She was discharged for follow-up in Neurology outpatient clinic and physical rehabilitation. At follow up after 12 months of delivery, she had complete motor and sensory recovery. Management of spinal cord lesions in pregnancy and delivery requires specialist multidisciplinary care due to risk of medical and obstetric complications. This case demonstrates a rare scenario of a primigravida at term gestation with residual deficits of a past spinal cord lesion.

A DTI study of the spinal cord lesion in patients with multiple sclerosis during the follow-up after relapse

2016 ◽  
Vol 116 (2. Vyp. 2) ◽  
pp. 21 ◽  
Author(s):  
S. N. Morozova ◽  
V. V. Bryukhov ◽  
O. V. Trifonova ◽  
E. I. Kremneva ◽  
M. V. Krotenkova
Keyword(s):  
Multiple Sclerosis ◽  
Spinal Cord ◽  
Spinal Cord Lesion ◽  
Cord Lesion

Thoracic meningomyelocele associated with spina bifida in a Malinois dog

2019 ◽  
Vol 7 (3) ◽  
pp. e000894
Author(s):  
Clément Musso ◽  
Camille Bismuth ◽  
Laurent Cauzinille
Keyword(s):  
Spinal Cord ◽  
Spina Bifida ◽  
Spinal Cord Lesion ◽  
Fistula Tract ◽  
Clear Fluid ◽  
Ct Myelography ◽  
Spina Bifida Aperta ◽  
Healing Wound ◽  
Cord Lesion

An 8-month-old male Malinois dog was presented for progressive chronic pelvic limbs ataxia, paraparesis, arched back since ambulation associated with mild urinary and faecal incontinence. Clinical evaluation revealed a dorsocaudal thoracic spine non-healing wound with a fistula from which a clear fluid leaked. Neurological examination was compatible with a T3-L3 spinal cord lesion. CT myelography showed a T11-T12 spina bifida associated with a meningomyelocele with a fistula tract to the skin (spina bifida aperta). An MRI revealed a syringohydromyelia cranially and caudally to the meningomyelocele. Surgical correction involved removal of the fistula up to the dura mater and closure. A 4 and 12 months clinical and MRI follow up revealed a good locomotion improvement with residual mild ataxia without incontinence. MRI showed no relapse of the meningomyelocele but persistent although reduced syringohydromyelia.

Subdural haematoma, the great imitator, mimicking acute spinal cord lesion

2021 ◽  
Vol 14 (4) ◽  
pp. e240329
Author(s):  
Kensuke Shiga ◽  
Aiko Tamura
Keyword(s):  
Spinal Cord ◽  
Urinary Incontinence ◽  
Subdural Haematoma ◽  
Spinal Cord Lesion ◽  
Lower Limbs ◽  
Sensory Level ◽  
Thoracic Myelopathy ◽  
Cranial Ct ◽  
Cortical Pathology ◽  
Cord Lesion

Acute non-traumatic paraparesis is usually caused by vascular, inflammatory or neoplastic myelopathies; however, it is sometimes caused by non-myelopathic pathologies, including polyradiculoneuropathies, myopathies, psychogenic aetiologies or parasagittal cortical pathologies. A 73-year-old woman reported weakness of the bilateral lower limbs and urinary incontinence. Together with the sensory level at the left T6 dermatome, we initially considered thoracic myelopathy as the most likely diagnosis. However, MRI of the cervicothoracic cord was negative and subsequent cranial CT revealed a bilateral subdural haematoma. A parasagittal cortical pathology should not be excluded from differential diagnoses as a rare cause of paraparesis until its possibility is carefully ruled out.

scholarly journals Anti-amphiphysin antibody-positive paraneoplastic neurological syndrome with a longitudinally extensive spinal cord lesion of the dorsal column

2014 ◽  
Vol 54 (7) ◽  
pp. 572-576 ◽  
Author(s):  
Shuichiro Neshige ◽  
Naoyuki Hara ◽  
Shinichi Takeshima ◽  
Hirotaka Iwaki ◽  
Yutaka Shimoe ◽  
...  
Keyword(s):  
Spinal Cord ◽  
Paraneoplastic Neurological Syndrome ◽  
Dorsal Column ◽  
Spinal Cord Lesion ◽  
Neurological Syndrome ◽  
Cord Lesion

Cross-cultural adaptation of the spinal cord lesion-related coping strategies questionnaire for use in Iran

Injury ◽  
2015 ◽  
Vol 46 (8) ◽  
pp. 1539-1544 ◽  
Author(s):  
Mohsen Saffari ◽  
Amir H. Pakpour ◽  
Mohammad Yaghobidoot ◽  
Faten Al Zaben ◽  
Harold G. Koenige
Keyword(s):  
Spinal Cord ◽  
Coping Strategies ◽  
Cultural Adaptation ◽  
Spinal Cord Lesion ◽  
Cross Cultural ◽  
Cross Cultural Adaptation ◽  
Cord Lesion

Reduction of Common Synaptic Drive to Ankle Dorsiflexor Motoneurons During Walking in Patients With Spinal Cord Lesion

2005 ◽  
Vol 94 (2) ◽  
pp. 934-942 ◽  
Author(s):  
N. L. Hansen ◽  
B. A. Conway ◽  
D. M. Halliday ◽  
S. Hansen ◽  
H. S. Pyndt ◽  
...  
Keyword(s):  
Spinal Cord ◽  
Motor Unit ◽  
Healthy Subjects ◽  
Motor Units ◽  
Central Peak ◽  
Spinal Cord Lesion ◽  
Short Term ◽  
Physiological Markers ◽  
Synaptic Drive ◽  
Cord Lesion

It is possible to obtain information about the synaptic drive to motoneurons during walking by analyzing motor-unit coupling in the time and frequency domains. The purpose of the present study was to compare motor-unit coupling during walking in healthy subjects and patients with incomplete spinal cord lesion to obtain evidence of differences in the motoneuronal drive that result from the lesion. Such information is of importance for development of new strategies for gait restoration. Twenty patients with incomplete spinal cord lesion (SCL) participated in the study. Control experiments were performed in 11 healthy subjects. In all healthy subjects, short-term synchronization was evident in the discharge of tibialis anterior (TA) motor units during the swing phase of treadmill walking. This was identified from the presence of a narrow central peak in cumulant densities constructed from paired EMG recordings and from the presence of significant coherence between these signals in the 10- to 20-Hz band. Such indicators of short-term synchrony were either absent or very small in the patient group. The relationship between the amount of short-term synchrony and the magnitude of the 10- to 20-Hz coherence in the patients is discussed in relation to gait ability. It is suggested that supraspinal drive to the spinal cord is responsible for short-term synchrony and coherence in the 10- to 20-Hz frequency band during walking in healthy subjects. Absence or reduction of these features may serve as physiological markers of impaired supraspinal control of gait in SCL patients. Such markers could have diagnostic and prognostic value in relation to the recovery of locomotion in patients with central motor lesions.

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