scholarly journals The effect of epidemic disease outbreaks on the dynamic behavior of a prey-predator model with Holling type II functional response

2020 ◽  
Vol 30 (01) ◽  
pp. 2050011 ◽  
Author(s):  
Peng Yang ◽  
Yuanshi Wang

This paper is devoted to the study of a new delayed eco-epidemiological model with infection-age structure and Holling type II functional response. Firstly, the disease transmission rate function among the predator population is treated as the piecewise function concerning the incubation period [Formula: see text] of the epidemic disease and the model is rewritten as an abstract nondensely defined Cauchy problem. Besides, the prerequisite which guarantees the presence of the coexistence equilibrium is achieved. Secondly, via utilizing the theory of integrated semigroup and the Hopf bifurcation theorem for semilinear equations with nondense domain, it is found that the model exhibits a Hopf bifurcation near the coexistence equilibrium, which suggests that this model has a nontrivial periodic solution that bifurcates from the coexistence equilibrium as the bifurcation parameter [Formula: see text] crosses the bifurcation critical value [Formula: see text]. That is, there is a continuous periodic oscillation phenomenon. Finally, some numerical simulations are shown to support and extend the analytical results and visualize the interesting phenomenon.


2021 ◽  
Vol 0 (0) ◽  
Author(s):  
Dipankar Ghosh ◽  
Prasun K. Santra ◽  
Abdelalim A. Elsadany ◽  
Ghanshaym S. Mahapatra

Abstract This paper focusses on developing two species, where only prey species suffers by a contagious disease. We consider the logistic growth rate of the prey population. The interaction between susceptible prey and infected prey with predator is presumed to be ruled by Holling type II and I functional response, respectively. A healthy prey is infected when it comes in direct contact with infected prey, and we also assume that predator-dependent disease spreads within the system. This research reveals that the transmission of this predator-dependent disease can have critical repercussions for the shaping of prey–predator interactions. The solution of the model is examined in relation to survival, uniqueness and boundedness. The positivity, feasibility and the stability conditions of the fixed points of the system are analysed by applying the linearization method and the Jacobian matrix method.


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