Polarographic Study of Cortical Blood Flow in Man

1957 ◽  
Vol 14 (4) ◽  
pp. 382-399 ◽  
Author(s):  
John S. Meyer ◽  
John Hunter
Keyword(s):  
Blood Flow ◽  
Polarographic Study ◽  
Cortical Blood Flow

Effects of Ethanol on EEG and Cortical Blood Flow in the Cat

1968 ◽  
Vol 29 (4) ◽  
pp. 828-838 ◽  
Author(s):  
A. A. Hadji-Dimo ◽  
R. Ekberg ◽  
D. H. Ingvar
Keyword(s):  
Blood Flow ◽  
Cortical Blood Flow

Cortical Blood Flow and Negative Symptoms in Schizophrenia

1996 ◽  
Vol 33 (3) ◽  
pp. 127-131 ◽  
Author(s):  
Zvi Zemishlany ◽  
Gene E. Alexander ◽  
Isak Prohovnik ◽  
Ron G. Goldman ◽  
Sukdeb Mukherjee ◽  
...  
Keyword(s):  
Blood Flow ◽  
Negative Symptoms ◽  
Cortical Blood Flow

Music and cortical blood flow: A functional near-infrared spectroscopy (fNIRS) study.

2014 ◽  
Vol 7 (4) ◽  
pp. 545-550 ◽  
Author(s):  
Marcelo Bigliassi ◽  
Vinícius Barreto-Silva ◽  
Thiago Ferreira Dias Kanthack ◽  
Leandro Ricardo Altimari
Keyword(s):  
Blood Flow ◽  
Infrared Spectroscopy ◽  
Near Infrared Spectroscopy ◽  
Near Infrared ◽  
Functional Near Infrared Spectroscopy ◽  
Cortical Blood Flow

Patients with chronic fatigue syndrome have reduced absolute cortical blood flow

2006 ◽  
Vol 26 (2) ◽  
pp. 83-86 ◽  
Author(s):  
Kazuhiro Yoshiuchi ◽  
Jeffrey Farkas ◽  
Benjamin H. Natelson
Keyword(s):  
Blood Flow ◽  
Chronic Fatigue Syndrome ◽  
Chronic Fatigue ◽  
Cortical Blood Flow ◽  
Fatigue Syndrome

Autoradiographic Evidence for Flow-Metabolism Uncoupling During Stimulation of the Nucleus Basalis of Meynert in the Conscious Rat

1997 ◽  
Vol 17 (6) ◽  
pp. 686-694 ◽  
Author(s):  
Elvire Vaucher ◽  
Josiane Borredon ◽  
Gilles Bonvento ◽  
Jacques Seylaz ◽  
Pierre Lacombe
Keyword(s):  
Blood Flow ◽  
Electrical Stimulation ◽  
Metabolic Activity ◽  
Nucleus Basalis ◽  
Nucleus Basalis Of Meynert ◽  
Conscious Rat ◽  
Local Flow ◽  
Cortical Blood Flow ◽  
Subcortical Regions ◽  
Stimulation Of

We earlier reported that electrical stimulation of the rat nucleus basalis of Meynert (NBM) induces large cerebral blood flow increases, particularly in frontal cortical areas but also in some subcortical regions. The present study was designed to address the issue of blood flow control exerted by NBM projections. To this aim, we have determined whether these flow increases were associated with proportionate changes in metabolic activity as evaluated by cerebral glucose utilization (CGU) strictly under the same experimental conditions in the conscious rat. An electrode was chronically implanted in a reactive site of the NBM as determined by laser-Doppler flowmetry (LDF) of the cortical circulation. One to two weeks later, while the cortical blood flow was monitored by LDF, we measured CGU using the [14C]2-deoxyglucose autoradiographic technique during unilateral electrical stimulation of the NBM, and analyzed the local flow-metabolism relationship. The large increases in cortical blood flow induced by NBM stimulation, exceeding 300% in various frontal areas, were associated with at most 24% increases in CGU (as compared with the control group) in one frontal area. By contrast, strong increases in CGU exceeding 150% were observed in subcortical regions ipsilateral to the stimulation, especially in extrapyramidal structures, associated with proportionate CBF changes. Thus, none of the blood flow changes observed in the cortex can be ascribed to an increased metabolic activity, whereas CBF and CGU were coupled in many subcortical areas. This result indicates that different mechanisms, which do not necessarily involve any metabolic factor, contribute to the regulation of the cerebral circulation at the cortical and subcortical level. Because the distribution of the uncoupling is coincident with that of cholinergic NBM projections directly reaching cortical microvessels, these data strongly support the hypothesis that NBM neurons are capable of exerting a neurogenic control of the cortical microcirculation.

Vascular pressures and cortical blood flow in cavernous angioma of the brain

1990 ◽  
Vol 73 (4) ◽  
pp. 555-559 ◽  
Author(s):  
John R. Little ◽  
Issam A. Awad ◽  
Stephen C. Jones ◽  
Zeyd Y. Ebrahim
Keyword(s):  
Blood Flow ◽  
Cavernous Angioma ◽  
Sitting Position ◽  
Cortical Blood Flow ◽  
Content Type ◽  
Cavernous Angiomas ◽  
Arterial Blood ◽  
Mean Pressure ◽  
Local Cortical Blood Flow ◽  
The Brain

✓ This study was designed to investigate the hemodynamic characteristics of cavernous angiomas of the brain. Five adult patients with a cavernous angioma underwent local cortical blood flow studies and vascular pressure measurements during surgery for the excision of the cavernous angioma. Clinical presentation included headache in four patients, seizures in four patients, and recurring diplopia in one patient. Magnetic resonance imaging demonstrated the cavernous angiomas in all patients and revealed an associated small hematoma in two. Four patients with a cerebral cavernous angioma were operated on in the supine position and the remaining patient, whose lesion involved the brain stem, was operated on in the sitting position. Mean local cortical blood flow (± standard error of the mean) in the cerebral cortex adjacent to the lesion was 60.5 ± 8.3 ml/100 gm/min at a mean PaCO2 of 35.0 ± 0.6 torr. Mean CO2 reactivity was 1.1 ± 0.2 ml/100 gm/min/torr. The local cortical blood flow results were similar to established normal control findings. Mean pressure within the lesion in the patients undergoing surgery while supine was 38.2 ± 0.5 mm Hg; a slight decline in cavernous angioma pressure occurred with a drop in mean systemic arterial blood pressure and PaCO2. Mean pressure in the cavernous angioma in the patient operated on in the sitting position was 7 mm Hg. Jugular compression resulted in a 9-mm Hg rise in cavernous angioma pressure in one supine patient but no change in the patient in the sitting position. Direct microscopic observation revealed slow circulation within the lesions. The hemodynamic features demonstrated in this study indicate that cavernous angiomas are relatively passive vascular anomalies that are unlikely to produce ischemia in adjacent brain. Frank hemorrhage would be expected to be self-limiting because of relatively low driving pressures.

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