Membrane permeability changes in radiation-induced apoptotic mouse thymocytes

2018 ◽  
Vol 9 (3) ◽  
pp. 119
Author(s):  
KaushalaPrasad Mishra ◽  
BadriNarain Pandey
Keyword(s):  
Membrane Permeability ◽  
Permeability Changes ◽  
Radiation Induced

scholarly journals Nuclear magnetic resonanceimaging of membrane permeability changes in plants during osmoticstress

2002 ◽  
Vol 25 (11) ◽  
pp. 1539-1549 ◽  
Author(s):  
L. Van Der Weerd ◽  
M. M. A. E. Claessens ◽  
C. Efdé ◽  
H. Van As
Keyword(s):  
Membrane Permeability ◽  
Permeability Changes ◽  
Nuclear Magnetic

Study of membrane permeability changes by fluctuation analysis

Nature ◽  
1977 ◽  
Vol 270 (5636) ◽  
pp. 391-396 ◽  
Author(s):  
Charles F. Stevens
Keyword(s):  
Membrane Permeability ◽  
Fluctuation Analysis ◽  
Permeability Changes

scholarly journals Analytical Review: Leaky Red Cells

Blood ◽  
1965 ◽  
Vol 26 (3) ◽  
pp. 367-382 ◽  
Author(s):  
JAMES H. JANDL
Keyword(s):  
Membrane Permeability ◽  
Active Transport ◽  
Hereditary Spherocytosis ◽  
Metabolic Stress ◽  
Glucose Deprivation ◽  
Red Cells ◽  
Osmotic Swelling ◽  
Permeability Changes ◽  
Metabolic Conditions ◽  
Analytical Review

Abstract The normal survival of red cells requires maintained regulation of cell size and shape. This regulation is to a large extent dependent upon membrane permeability and the active transport of cations. Agents such as C' that affect permeability markedly by creating large holes in the membrane lead to rapid cell death. Most hemolytic disorders thus far studied involve lesser increases in membrane permeability and hemolysis occurs more gradually by the sequence of colloid osmotic swelling, loss of cell surface, and spherocytosis. With very mild permeability changes, as in hereditary spherocytosis, the cell may compensate for an increased leak-rate for cations by increased active transport. This compensation requires increased glycolysis and optimal metabolic conditions, however, and the cell rapidly decompensates during glucose deprivation or metabolic stress. The interaction between reticuloendothelial tissues and red cells provides such a stress for leaky cells and hastens their destruction.

Membrane permeability changes during excitation

2013 ◽  
pp. 34-48
Author(s):  
Richard D. Keynes ◽  
David J. Aidley ◽  
Christopher L.-H. Huang
Keyword(s):  
Membrane Permeability ◽  
Permeability Changes

scholarly journals Photosensitivity of the Frog Iris

1963 ◽  
Vol 46 (6) ◽  
pp. 1249-1265 ◽  
Author(s):  
Lloyd Barr ◽  
Mathew Alpern
Keyword(s):  
Membrane Permeability ◽  
Response Amplitude ◽  
Response Criteria ◽  
Half Time ◽  
Tension Development ◽  
Membrane Polarization ◽  
Permeability Changes ◽  
Unit Slope ◽  
Slope Line ◽  
Short Times

The absorption of quanta by rhodopsin leads to the contraction of frog iris muscle. The contractions reach a maximum after about 8 sec. in the light. When the light is turned off the irises relax exponentially with a half-time of about 6 sec. Membrane polarization is not necessary for the response but calcium movement and membrane permeability changes probably are. The response is not mediated by acetylcholine or epinephrine. The curves of log It vs. log t for constant response amplitude bend progressively upward away from a unit slope line at short times as larger response criteria are used because (a) light influences tension development over longer times and (b) the higher intensity, shorter duration flashes are less effective.

Membrane Permeability Changes and Ultrastructural Abnormalities Observed During Protoplast Fusion

2000 ◽  
Vol 156 (5-6) ◽  
pp. 628-634 ◽  
Author(s):  
L.F. De Filippis ◽  
R. Hampp ◽  
H. Ziegler
Keyword(s):  
Membrane Permeability ◽  
Protoplast Fusion ◽  
Permeability Changes
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