A Rare Complication of the Pulmonary Arterial Catheterization: Migration of the Catheter in Venous Cannula Within Superior Vena Cava

Author(s):  
Murat Aksun ◽  
Senem Girgin ◽  
Ufuk Yetkin ◽  
Tayfun Goktogan ◽  
Ali Gurbuz ◽  
...  
1977 ◽  
Vol 55 (6) ◽  
pp. 1369-1377 ◽  
Author(s):  
Philip J. Kadowitz ◽  
Ernst W. Spannhake ◽  
Stan Greenberg ◽  
Larry P. Feigen ◽  
Albert L. Hyman

The effects of bolus injections of the postaglandin precursor, arachidonic acid, and PGD2, PGF2α, PGE2, and the PGH2 analog ((15S)-hydroxyl-9α,11α(epoxymethano)-prosta-5Z-dienoic acid) were compared on the pulmonary circulation in the intact spontaneously breathing pentobarbital-anesthetized dog. Arachidonic acid increased pulmonary arterial pressure, decreased aortic pressure, and increased cardiac output when injected into the superior vena cava or right atrium. PGE2, like arachidonic acid, increased pulmonary arterial pressure and cardiac output and decreased aortic pressure, whereas PGF2α and PGD2 increased pulmonary arterial pressure but did not affect cardiac output or aortic pressure when injected into the superior vena cava or right atrium. The PGH2 analog increased pulmonary arterial pressure and to a lesser extent, aortic pressure, without affecting cardiac output. None of these substances changed left atrial or right atrial pressure. The cardiopulmonary effects of arachidonic acid were blocked by indomethacin whereas the rise in pulmonary arterial pressure in response to the bisenoic prostaglandins and the analog were enhanced by the cyclooxygenase inhibitor. These data suggest that the increase in pulmonary vascular resistance in response to arachidonic acid may be due to conversion of the precursor into vasoactive intermediates and products such as bisenoic prostaglandins whereas the decrease in systemic vascular resistance is probably due to the formation of PGE2 and other peripheral vasodilator substances.


2018 ◽  
Vol 35 (8) ◽  
pp. 1233-1236
Author(s):  
Mozhgan Parsaee ◽  
Hamidreza Pouraliakbar ◽  
Behshid Ghadrdoost ◽  
Jamal Moosavi ◽  
Mohaddeseh Behjati

2019 ◽  
Vol 73 (9) ◽  
pp. 2379
Author(s):  
Bao X. Nguyen ◽  
J. Patrik Hornak ◽  
Nicole A. Christians ◽  
Jianping Zhao ◽  
Judith F. Aronson ◽  
...  

2007 ◽  
Vol 11 (4) ◽  
pp. 385-391 ◽  
Author(s):  
Hadim AKOGLU ◽  
Rahmi YILMAZ ◽  
Bora PEYNIRCIOGLU ◽  
Mustafa ARICI ◽  
Alper KIRKPANTUR ◽  
...  

2017 ◽  
Vol 22 (1) ◽  
pp. 20-21
Author(s):  
Priyank Shah ◽  
Rahul Vasudev ◽  
Raja Pullatt ◽  
Fayez Shamoon

Abstract A 42-year-old woman with past medical history of intravenous drug abuse was admitted to hospital with fever and heart murmur. A peripherally inserted central catheter (PICC) was inserted because the patient had poor venous access. Transesophageal echocardiography was done to rule out infective endocarditis. The test showed thrombus attached to the PICC line. Thrombus arising from a catheter is known complication of PICCs. Classifications of right heart thromboembolism (RHTE) are based on morphology. Type A thrombi are highly mobile and may prolapse through the tricuspid valve. Conversely, type B thrombi are attached to the right atrial or ventricular wall and may originate in association with foreign bodies or in structurally abnormal chambers. RHTEs are associated with pulmonary embolism in approximately 4%–6% of cases and increase the 3-month mortality rate from 16% to 29%. On echocardiography, partial dissection of the superior vena cava (SVC) was also noted. This is a very rare complication of PICC. To the best of our knowledge this is the first reported case of PICC-induced thrombosis with partial dissection of SVC. The PICC line was removed and echocardiography postremoval did not show any thrombus. The patient remained asymptomatic without any signs of hemodynamically significant pulmonary embolism. SVC dissection was also managed conservatively. Use of central venous catheters in clinical practice is increasing but it is not a benign procedure. It may be associated with serious complications.


2002 ◽  
Vol 9 (5) ◽  
pp. 680-684 ◽  
Author(s):  
Matthias Martin ◽  
Iris Baumgartner ◽  
Martin Kolb ◽  
Jürgen Triller ◽  
Hans-Peter Dinkel

Purpose: To report a rare, fatal complication of superior vena cava Wallstent implantation. Case Report: A 59-year-old man presenting with superior vena cava syndrome caused by small-cell lung cancer underwent stent implantation of 2 kissing Wallstents >1.5 cm above the right atrium. Despite correct stent deployment, vessel perforation occurred in a section not encased by tumor, which led to fatal pericardial tamponade shortly after the procedure. Autopsy revealed perforation of a stent strut through the caval wall into the pericardial space. Anatomical and methodological reasons are discussed. Conclusions: The interventionist should be aware of this rare complication. Alternative stent designs avoiding the sharp ends of Wallstents and Palmaz stents should be considered.


2008 ◽  
Vol 43 (11) ◽  
pp. 891-892 ◽  
Author(s):  
A Abdelkefi ◽  
O Ben Gaied ◽  
S Ladeb ◽  
I Labbène ◽  
L Torjman ◽  
...  

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