scholarly journals An Unusual Case of Carbon Monoxide Poisoning from Formic and Sulfuric Acid Mixture

2019 ◽  
Vol 4 (1) ◽  
pp. 51-54
Author(s):  
Muhammed Ershad ◽  
Athanasios Melisiotis ◽  
Zachary Gaskill ◽  
Matthew Kelly ◽  
Richard Hamilton
Keyword(s):  
Carbon Monoxide ◽  
Sulfuric Acid ◽  
Formic Acid ◽  
Hyperbaric Oxygen Therapy ◽  
Oxygen Therapy ◽  
Unusual Case ◽  
Carbon Monoxide Poisoning ◽  
Acid Mixture ◽  
Neurological Sequelae

Formic acid, when combined with sulfuric acid, gets dehydrated to form carbon monoxide (CO). A 27-year-old female was found unconscious inside a car, next to a container with a mixture of sulfuric acid and formic acid. Concentrations of up to 400 parts per million of CO were measured inside the car post ventilation. Serum carboxyhemoglobin level was 15% after receiving 100% oxygen for two hours. The patient received hyperbaric oxygen therapy after which she was extubated with normal mental status. On follow-up after three months, she demonstrated neurocognitive abnormalities suggestive of delayed neurological sequelae from CO exposure.

scholarly journals Impact of Hyperbaric Oxygen Therapy on Subsequent Neurological Sequelae Following Carbon Monoxide Poisoning

2018 ◽  
Vol 7 (10) ◽  
pp. 349 ◽  
Author(s):  
Chien-Cheng Huang ◽  
Chung-Han Ho ◽  
Yi-Chen Chen ◽  
Chien-Chin Hsu ◽  
Yi-Fong Wang ◽  
...  
Keyword(s):  
Carbon Monoxide ◽  
Hyperbaric Oxygen ◽  
Hyperbaric Oxygen Therapy ◽  
Oxygen Therapy ◽  
Carbon Monoxide Poisoning ◽  
Population Based ◽  
Chronic Obstructive ◽  
Monthly Income ◽  
Neurological Sequelae ◽  
Increased Risk

The purpose of this study was to evaluate the effects of hyperbaric oxygen therapy (HBOT) on reducing neurological sequelae (NS) in patients with carbon monoxide poisoning (COP). Using a nationwide database of insurance claims in Taiwan, we conducted a population-based cohort study to identify 24,046 patients with COP diagnosed between 1999 and 2012, including 6793 (28.2%) patients who received HBOT and 17,253 (71.8%) patients who did not. We followed the two cohorts of patients and compared the occurrence of NS. The two cohorts had similar sex ratios, but patients who received HBOT were younger (34.8 ± 14.8 vs. 36.1 ± 17.2 years, p < 0.001). Patients who received HBOT had a higher risk for NS (adjusted hazard ratio [AHR]: 1.4; 95% confidence interval [CI]: 1.4–1.5), after adjusting for age, sex, underlying comorbidities (hypertension, diabetes, chronic obstructive pulmonary disease, hyperlipidemia, malignancy, coronary artery disease, congestive heart failure, liver disease, renal disease, connective tissue disease, human immunodeficiency virus [HIV] infection, and alcoholism), monthly income, suicide, drug poisoning, and acute respiratory failure. We observed similar findings when we stratified the patients by age, sex, underlying comorbidities, and monthly income. The increased risk was most prominent in the first 2 weeks (AHR: 2.4; 95% CI: 2.1–2.7) and remained significant up to 6 months later (AHR: 1.6; 95% CI: 1.4–1.7). The risk for NS was higher in patients with COP who received HBOT than in those who did not, even after considering the possible impact of longer observation periods on survivors. Further studies that included the potential confounding factors we did not measure are needed to confirm findings in this study.

scholarly journals Delayed neuropsychiatric syndrome after carbon monoxide poisoning: inclusion of hyperbaric oxygen therapy in the recovery protocol

2016 ◽  
Vol 1 (1) ◽  
Author(s):  
Dante Lo Pardo ◽  
Davide Amedola ◽  
Giuliana Senatore ◽  
Alberto Damiano ◽  
Gabriela Pezzuti ◽  
...  
Keyword(s):  
Carbon Monoxide ◽  
Case Report ◽  
Diagnostic Imaging ◽  
Hyperbaric Oxygen ◽  
Hyperbaric Oxygen Therapy ◽  
Oxygen Therapy ◽  
Carbon Monoxide Poisoning ◽  
Neuropsychological Rehabilitation ◽  
Psychological Recovery

The delayed neuropsychiatric syndrome can arise in the period from 4 days to 5 weeks following carbon monoxide poisoning, and is characterized by neuropsychological deficits, which in some cases become chronic. This case report describes an adult female who apparently suffered self-inflicted carbon monoxide poisoning. She was not treated with hyperbaric oxygen and developed delayed sequelae on day 20. The treatment started with 40 sessions of hyperbaric oxygen therapy and subsequently with neuropsychological rehabilitation and physiotherapy. The treatment resulted in a progressive and almost complete physical and psychological recovery as evidenced by neuropsychometric tests and diagnostic imaging performed in the follow-up. Recovery was attributed to hyperbaric oxygen therapy. Although further research is required, we propose hyperbaric oxygen therapy also in the recovery protocol in the treatment of delayed neuropsychiatric syndrome.

Hyperthermia occurred after hyperbaric oxygen therapy for carbon monoxide poisoning

2010 ◽  
Vol 28 (7) ◽  
pp. 845.e5-845.e6 ◽  
Author(s):  
Seung Han Lee ◽  
Seung Ryu ◽  
Jin Woong Lee ◽  
Seung Whan Kim ◽  
In Sool Yoo ◽  
...  
Keyword(s):  
Carbon Monoxide ◽  
Hyperbaric Oxygen ◽  
Hyperbaric Oxygen Therapy ◽  
Oxygen Therapy ◽  
Carbon Monoxide Poisoning

scholarly journals Non-convulsive status epilepticus in a patient with carbon-monoxide poisoning treated with hyperbaric oxygen therapy

2016 ◽  
Vol 29 (6) ◽  
pp. 431-435 ◽  
Author(s):  
Simone Marziali ◽  
Francesca Di Giuliano ◽  
Eliseo Picchi ◽  
Silvia Natoli ◽  
Carlo Leonardis ◽  
...  
Keyword(s):  
Carbon Monoxide ◽  
Status Epilepticus ◽  
Hyperbaric Oxygen ◽  
Hyperbaric Oxygen Therapy ◽  
Oxygen Therapy ◽  
Carbon Monoxide Poisoning ◽  
Convulsive Status Epilepticus ◽  
Mri Findings

The presentation of carbon monoxide poisoning is non-specific and highly variable. Hyperbaric oxygen therapy is used for the treatment of this condition. Various reports show the occurrence of self-limiting seizures after carbon monoxide poisoning and as a consequence of hyperbaric oxygen therapy. Contrary to the seizures, status epilepticus has been rarely observed in these conditions. The exact pathophysiology underlying seizures and status epilepticus associated with carbon monoxide poisoning and hyperbaric oxygen therapy is not really clear, and some elements appear to be common to both conditions. We describe a case of non-convulsive status epilepticus in a patient with carbon monoxide poisoning treated with hyperbaric oxygen therapy. The mechanism, MRI findings and implications are discussed.

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