MASTOIDITIS IN ACUTE NUTRITIONAL DISTURBANCE

1929 ◽  
Vol 18 (4) ◽  
pp. 1774 ◽  
Author(s):  
LYMAN RICHARDS
Keyword(s):  
Nutritional Disturbance

scholarly journals CONCERNING COMPENSATORY HYPERPLASIA OF THE INTIMA

1910 ◽  
Vol 12 (6) ◽  
pp. 707-725 ◽  
Author(s):  
Oskar Klotz
Keyword(s):  
Connective Tissue ◽  
Muscle Fatigue ◽  
Intimal Hyperplasia ◽  
Bacterial Toxins ◽  
Peripheral Arteries ◽  
Nutritional Disturbance ◽  
Medial Degeneration ◽  
The Media ◽  
Arterial Tension

We agree with Jores and others that not one but many factors may be at work leading to intimal hyperplasia. Among these factors may be mentioned infection, bacterial toxins, organic poisons, inflammation and increased arterial tension. The theory of Thoma that the connective tissue developed in the intima is compensatory cannot be sustained. From the evidence which we have at hand it is not possible to state that the proliferative changes in the intima are uniformly secondary to the weakening of the media. Common influences may act simultaneously upon the media and the intima. Progressive medial degeneration of the peripheral arteries (Moenckeberg's sclerosis) is the result of muscle fatigue coupled with nutritional disturbance.

A Controlled Study of the EEG in Anorexia Nervosa

1968 ◽  
Vol 114 (514) ◽  
pp. 1149-1160 ◽  
Author(s):  
Arthur H. Crisp ◽  
George W. Fenton ◽  
Leila Scotton
Keyword(s):  
Anorexia Nervosa ◽  
Carbohydrate Intake ◽  
Controlled Study ◽  
Physiological Changes ◽  
Diurnal Temperature ◽  
Nutritional Disturbance ◽  
Carbohydrate Starvation ◽  
Temperature Rhythm

The nutritional disturbance in anorexia nervosa almost invariably leads to striking physiological changes, which include amenorrhoea, bradycardia, hypotension and relative hypothermia with reversal of the usual diurnal temperature rhythm (Bliss and Branch, 1960; Mayer-Grosset al., 1960; Crisp and Roberts, 1962; Crisp, 1967a). Crisp (1965a, 1967a) has commented on the characteristic and excessive alertness, restlessness and insomnia displayed by anorexia nervosa patients, whom he regards as showing a specific type of malnutrition associated with carbohydrate starvation. Russell (1967) has also recently demonstrated that patients with this illness restrict especially their carbohydrate intake.

CEROID STORAGE DISEASE IN CHILDHOOD

PEDIATRICS ◽  
1959 ◽  
Vol 23 (6) ◽  
pp. 1091-1102
Author(s):  
Ella H. Oppenheimer ◽  
Edward C. Andrews
Keyword(s):  
Smooth Muscle ◽  
Vitamin E ◽  
Intestinal Mucosa ◽  
Storage Disease ◽  
Cirrhosis Of The Liver ◽  
The Other ◽  
Vitamin E Deficiency ◽  
Nutritional Disturbance ◽  
History Of ◽  
Parenchymal Cells

Two cases of ceroid storage in childhood are reported. In both cases there was a long history of nutritional disturbance and cirrhosis of the liver was found at necropsy. In one case the storage of ceroid followed the pattern of so-called "lipidoses," with massive numbers of ceroid-filled cells in the spleen, liver and intestinal mucosa. In the other case the deposition of ceroid occurred mostly in macrophages in the liver. No ceroid was found in smooth muscle or other parenchymal cells as in vitamin E deficiency.

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