scholarly journals Reduced levels of XPA, ERCC1 and XPF DNA repair proteins in testis tumor cell lines

2004 ◽  
Vol 110 (3) ◽  
pp. 352-361 ◽  
Author(s):  
Carey Welsh ◽  
Roger Day ◽  
Claire McGurk ◽  
John R.W. Masters ◽  
Richard D. Wood ◽  
...  
Keyword(s):  
Dna Repair ◽  
Tumor Cell ◽  
Cell Lines ◽  
Tumor Cell Lines ◽  
Testis Tumor ◽  
Dna Repair Proteins

DNA-repair in mammary tumor cell lines with different X-ray sensitivities

1979 ◽  
Vol 35 (2) ◽  
pp. 190-191 ◽  
Author(s):  
J. Shaeffer ◽  
A. M. El-Mahdi ◽  
B. M. Karr
Keyword(s):  
Dna Repair ◽  
Tumor Cell ◽  
Cell Lines ◽  
Mammary Tumor ◽  
Tumor Cell Lines ◽  
Mammary Tumor Cell ◽  
X Ray

Complementation of a DNA repair deficiency in six human tumor cell lines by chromosome 11

1992 ◽  
Vol 88 (5) ◽  
pp. 524-528 ◽  
Author(s):  
R. Parshad ◽  
F. M. Price ◽  
M. Oshimura ◽  
J. C. Barrett ◽  
H. Satoh ◽  
...  
Keyword(s):  
Dna Repair ◽  
Tumor Cell ◽  
Cell Lines ◽  
Human Tumor ◽  
Tumor Cell Lines ◽  
Chromosome 11 ◽  
Human Tumor Cell ◽  
Human Tumor Cell Lines ◽  
Repair Deficiency ◽  
Dna Repair Deficiency

scholarly journals Loss of DUSP3 activity radiosensitizes human tumor cell lines via attenuation of DNA repair pathways

2017 ◽  
Vol 1861 (7) ◽  
pp. 1879-1894 ◽  
Author(s):  
Thompson E.P. Torres ◽  
Lilian C. Russo ◽  
Alexsandro Santos ◽  
Gabriela R. Marques ◽  
Yuli T. Magalhaes ◽  
...  
Keyword(s):  
Dna Repair ◽  
Tumor Cell ◽  
Cell Lines ◽  
Human Tumor ◽  
Tumor Cell Lines ◽  
Human Tumor Cell ◽  
Human Tumor Cell Lines ◽  
Repair Pathways

Interaction of Human Tumor Cells with Human Platelets and the Coagulation System

1983 ◽  
Vol 50 (03) ◽  
pp. 726-730 ◽  
Author(s):  
Hamid Al-Mondhiry ◽  
Virginia McGarvey ◽  
Kim Leitzel
Keyword(s):  
Tumor Cell ◽  
Tumor Cells ◽  
Cell Lines ◽  
Human Tumor ◽  
Human Platelets ◽  
Factor Vii ◽  
Coagulation System ◽  
Breast Adenocarcinoma ◽  
Activate Factor ◽  
Tumor Cell Lines

SummaryThis paper reports studies on the interaction between human platelets, the plasma coagulation system, and two human tumor cell lines grown in tissue culture: Melanoma and breast adenocarcinoma. The interaction was monitored through the use of 125I- labelled fibrinogen, which measures both thrombin activity generated by cell-plasma interaction and fibrin/fibrinogen binding to platelets and tumor cells. Each tumor cell line activates both the platelets and the coagulation system simultaneously resulting in the generation of thrombin or thrombin-like activity. The melanoma cells activate the coagulation system through “the extrinsic pathway” with a tissue factor-like effect on factor VII, but the breast tumor seems to activate factor X directly. Both tumor cell lines activate platelets to “make available” a platelet- derived procoagulant material necessary for the conversion of prothrombin to thrombin. The tumor-derived procoagulant activity and the platelet aggregating potential of cells do not seem to be inter-related, and they are not specific to malignant cells.

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