Urinary ethyl glucuronide testing detects alcohol consumption in alcoholic liver disease patients awaiting liver transplantation

2007 ◽  
Vol 13 (5) ◽  
pp. 757-761 ◽  
Author(s):  
Yesim Erim ◽  
Michael Böttcher ◽  
Uta Dahmen ◽  
Olof Beck ◽  
Christoph E. Broelsch ◽  
...  

2006 ◽  
Vol 12 (5) ◽  
pp. 813-820 ◽  
Author(s):  
Andrea DiMartini ◽  
Nancy Day ◽  
Mary Amanda Dew ◽  
Lubna Javed ◽  
Mary Grace Fitzgerald ◽  
...  


2005 ◽  
Vol 18 (11) ◽  
pp. 1292-1297 ◽  
Author(s):  
Pascal Perney ◽  
Michael Bismuth ◽  
Helene Sigaud ◽  
Marie Christine Picot ◽  
Eric Jacquet ◽  
...  




2001 ◽  
Vol 120 (5) ◽  
pp. A117-A117
Author(s):  
K DEAR ◽  
M BRADLEY ◽  
K MCCORMACK ◽  
R PECK ◽  
D GLEESON


HPB ◽  
2018 ◽  
Vol 20 ◽  
pp. S284
Author(s):  
B. Pattnaik ◽  
P. Sinha ◽  
S. Sasturkar ◽  
N. Mohapatra ◽  
V. Pamecha


2009 ◽  
Vol 29 (02) ◽  
pp. 233-237
Author(s):  
Stephen Ward ◽  
Charissa Chang ◽  
Lan Peng ◽  
Lawrence Liu


2018 ◽  
Vol 19 (12) ◽  
pp. 3857 ◽  
Author(s):  
Marica Meroni ◽  
Miriam Longo ◽  
Raffaela Rametta ◽  
Paola Dongiovanni

Alcoholic liver disease (ALD), a disorder caused by excessive alcohol consumption is a global health issue. More than two billion people consume alcohol in the world and about 75 million are classified as having alcohol disorders. ALD embraces a wide spectrum of hepatic lesions including steatosis, alcoholic steatohepatitis (ASH), fibrosis, cirrhosis, and hepatocellular carcinoma (HCC). ALD is a complex disease where environmental, genetic, and epigenetic factors contribute to its pathogenesis and progression. The severity of alcohol-induced liver disease depends on the amount, method of usage and duration of alcohol consumption as well as on age, gender, presence of obesity, and genetic susceptibility. Genome-wide association studies and candidate gene studies have identified genetic modifiers of ALD that can be exploited as non-invasive biomarkers, but which do not completely explain the phenotypic variability. Indeed, ALD development and progression is also modulated by epigenetic factors. The premise of this review is to discuss the role of genetic variants and epigenetic modifications, with particular attention being paid to microRNAs, as pathogenic markers, risk predictors, and therapeutic targets in ALD.



2019 ◽  
Vol 54 (9) ◽  
pp. 1146-1154 ◽  
Author(s):  
Andreas Schult ◽  
Knut Stokkeland ◽  
Bo-Göran Ericzon ◽  
Rolf Hultcrantz ◽  
Johan Franck ◽  
...  


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